Poh‐Gek Forkert

2.0k total citations
69 papers, 1.5k citations indexed

About

Poh‐Gek Forkert is a scholar working on Pharmacology, Molecular Biology and Cancer Research. According to data from OpenAlex, Poh‐Gek Forkert has authored 69 papers receiving a total of 1.5k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Pharmacology, 21 papers in Molecular Biology and 21 papers in Cancer Research. Recurrent topics in Poh‐Gek Forkert's work include Pharmacogenetics and Drug Metabolism (25 papers), Carcinogens and Genotoxicity Assessment (20 papers) and Eicosanoids and Hypertension Pharmacology (10 papers). Poh‐Gek Forkert is often cited by papers focused on Pharmacogenetics and Drug Metabolism (25 papers), Carcinogens and Genotoxicity Assessment (20 papers) and Eicosanoids and Hypertension Pharmacology (10 papers). Poh‐Gek Forkert collaborates with scholars based in Canada, United States and Bangladesh. Poh‐Gek Forkert's co-authors include Alison B. Froese, Madeleine Moussa, Edward S. Reynolds, Taylor F. Dowsley, Judith B. Ulreich, Daniel W. Birch, H V Gelboin, Thomas E. Massey, William J. Racz and John S. Poland and has published in prestigious journals such as Journal of Pharmacology and Experimental Therapeutics, International Journal of Cancer and Molecular Pharmacology.

In The Last Decade

Poh‐Gek Forkert

68 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Poh‐Gek Forkert Canada	23	474	436	431	384	258	69	1.5k
Manfred Kiese Germany	25	465 1.0×	148 0.3×	561 1.3×	181 0.5×	333 1.3×	114	1.9k
H.P. Witschi United States	20	181 0.4×	274 0.6×	408 0.9×	287 0.7×	72 0.3×	43	1.5k
Anders Tunek Sweden	23	226 0.5×	430 1.0×	465 1.1×	333 0.9×	162 0.6×	42	1.7k
H.J. Segall United States	31	643 1.4×	446 1.0×	1.4k 3.3×	181 0.5×	84 0.3×	83	2.3k
Shu Ning United States	19	630 1.3×	117 0.3×	478 1.1×	219 0.6×	191 0.7×	41	1.3k
T.C. Orton United Kingdom	16	292 0.6×	78 0.2×	633 1.5×	271 0.7×	75 0.3×	40	1.4k
D. V. Parke United Kingdom	23	489 1.0×	43 0.1×	435 1.0×	284 0.7×	170 0.7×	50	1.5k
John G. Dent United States	23	400 0.8×	68 0.2×	255 0.6×	368 1.0×	124 0.5×	54	1.3k
Anne P. Autor United States	23	161 0.3×	268 0.6×	785 1.8×	81 0.2×	141 0.5×	43	2.0k
Francis J. Peterson United States	17	216 0.5×	93 0.2×	238 0.6×	75 0.2×	113 0.4×	29	977

Countries citing papers authored by Poh‐Gek Forkert

Since Specialization

Citations

This map shows the geographic impact of Poh‐Gek Forkert's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Poh‐Gek Forkert with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Poh‐Gek Forkert more than expected).

Fields of papers citing papers by Poh‐Gek Forkert

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Poh‐Gek Forkert. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Poh‐Gek Forkert. The network helps show where Poh‐Gek Forkert may publish in the future.

Co-authorship network of co-authors of Poh‐Gek Forkert

This figure shows the co-authorship network connecting the top 25 collaborators of Poh‐Gek Forkert. A scholar is included among the top collaborators of Poh‐Gek Forkert based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Poh‐Gek Forkert. Poh‐Gek Forkert is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Soeteman‐Hernández, Lya G. & Poh‐Gek Forkert. (2008). Inhibition of vinyl carbamate-induced lung tumors and Kras2 mutations by the garlic derivative diallyl sulfone. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 662(1-2). 16–21. 6 indexed citations

Soeteman‐Hernández, Lya G. & Poh‐Gek Forkert. (2007). Inhibition of vinyl carbamate-induced mutagenicity and clastogenicity by the garlic constituent diallyl sulfone in F1 (Big Blue® × A/J) transgenic mice. Carcinogenesis. 28(8). 1824–1830. 5 indexed citations

Forkert, Poh‐Gek, et al.. (2007). Oxidation of Vinyl Carbamate and Formation of 1,N6-Ethenodeoxyadenosine in Murine Lung. Drug Metabolism and Disposition. 35(5). 713–720. 9 indexed citations

Ghanayem, Burhan I., Ashish Sharma, Christopher A. Reilly, et al.. (2004). Bioactivation of 1,1-Dichloroethylene by CYP2E1 and CYP2F2 in Murine Lung. Journal of Pharmacology and Experimental Therapeutics. 310(3). 855–864. 18 indexed citations

