Qingcheng Yang

2.8k total citations
55 papers, 1.0k citations indexed

About

Qingcheng Yang is a scholar working on Molecular Biology, Oncology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Qingcheng Yang has authored 55 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 16 papers in Oncology and 12 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Qingcheng Yang's work include Sarcoma Diagnosis and Treatment (12 papers), RNA modifications and cancer (10 papers) and RNA Research and Splicing (10 papers). Qingcheng Yang is often cited by papers focused on Sarcoma Diagnosis and Treatment (12 papers), RNA modifications and cancer (10 papers) and RNA Research and Splicing (10 papers). Qingcheng Yang collaborates with scholars based in China, Germany and United States. Qingcheng Yang's co-authors include Dongdong Cheng, Bin Zhu, Shumin Zhou, Tu Hu, Cunyi Fan, Kan Yang, Jielai Yang, Bingfang Zeng, Jin Huang and Hua Guo and has published in prestigious journals such as Nature Communications, Journal of Clinical Oncology and SHILAP Revista de lepidopterología.

In The Last Decade

Qingcheng Yang

52 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Qingcheng Yang China 22 658 411 214 196 93 55 1.0k
Jun Hao China 18 735 1.1× 533 1.3× 143 0.7× 192 1.0× 81 0.9× 38 1.0k
Xiaojun Zhou China 19 779 1.2× 611 1.5× 219 1.0× 190 1.0× 70 0.8× 85 1.2k
Na Qin China 21 717 1.1× 534 1.3× 335 1.6× 292 1.5× 86 0.9× 85 1.2k
Tong Lu China 19 716 1.1× 523 1.3× 259 1.2× 181 0.9× 70 0.8× 67 1.1k
María Sol Brassesco Brazil 20 795 1.2× 432 1.1× 150 0.7× 218 1.1× 64 0.7× 106 1.4k
Cuijuan Zhang China 18 790 1.2× 527 1.3× 80 0.4× 218 1.1× 156 1.7× 39 1.1k
Xiaoming Hou China 19 382 0.6× 207 0.5× 126 0.6× 175 0.9× 111 1.2× 60 747
Coro Bescós Spain 12 785 1.2× 847 2.1× 197 0.9× 343 1.8× 130 1.4× 35 1.6k
Gervaise H. Henry United States 11 440 0.7× 205 0.5× 317 1.5× 152 0.8× 123 1.3× 17 1.1k
Momoe Itsumi Japan 17 455 0.7× 252 0.6× 229 1.1× 182 0.9× 251 2.7× 38 1.0k

Countries citing papers authored by Qingcheng Yang

Since Specialization
Citations

This map shows the geographic impact of Qingcheng Yang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qingcheng Yang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qingcheng Yang more than expected).

Fields of papers citing papers by Qingcheng Yang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qingcheng Yang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qingcheng Yang. The network helps show where Qingcheng Yang may publish in the future.

Co-authorship network of co-authors of Qingcheng Yang

This figure shows the co-authorship network connecting the top 25 collaborators of Qingcheng Yang. A scholar is included among the top collaborators of Qingcheng Yang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qingcheng Yang. Qingcheng Yang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Fei, Xiang, Zhaohui Li, Zhen Pan, et al.. (2024). Avermectin B1 mediates antitumor activity and induces autophagy in osteosarcoma through the AMPK/ULK1 signaling pathway. Cancer Chemotherapy and Pharmacology. 94(4). 599–613.
3.
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Zhang, Zhichang, Weiping Ji, Jin Huang, et al.. (2022). Characterization of the tumour microenvironment phenotypes in malignant tissues and pleural effusion from advanced osteoblastic osteosarcoma patients. Clinical and Translational Medicine. 12(11). e1072–e1072. 25 indexed citations
5.
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Pan, Zhen, Shijie Li, Hua Guo, et al.. (2022). Ebastine exerts antitumor activity and induces autophagy by activating AMPK/ULK1 signaling in an IPMK-dependent manner in osteosarcoma. International Journal of Biological Sciences. 19(2). 537–551. 14 indexed citations
7.
Yu, Miao, Yang Dong, Qingcheng Yang, et al.. (2022). ANGPTL4 negatively regulates the progression of osteosarcoma by remodeling branched-chain amino acid metabolism. Cell Death Discovery. 8(1). 225–225. 15 indexed citations
8.
He, Yunfei, Dongdong Cheng, Cheng Lian, et al.. (2021). Serglycin induces osteoclastogenesis and promotes tumor growth in giant cell tumor of bone. Cell Death and Disease. 12(10). 868–868. 18 indexed citations
9.
Cheng, Dongdong, et al.. (2021). Chitooligosaccharides inhibit tumor progression and induce autophagy through the activation of the p53/mTOR pathway in osteosarcoma. Carbohydrate Polymers. 258. 117596–117596. 46 indexed citations
10.
Li, Xiaoxun, Yajun Liang, Cheng Lian, et al.. (2021). CST6 protein and peptides inhibit breast cancer bone metastasis by suppressing CTSB activity and osteoclastogenesis. Theranostics. 11(20). 9821–9832. 46 indexed citations
11.
Dong, Yang, et al.. (2021). Alpha-(1,6)-fucosyltransferase (FUT8) affects the survival strategy of osteosarcoma by remodeling TNF/NF-κB2 signaling. Cell Death and Disease. 12(12). 1124–1124. 17 indexed citations
12.
Cheng, Dongdong, et al.. (2018). EEF1D overexpression promotes osteosarcoma cell proliferation by facilitating Akt-mTOR and Akt-bad signaling. Journal of Experimental & Clinical Cancer Research. 37(1). 50–50. 26 indexed citations
13.
Cheng, Dongdong, et al.. (2017). CSE1L interaction with MSH6 promotes osteosarcoma progression and predicts poor patient survival. Scientific Reports. 7(1). 46238–46238. 22 indexed citations
14.
Cheng, Dongdong, et al.. (2017). Down-regulation of RPS9 Inhibits Osteosarcoma Cell Growth through Inactivation of MAPK Signaling Pathway. Journal of Cancer. 8(14). 2720–2728. 24 indexed citations
15.
Hu, Tu, et al.. (2015). DEC2 expression is positively correlated with HIF-1 activation and the invasiveness of human osteosarcomas. Journal of Experimental & Clinical Cancer Research. 34(1). 22–22. 35 indexed citations
16.
Zheng, Shuier, Guanglei Qiao, Daliu Min, et al.. (2015). Heterogeneous expression and biological function of ubiquitin carboxy-terminal hydrolase-L1 in osteosarcoma. Cancer Letters. 359(1). 36–46. 31 indexed citations
17.
Yang, Jielai, Bin Zhu, Kai Fu, & Qingcheng Yang. (2015). The long-term outcomes following the use of inactivated autograft in the treatment of primary malignant musculoskeletal tumor. Journal of Orthopaedic Surgery and Research. 10(1). 177–177. 7 indexed citations
18.
Yang, Qingcheng, et al.. (2014). miR-133a mediates the hypoxia-induced apoptosis by inhibiting TAGLN2 expression in cardiac myocytes. Molecular and Cellular Biochemistry. 400(1-2). 173–181. 47 indexed citations
19.
Cheng, Dongdong, et al.. (2012). Antitumor Activity of Histone Deacetylase Inhibitor Trichostatin A in Osteosarcoma Cells. Asian Pacific Journal of Cancer Prevention. 13(4). 1395–1399. 18 indexed citations
20.
Zhang, Lizhi, et al.. (2010). Tumor‐initiating cells and tumor vascularization. Pediatric Blood & Cancer. 56(3). 335–340. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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