Ray Chang
About
In The Last Decade
Ray Chang
26 papers receiving 314 citations
Peers
Comparison fields: 5 of 79
- Molecular Biology 129
- Biomedical Engineering 109
- Oncology 55
- Materials Chemistry 44
- Biomaterials 43
Countries citing papers authored by Ray Chang
This map shows the geographic impact of Ray Chang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ray Chang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ray Chang more than expected).
Fields of papers citing papers by Ray Chang
This network shows the impact of papers produced by Ray Chang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ray Chang. The network helps show where Ray Chang may publish in the future.
Co-authorship network of co-authors of Ray Chang
This figure shows the co-authorship network connecting the top 25 collaborators of Ray Chang. A scholar is included among the top collaborators of Ray Chang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ray Chang. Ray Chang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 4 | |
| 3 | 6 | |
| 4 | 3 | |
| 5 | 3 | |
| 6 | 6 | |
| 7 | 1 | |
| 8 | 41 | |
| 9 | 3 | |
| 10 | 2 | |
| 11 | 48 | |
| 12 | 14 | |
| 13 | 10 | |
| 14 | 5 | |
| 15 | 6 | |
| 16 | 2 | |
| 17 | 5 | |
| 18 | Influence of hepatocyte growth factor/scatter factor (HGF/SF) on fibroblast growth factor-2 (FGF-2) levels in external auditory canal cholesteatoma (EACC) cell culture. | 2 |
| 19 | In vitro sensitivity of T-cell lymphoblastic leukemia to UCN-01 (7-hydroxystaurosporine) is dependent on p16 protein status: a Pediatric Oncology Group study. | 7 |
| 20 | Universal inactivation of both p16 and p15 but not downstream components is an essential event in the pathogenesis of T-cell acute lymphoblastic leukemia. | 37 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.