Vultur, Adina, Katherine A. Peebles, Alvin M. Malkinson, et al.. (2003). Gap Junctional Intercellular Communication in Cells Isolated from Urethane-Induced Tumors in A/J Mice. DNA and Cell Biology. 22(1). 33–40. 7 indexed citations

Racz, William J., et al.. (2003). Mitochondrial Dysfunction Is an Early Manifestation of 1,1-Dichloroethylene-Induced Hepatotoxicity in Mice. Journal of Pharmacology and Experimental Therapeutics. 304(1). 121–129. 12 indexed citations

Forkert, Poh‐Gek, Lawrence H. Lash, Robert Tardif, et al.. (2003). Identification of Trichloroethylene and Its Metabolites in Human Seminal Fluid of Workers Exposed to Trichloroethylene. Drug Metabolism and Disposition. 31(3). 306–311. 35 indexed citations

Forkert, Poh‐Gek, et al.. (1999). Inactivation of Cytochrome P-450 (CYP2E1) and Carboxylesterase (Hydrolase A) Enzymes by Vinyl Carbamate in Murine Pulmonary Microsomes. Drug Metabolism and Disposition. 27(2). 233–239. 14 indexed citations

Forkert, Poh‐Gek. (1997). Conjugation of glutathione with the reactive metabolites of 1,1-dichloroethylene in murine lung and liver. Microscopy Research and Technique. 36(4). 234–242. 8 indexed citations

10.

Forkert, Poh‐Gek, et al.. (1997). Metabolism of Ethyl Carbamate by Pulmonary Cytochrome P450 and Carboxylesterase Isozymes: Involvement of CYP2E1 and Hydrolase A. Toxicology and Applied Pharmacology. 146(2). 245–254. 32 indexed citations

11.

Dowsley, Taylor F., Judith B. Ulreich, Judy L. Bolton, Sang S. Park, & Poh‐Gek Forkert. (1996). CYP2E1-Dependent Bioactivation of 1,1-Dichloroethylene in Murine Lung: Formation of Reactive Intermediates and Glutathione Conjugates. Toxicology and Applied Pharmacology. 139(1). 42–48. 21 indexed citations

12.

Forkert, Poh‐Gek, Alan C. Jackson, A. Parkinson, & Shawn Chen. (1996). Diminished Expression of CYP1A1 in Urethane-Induced Lung Tumors in Strain A/J Mice: Analysis by in Situ Hybridization and Immunohistochemical Methods. American Journal of Respiratory Cell and Molecular Biology. 14(5). 444–453. 7 indexed citations

13.

Forkert, Poh‐Gek, et al.. (1996). Protection from 1,1-dichloroethylene-induced Clara cell injury by diallyl sulfone, a derivative of garlic.. Journal of Pharmacology and Experimental Therapeutics. 277(3). 1665–1671. 21 indexed citations

14.

Forkert, Poh‐Gek, Jason Lord, & Andrew Parkinson. (1996). Alterations in expression of CYPlA1 and NADPH-cytochrome P450 reductase during lung tumor development in SWR/J mice. Carcinogenesis. 17(1). 127–132. 19 indexed citations

15.

Forkert, Poh‐Gek. (1995). CYP2E1 is Preferentially Expressed in Clara Cells of Murine Lung: Localization by in Situ Hybridization and Immunohistochemical Methods. American Journal of Respiratory Cell and Molecular Biology. 12(6). 589–596. 52 indexed citations

16.

Forkert, Poh‐Gek, et al.. (1994). Induction and regulation of CYP2E1 in murine liver after acute and chronic acetone administration.. Drug Metabolism and Disposition. 22(2). 248–253. 30 indexed citations

17.

Forkert, Poh‐Gek, et al.. (1994). In vitro biotransformation of 1,1-dichloroethylene by hepatic cytochrome P-450 2E1 in mice.. Journal of Pharmacology and Experimental Therapeutics. 270(1). 371–376. 20 indexed citations

18.

Moussa, Madeleine & Poh‐Gek Forkert. (1992). 1,1‐dichloroethylene‐induced alterations in glutathione and covalent binding in murine lung: Morphological, histochemical, and biochemical studies. The Journal of Pathology. 166(2). 199–207. 22 indexed citations

19.

Scott, J. E., et al.. (1988). Pulmonary toxicity of trichloroethylene: Induction of changes in surfactant phospholipids and phospholipase A2 activity in the mouse lung. Experimental and Molecular Pathology. 49(1). 141–150. 15 indexed citations

20.

Forkert, Poh‐Gek, et al.. (1988). Lung Volume Maintenance Prevents Lung Injury during High Frequency Oscillatory Ventilation in Surfactant-deficient Rabbits. American Review of Respiratory Disease. 137(5). 1185–1192. 285 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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