Philippe Dhordain

1.6k total citations
12 papers, 1.4k citations indexed

About

Philippe Dhordain is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Hematology. According to data from OpenAlex, Philippe Dhordain has authored 12 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 2 papers in Pathology and Forensic Medicine and 2 papers in Hematology. Recurrent topics in Philippe Dhordain's work include Cancer-related gene regulation (7 papers), Genomics and Chromatin Dynamics (4 papers) and Kruppel-like factors research (3 papers). Philippe Dhordain is often cited by papers focused on Cancer-related gene regulation (7 papers), Genomics and Chromatin Dynamics (4 papers) and Kruppel-like factors research (3 papers). Philippe Dhordain collaborates with scholars based in France, United Kingdom and United States. Philippe Dhordain's co-authors include Olivier Albagli, Dominique Leprince, Sabine Quief, Clotilde Deweindt, Jean‐Pierre Kerckaert, Danièle Lantoine, Ronald M. Evans, Richard J. Lin, Stéphane Ansieau and Achim Leutz and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Oncogene.

In The Last Decade

Philippe Dhordain

12 papers receiving 1.4k citations

Peers

Philippe Dhordain
Comparison fields: 5 of 76
  • Molecular Biology 1.0k
  • Immunology 321
  • Oncology 202
  • Genetics 161
  • Cancer Research 151
Replace Naoki Kakazu with:
Naoki Kakazu Japan
Anna Voronova United States
Tsuneyuki Oikawa Japan
Melanie W. Quong United States
Petra Dörfler Austria
Danièle Lantoine France
Christy J. Fryer United States
Nicola Hawe United States
K Toyoshima Japan
Joanna Liliental United States
Naoki Kakazu Japan View profile →
Citations per field, relative to Philippe Dhordain
Philippe Dhordain · 1×
Citations per year, relative to Philippe Dhordain
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Countries citing papers authored by Philippe Dhordain

Since Specialization
Citations

This map shows the geographic impact of Philippe Dhordain's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philippe Dhordain with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philippe Dhordain more than expected).

Fields of papers citing papers by Philippe Dhordain

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philippe Dhordain. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philippe Dhordain. The network helps show where Philippe Dhordain may publish in the future.

Co-authorship network of co-authors of Philippe Dhordain

This figure shows the co-authorship network connecting the top 25 collaborators of Philippe Dhordain. A scholar is included among the top collaborators of Philippe Dhordain based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philippe Dhordain. Philippe Dhordain is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
# Work Indexed citations
1 66
2 121
3 202
4 21
5 296
6 42
7 71
8
The BTB/POZ domain: a new protein-protein interaction motif common to DNA- and actin-binding proteins.
341
9
The LAZ3/BCL6 oncogene encodes a sequence-specific transcriptional inhibitor: a novel function for the BTB/POZ domain as an autonomous repressing domain.
130
10
The BTB/POZ domain targets the LAZ3/BCL6 oncoprotein to nuclear dots and mediates homomerisation in vivo.
102
11 11
12
A model for gene evolution of the ets-1/ets-2 transcription factors based on structural and functional homologies.
13

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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