Rashu B. Seth

6.2k total citations · 3 hit papers
8 papers, 5.1k citations indexed

About

Rashu B. Seth is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Rashu B. Seth has authored 8 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Immunology, 5 papers in Molecular Biology and 2 papers in Oncology. Recurrent topics in Rashu B. Seth's work include interferon and immune responses (6 papers), Immune Response and Inflammation (3 papers) and NF-κB Signaling Pathways (2 papers). Rashu B. Seth is often cited by papers focused on interferon and immune responses (6 papers), Immune Response and Inflammation (3 papers) and NF-κB Signaling Pathways (2 papers). Rashu B. Seth collaborates with scholars based in United States, Czechia and Finland. Rashu B. Seth's co-authors include Lijun Sun, Zhijian J. Chen, Chee-Kwee Ea, Gabriel Pineda, Xiao-Dong Li, Abdullah Shaito, Mei Hong, Atsuhiro Kanayama, Li Deng and Yu‐Hsin Chiu and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Rashu B. Seth

8 papers receiving 5.1k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3

2005 2.7k citations Rashu B. Seth, Lijun Sun et al. Cell profile →
TAB2 and TAB3 Activate the NF-κB Pathway through Binding to Polyubiquitin Chains

2004 724 citations Atsuhiro Kanayama, Rashu B. Seth et al. Molecular Cell profile →
Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity

2005 666 citations Xiao-Dong Li, Lijun Sun et al. Proceedings of the National Academy of Sciences profile →

Peers

Countries citing papers authored by Rashu B. Seth

Since Specialization

Citations

This map shows the geographic impact of Rashu B. Seth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rashu B. Seth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rashu B. Seth more than expected).

Fields of papers citing papers by Rashu B. Seth

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rashu B. Seth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rashu B. Seth. The network helps show where Rashu B. Seth may publish in the future.

Co-authorship network of co-authors of Rashu B. Seth

This figure shows the co-authorship network connecting the top 25 collaborators of Rashu B. Seth. A scholar is included among the top collaborators of Rashu B. Seth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rashu B. Seth. Rashu B. Seth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

#	Work	Indexed citations
1	Topologically Associated Domains Delineate Susceptibility to Somatic Hypermutation 2019 ·Cell Reports ·Filip Šenigl, Yaakov Maman, Ravi K. Dinesh, Jukka Alinikula, Rashu B. Seth, (unknown), Arina D. Omer, Suhas S.P. Rao, David Weisz, Jean-Marie Buerstedde, Erez Lieberman Aiden, Rafael Casellas, Jiřı́ Hejnar, David G. Schatz	28
2	RAG1 targeting in the genome is dominated by chromatin interactions mediated by the non-core regions of RAG1 and RAG2 2016 ·Nucleic Acids Research ·Yaakov Maman, Grace Teng, Rashu B. Seth, Steven H. Kleinstein, David G. Schatz	19
3	Antiviral innate immunity pathways 2006 ·Cell Research ·Rashu B. Seth, Lijun Sun, Zhijian J. Chen	382
4	The Specific and Essential Role of MAVS in Antiviral Innate Immune Responses 2006 ·Immunity ·(unknown), Lijun Sun, Hong-Hsing Liu, Xiang Chen, Rashu B. Seth, James Forman, Zhijian J. Chen	499
5	Ubiquitin, TAK1 and IKK: is there a connection? 2006 ·Cell Death and Differentiation ·Zhe Chen, Vijay Bhoj, Rashu B. Seth	102
6	Hepatitis C virus protease NS3/4A cleaves mitochondrial antiviral signaling protein off the mitochondria to evade innate immunity breakdown → 2005 ·Proceedings of the National Academy of Sciences ·Xiao-Dong Li, Lijun Sun, Rashu B. Seth, Gabriel Pineda, Zhijian J. Chen	666
7	Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3 breakdown → 2005 ·Cell ·Rashu B. Seth, Lijun Sun, Chee-Kwee Ea, Zhijian J. Chen	2703
8	TAB2 and TAB3 Activate the NF-κB Pathway through Binding to Polyubiquitin Chains breakdown → 2004 ·Molecular Cell ·Atsuhiro Kanayama, Rashu B. Seth, Lijun Sun, Chee-Kwee Ea, Mei Hong, Abdullah Shaito, Yu‐Hsin Chiu, Li Deng, Zhijian J. Chen	724

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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