R. A. Metcalfe
- Endocrinology, Diabetes and Metabolism top 5%
- Pathology and Forensic Medicine top 10%
- Molecular Biology
- Immunology
- Genetics
- Co-authors
- Anthony P. WeetmanM. CrispRamzi AjjanMarian LudgateA. P. WeetmanNikhil TandonDavid MilneJohn Thompson
- Topics
- Thyroid Disorders and Treatments (9 papers)Ophthalmology and Eye Disorders (5 papers)Monoclonal and Polyclonal Antibodies Research (5 papers)
- Partner nations
- United KingdomDenmarkCanada
In The Last Decade
R. A. Metcalfe
28 papers receiving 684 citations
Peers
Comparison fields: 5 of 80
- Endocrinology, Diabetes and Metabolism 313
- Pathology and Forensic Medicine 141
- Molecular Biology 129
- Immunology 119
- Genetics 93
Countries citing papers authored by R. A. Metcalfe
This map shows the geographic impact of R. A. Metcalfe's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R. A. Metcalfe with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R. A. Metcalfe more than expected).
Fields of papers citing papers by R. A. Metcalfe
This network shows the impact of papers produced by R. A. Metcalfe. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R. A. Metcalfe. The network helps show where R. A. Metcalfe may publish in the future.
Co-authorship network of co-authors of R. A. Metcalfe
This figure shows the co-authorship network connecting the top 25 collaborators of R. A. Metcalfe. A scholar is included among the top collaborators of R. A. Metcalfe based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with R. A. Metcalfe. R. A. Metcalfe is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 59 | |
| 2 | 10 | |
| 3 | 15 | |
| 4 | 12 | |
| 5 | 95 | |
| 6 | 16 | |
| 7 | 56 | |
| 8 | 24 | |
| 9 | 20 | |
| 10 | 7 | |
| 11 | 11 | |
| 12 | 8 | |
| 13 | 98 | |
| 14 | 14 | |
| 15 | 11 | |
| 16 | Adhesion molecule monoclonal antibodies inhibit experimental autoimmune thyroiditis. | 18 |
| 17 | 4 | |
| 18 | 19 | |
| 19 | 7 | |
| 20 | 43 |
About R. A. Metcalfe
R. A. Metcalfe is a scholar working on Endocrinology, Diabetes and Metabolism, Pathology and Forensic Medicine and Immunology, having authored 31 papers that have together received 717 indexed citations. Recurring topics across this work include Thyroid Disorders and Treatments (9 papers), Ophthalmology and Eye Disorders (5 papers) and Monoclonal and Polyclonal Antibodies Research (5 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (313 citations), Pathology and Forensic Medicine (141 citations) and Ophthalmology (61 citations). R. A. Metcalfe has collaborated with scholars based in United Kingdom, Denmark and Canada. Frequent co-authors include Anthony P. Weetman, M. Crisp, Ramzi Ajjan, Marian Ludgate, A. P. Weetman, Nikhil Tandon, David Milne, John Thompson, Elizabeth A. Edwards and Catherine A. Byrnes. Their work appears in journals such as The Journal of Clinical Endocrinology & Metabolism, Neurology and Human Reproduction.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.