Qing‐Ping Yao

1.1k total citations
46 papers, 899 citations indexed

About

Qing‐Ping Yao is a scholar working on Molecular Biology, Cancer Research and Surgery. According to data from OpenAlex, Qing‐Ping Yao has authored 46 papers receiving a total of 899 indexed citations (citations by other indexed papers that have themselves been cited), including 28 papers in Molecular Biology, 15 papers in Cancer Research and 7 papers in Surgery. Recurrent topics in Qing‐Ping Yao's work include MicroRNA in disease regulation (10 papers), Cancer-related molecular mechanisms research (10 papers) and Circular RNAs in diseases (7 papers). Qing‐Ping Yao is often cited by papers focused on MicroRNA in disease regulation (10 papers), Cancer-related molecular mechanisms research (10 papers) and Circular RNAs in diseases (7 papers). Qing‐Ping Yao collaborates with scholars based in China, United States and Israel. Qing‐Ping Yao's co-authors include Zong‐Lai Jiang, Ying‐Xin Qi, Yue Han, Bao‐Rong Shen, Kai Huang, Zhiqiang Yan, Lu Wang, Ping Zhang, Jun Jiang and Shu Chien and has published in prestigious journals such as Proceedings of the National Academy of Sciences, SHILAP Revista de lepidopterología and PLoS ONE.

In The Last Decade

Qing‐Ping Yao

45 papers receiving 886 citations

Peers

Qing‐Ping Yao
Zhenwu Zhuang United States
Wei Tian China
Malgorzata Furmanik United Kingdom
Yao Wei Lu United States
Marcy Martin United States
Zhenwu Zhuang United States
Qing‐Ping Yao
Citations per year, relative to Qing‐Ping Yao Qing‐Ping Yao (= 1×) peers Zhenwu Zhuang

Countries citing papers authored by Qing‐Ping Yao

Since Specialization
Citations

This map shows the geographic impact of Qing‐Ping Yao's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qing‐Ping Yao with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qing‐Ping Yao more than expected).

Fields of papers citing papers by Qing‐Ping Yao

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qing‐Ping Yao. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qing‐Ping Yao. The network helps show where Qing‐Ping Yao may publish in the future.

