Qing‐Peng Kong

6.3k citations
111 papers · 3.8k indexed · h-index 36
Topics
Mitochondrial Function and Pathology (29 papers)Forensic and Genetic Research (26 papers)Genetics, Aging, and Longevity in Model Organisms (18 papers)

In The Last Decade

Qing‐Peng Kong

106 papers receiving 3.7k citations

Peers

Qing‐Peng Kong
Comparison fields: 5 of 145
  • Molecular Biology 2.3k
  • Genetics 1.6k
  • Archeology 548
  • Clinical Biochemistry 426
  • Cancer Research 326
Replace Martin Brandon with:
Martin Brandon United States
Yvonne H. Edwards United Kingdom
Kirsi Huoponen Finland
Mannis van Oven Netherlands
Paweł Golik Poland
Douglas C. Wallace United States
N. O. Bianchi Argentina
Chad D. Huff United States
Almut Nebel Germany
Andrea Novelletto Italy
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Citations per field
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Martin Brandon · 1×
Citations per year

Countries citing papers authored by Qing‐Peng Kong

Since Specialization
Citations

This map shows the geographic impact of Qing‐Peng Kong's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qing‐Peng Kong with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qing‐Peng Kong more than expected).

Fields of papers citing papers by Qing‐Peng Kong

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qing‐Peng Kong. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qing‐Peng Kong. The network helps show where Qing‐Peng Kong may publish in the future.

Co-authorship network of co-authors of Qing‐Peng Kong

This figure shows the co-authorship network connecting the top 25 collaborators of Qing‐Peng Kong. A scholar is included among the top collaborators of Qing‐Peng Kong based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qing‐Peng Kong. Qing‐Peng Kong is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 7
2 42
3 3
4 176
5 90
6 15
7 69
8 42
9 6
10 3
11
[Mitochondrial respiratory chain complex I deficiency due to 10191T>C mutation in ND3 gene].
5
12 11
13 22
14 7
15 59
16 50
17 330
18 17
19 244
20 85

About Qing‐Peng Kong

Qing‐Peng Kong is a scholar working on Aging, Clinical Biochemistry and Genetics, having authored 111 papers that have together received 3.8k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (29 papers), Forensic and Genetic Research (26 papers) and Genetics, Aging, and Longevity in Model Organisms (18 papers). The work is most often cited by research in Aging (127 citations), Clinical Biochemistry (426 citations) and Genetics (1.6k citations). Qing‐Peng Kong has collaborated with scholars based in China, Hong Kong and United States. Frequent co-authors include Ya‐Ping Zhang, Yong‐Gang Yao, Hans‐Jürgen Bandelt, Chang Sun, Yonghan He, Chunling Zhu, Chengye Wang, Fu‐Hui Xiao, Malliya Gounder Palanichamy and Tapas K. Chaudhuri. Their work appears in journals such as Science, Proceedings of the National Academy of Sciences and PLoS ONE.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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