R Prasad
- Hematology top 1%
- Acute Myeloid Leukemia Research 4
- Molecular Biology top 5%
- RNA Interference and Gene Delivery 4
- DNA Repair Mechanisms 3
- Protein Kinase Regulation and GTPase Signaling 1
- Genomics and Chromatin Dynamics 1
-
- Acute Lymphoblastic Leukemia research 6
- Genetics top 10%
- Genomic variations and chromosomal abnormalities 2
- Cell Biology top 10%
-
- Cytokine Signaling Pathways and Interactions 1
R Prasad
11 papers receiving 2.0k citations
Hit Papers
Peers
Comparison fields: 5 of 79
- Hematology 901
- Molecular Biology 1.6k
- Public Health, Environmental and Occupational Health 469
- Genetics 138
- Cell Biology 148
Countries citing papers authored by R Prasad
This map shows the geographic impact of R Prasad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R Prasad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R Prasad more than expected).
Fields of papers citing papers by R Prasad
This network shows the impact of papers produced by R Prasad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R Prasad. The network helps show where R Prasad may publish in the future.
Co-authorship network
The 25 scholars most cited alongside R Prasad, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 31 | |
| 2 | 1997 | 76 | |
| 3 | 1996 | 179 | |
| 4 | 1995 | 117 | |
| 5 | 1994 | 115 | |
| 6 | Sequence analysis of the breakpoint cluster region in the ALL-1 gene involved in acute leukemia. | 1994 | 134 |
| 7 | 1993 | 243 | |
| 8 | Cloning of the ALL-1 fusion partner, the AF-6 gene, involved in acute myeloid leukemias with the t(6;11) chromosome translocation. | 1993 | 227 |
| 9 | The t(4;11) chromosome translocation of human acute leukemias fuses the ALL-1 gene, related to Drosophila trithorax, to the AF-4 genebreakdown → | 1992 | 734 |
| 10 | An altered 11-kilobase transcript in leukemic cell lines with the t(4;11)(q21;q23) chromosome translocation. | 1992 | 50 |
| 11 | 1992 | 88 |
About R Prasad
R Prasad is a scholar working on Hematology, Public Health, Environmental and Occupational Health and Molecular Biology, having authored 11 papers that have together received 2.0k indexed citations. Recurring topics across this work include Acute Lymphoblastic Leukemia research (6 papers), Acute Myeloid Leukemia Research (4 papers), RNA Interference and Gene Delivery (4 papers), DNA Repair Mechanisms (3 papers), Genomic variations and chromosomal abnormalities (2 papers), Cytokine Signaling Pathways and Interactions (1 paper), Protein Kinase Regulation and GTPase Signaling (1 paper) and Genomics and Chromatin Dynamics (1 paper). The work is most often cited by research in Hematology (901 citations), Molecular Biology (1.6k citations) and Public Health, Environmental and Occupational Health (469 citations). R Prasad has collaborated with scholars based in United States, Israel and India. Frequent co-authors include Y Gu, Tatsuya Nakamura, Hansjüerg Alder, Ora Canaani, Carlo M. Croce, Eli Canaani, Giuseppe Cimino, Peter C. Nowell̀, Hirohisa Saito and R P Gale. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.