Co-authorship network of co-authors of Qing‐Ping Yao

This figure shows the co-authorship network connecting the top 25 collaborators of Qing‐Ping Yao. A scholar is included among the top collaborators of Qing‐Ping Yao based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qing‐Ping Yao. Qing‐Ping Yao is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Lou, Yue, Wenhao Tian, Jingjing Cui, et al.. (2025). MIAT-DHX9 spatiotemporal expression drives venous neointimal hyperplasia through nucleolar homeostasis and mitotic progression. Cell Reports. 44(12). 116578–116578.
2.
Wang, Wei, et al.. (2024). Development and optimization of green extraction of polyphenols in Michelia alba using natural deep eutectic solvents (NADES) and evaluation of bioactivity. Sustainable Chemistry and Pharmacy. 37. 101425–101425. 29 indexed citations
3.
Meng, Jie, et al.. (2024). Meta-analysis and sequential analysis of acupuncture compared to carbamazepine in the treatment of trigeminal neuralgia. World Journal of Clinical Cases. 12(22). 5083–5093. 1 indexed citations
4.
Xu, Jiaying, et al.. (2024). Transfer RNA-derived small RNA tRF-Glu-CTC attenuates neointimal formation via inhibition of fibromodulin. Cellular & Molecular Biology Letters. 29(1). 2–2. 6 indexed citations
6.
Li, Tao, Qin Xu, Yang Zeng, et al.. (2023). Methyl donor diet attenuates intimal hyperplasia after vascular injury in rats. The Journal of Nutritional Biochemistry. 123. 109486–109486. 2 indexed citations
7.
Liu, Ji-Ting, Qing‐Ping Yao, Yi Chen, et al.. (2022). Arterial cyclic stretch regulates Lamtor1 and promotes neointimal hyperplasia via circSlc8a1/miR-20a-5p axis in vein grafts. Theranostics. 12(11). 4851–4865. 5 indexed citations
8.
Liu, Ji-Ting, Han Bao, Qing‐Ping Yao, et al.. (2021). Platelet-Derived Microvesicles Promote VSMC Dedifferentiation After Intimal Injury via Src/Lamtor1/mTORC1 Signaling. Frontiers in Cell and Developmental Biology. 9. 744320–744320. 9 indexed citations
9.
Li, Haipeng, Ji-Ting Liu, Wenbin Wang, et al.. (2020). Suppressed nuclear envelope proteins activate autophagy of vascular smooth muscle cells during cyclic stretch application. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1868(1). 118855–118855. 9 indexed citations
10.
Yan, Jing, Wenbin Wang, Han Bao, et al.. (2020). Cyclic Stretch Induces Vascular Smooth Muscle Cells to Secrete Connective Tissue Growth Factor and Promote Endothelial Progenitor Cell Differentiation and Angiogenesis. Frontiers in Cell and Developmental Biology. 8. 606989–606989. 17 indexed citations
11.
Li, Shanshan, Shuang Gao, Yi Chen, et al.. (2020). Platelet-derived microvesicles induce calcium oscillations and promote VSMC migration via TRPV4. Theranostics. 11(5). 2410–2423. 16 indexed citations
12.
Zhu, Xiaoling, Tao Li, Yu Cao, et al.. (2020). tRNA-derived fragments tRFGlnCTG induced by arterial injury promote vascular smooth muscle cell proliferation. Molecular Therapy — Nucleic Acids. 23. 603–613. 20 indexed citations
13.
Rong, Zhihua, Qing‐Ping Yao, Tao Li, et al.. (2019). Suppression of circDcbld1 Alleviates Intimal Hyperplasia in Rat Carotid Artery by Targeting miR-145-3p/Neuropilin-1. Molecular Therapy — Nucleic Acids. 18. 999–1008. 20 indexed citations
14.
Yao, Qing‐Ping, Kaixuan Wang, Ping Zhang, et al.. (2017). Profiles of long noncoding RNAs in hypertensive rats. Journal of Hypertension. 35(6). 1195–1203. 37 indexed citations
15.
Han, Yue, Lu Wang, Qing‐Ping Yao, et al.. (2015). Nuclear envelope proteins Nesprin2 and LaminA regulate proliferation and apoptosis of vascular endothelial cells in response to shear stress. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1853(5). 1165–1173. 31 indexed citations
16.
Zhang, Ping, Ying‐Xin Qi, Qing‐Ping Yao, et al.. (2015). Neuropeptide Y Stimulates Proliferation and Migration of Vascular Smooth Muscle Cells from Pregnancy Hypertensive Rats via Y1 and Y5 Receptors. PLoS ONE. 10(7). e0131124–e0131124. 22 indexed citations
17.
Wang, Lu, Yue Han, Yan Shen, et al.. (2013). Endothelial Insulin-Like Growth Factor-1 Modulates Proliferation and Phenotype of Smooth Muscle Cells Induced by Low Shear Stress. Annals of Biomedical Engineering. 42(4). 776–786. 36 indexed citations
18.
Yao, Qing‐Ping, Ying‐Xin Qi, Ping Zhang, et al.. (2013). SIRT1 and Connexin40 Mediate the Normal Shear Stress-Induced Inhibition of the Proliferation of Endothelial Cells Co-Cultured with Vascular Smooth Muscle Cells. Cellular Physiology and Biochemistry. 31(2-3). 389–399. 19 indexed citations
19.
Yan, Zhiqiang, Qing‐Ping Yao, Bao‐Rong Shen, et al.. (2012). Association of SIRT1 expression with shear stress induced endothelial progenitor cell differentiation. Journal of Cellular Biochemistry. 113(12). 3663–3671. 31 indexed citations
20.
Qi, Ying‐Xin, Mingjuan Qu, Bo Liu, et al.. (2008). Rho-GDP dissociation inhibitor alpha downregulated by low shear stress promotes vascular smooth muscle cell migration and apoptosis: a proteomic analysis. Cardiovascular Research. 80(1). 114–122. 63 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026