Philip W. Askenase

945 total citations
34 papers, 737 citations indexed

About

Philip W. Askenase is a scholar working on Immunology, Physiology and Molecular Biology. According to data from OpenAlex, Philip W. Askenase has authored 34 papers receiving a total of 737 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Immunology, 11 papers in Physiology and 10 papers in Molecular Biology. Recurrent topics in Philip W. Askenase's work include Immune Cell Function and Interaction (12 papers), Asthma and respiratory diseases (10 papers) and Extracellular vesicles in disease (9 papers). Philip W. Askenase is often cited by papers focused on Immune Cell Function and Interaction (12 papers), Asthma and respiratory diseases (10 papers) and Extracellular vesicles in disease (9 papers). Philip W. Askenase collaborates with scholars based in United States, Poland and Japan. Philip W. Askenase's co-authors include Richard K. Gershon, Michael D. Gershon, Krzysztof Bryniarski, Katarzyna Nazimek, Włodzimierz Ptak, Ryohei Tsuji, Masanori Kikuchi, Robin Meade, Henk Van Loveren and Michel Dy and has published in prestigious journals such as The Journal of Experimental Medicine, The Journal of Immunology and International Journal of Molecular Sciences.

In The Last Decade

Philip W. Askenase

31 papers receiving 692 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Philip W. Askenase United States 16 431 207 133 104 92 34 737
Nadia Passini Italy 11 1.1k 2.5× 186 0.9× 156 1.2× 50 0.5× 41 0.4× 19 1.4k
Martin Ernst Germany 10 384 0.9× 215 1.0× 175 1.3× 123 1.2× 29 0.3× 12 921
Anne-Sophie Charbonnier France 13 816 1.9× 263 1.3× 347 2.6× 272 2.6× 132 1.4× 16 1.2k
Sonja Zahner United States 16 744 1.7× 209 1.0× 113 0.8× 63 0.6× 166 1.8× 18 1.0k
Mads Krogsgaard Thomsen Denmark 15 201 0.5× 126 0.6× 68 0.5× 62 0.6× 53 0.6× 28 552
Hilary Sandig United Kingdom 10 398 0.9× 263 1.3× 181 1.4× 101 1.0× 23 0.3× 11 777
Julian M. Stark Sweden 9 362 0.8× 137 0.7× 155 1.2× 60 0.6× 36 0.4× 13 574
Hendrik Jan de Heer Netherlands 7 868 2.0× 188 0.9× 411 3.1× 184 1.8× 69 0.8× 7 1.2k
Jayashree Dolpady United States 7 723 1.7× 388 1.9× 207 1.6× 148 1.4× 50 0.5× 7 1.2k
Daphne Tsitoura United Kingdom 15 409 0.9× 101 0.5× 408 3.1× 281 2.7× 76 0.8× 19 882

Countries citing papers authored by Philip W. Askenase

Since Specialization
Citations

This map shows the geographic impact of Philip W. Askenase's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip W. Askenase with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip W. Askenase more than expected).

Fields of papers citing papers by Philip W. Askenase

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip W. Askenase. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip W. Askenase. The network helps show where Philip W. Askenase may publish in the future.

Co-authorship network of co-authors of Philip W. Askenase

This figure shows the co-authorship network connecting the top 25 collaborators of Philip W. Askenase. A scholar is included among the top collaborators of Philip W. Askenase based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip W. Askenase. Philip W. Askenase is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Kawiková, Ivana, Václav Špička, James C. K. Lai, et al.. (2025). Extracellular vesicles as precision therapeutics for psychiatric conditions: targeting interactions among neuronal, glial, and immune networks. Frontiers in Immunology. 16. 1454306–1454306. 1 indexed citations
4.
Askenase, Philip W.. (2021). Ancient Evolutionary Origin and Properties of Universally Produced Natural Exosomes Contribute to Their Therapeutic Superiority Compared to Artificial Nanoparticles. International Journal of Molecular Sciences. 22(3). 1429–1429. 28 indexed citations
5.
Nazimek, Katarzyna, Bernadeta Nowak, Justyna Totoń‐Żurańska, et al.. (2021). Antibodies Enhance the Suppressive Activity of Extracellular Vesicles in Mouse Delayed-Type Hypersensitivity. Pharmaceuticals. 14(8). 734–734. 7 indexed citations
6.
Nazimek, Katarzyna, Krzysztof Bryniarski, Włodzimierz Ptak, Tom Groot Kormelink, & Philip W. Askenase. (2020). Orally Administered Exosomes Suppress Mouse Delayed-Type Hypersensitivity by Delivering miRNA-150 to Antigen-Primed Macrophage APC Targeted by Exosome-Surface Anti-Peptide Antibody Light Chains. International Journal of Molecular Sciences. 21(15). 5540–5540. 26 indexed citations
7.
Nazimek, Katarzyna, et al.. (2019). Delayed-Type Hypersensitivity Underlying Casein Allergy Is Suppressed by Extracellular Vesicles Carrying miRNA-150. Nutrients. 11(4). 907–907. 25 indexed citations
8.
Nazimek, Katarzyna, Bernadeta Nowak, Włodzimierz Ptak, et al.. (2019). Syngeneic red blood cell–induced extracellular vesicles suppress delayed‐type hypersensitivity to self‐antigens in mice. Clinical & Experimental Allergy. 49(11). 1487–1499. 17 indexed citations
9.
Nazimek, Katarzyna, Philip W. Askenase, & Krzysztof Bryniarski. (2018). Antibody Light Chains Dictate the Specificity of Contact Hypersensitivity Effector Cell Suppression Mediated by Exosomes. International Journal of Molecular Sciences. 19(9). 2656–2656. 15 indexed citations
10.
Askenase, Philip W.. (2015). Delayed-Type Hypersensitivity Recruitment of T Cell Subsets via Antigen-Specific Non-IgE Factors or IgE Antibodies: Relevance to Asthma, Autoimmunity and Immune Responses to Tumors and Parasites1. Chemical immunology/Fortschritte der Allergielehre/Progress in allergy/Chemical immunology and allergy. 54. 166–211.
11.
Askenase, Philip W., Monika Majewska‐Szczepanik, Steven M. Kerfoot, & Marian Szczepanik. (2011). Participation of Inkt Cells in the Early and Late Components of Tc1‐Mediated DNFB Contact Sensitivity: Cooperative Role of γδ‐T Cells. Scandinavian Journal of Immunology. 73(5). 465–477. 25 indexed citations
12.
Lorber, Marc I., Jean H. Wilson, Marie E. Robert, et al.. (1999). HUMAN ALLOGENEIC VASCULAR REJECTION AFTER ARTERIAL TRANSPLANTATION AND PERIPHERAL LYMPHOID RECONSTITUTION IN SEVERE COMBINED IMMUNODEFICIENT MICE1. Transplantation. 67(6). 897–903. 56 indexed citations
13.
Matsuda, Hiroshi, Włodzimierz Ptak, & Philip W. Askenase. (1995). Role of Mast Cells versus Basophils in IgE-Dependent Local Ear Skin Release of the Serotonin Required to Initiate Contact Sensitivity in Mice. International Archives of Allergy and Immunology. 107(1-3). 364–364. 5 indexed citations
14.
Matsuda, Hiroshi, Hiroko Ushio, Vipin Paliwal, W Ptak, & Philip W. Askenase. (1995). Adoptive cell transfer of contact sensitivity-initiation mediated by nonimmune cells sensitized with monoclonal IgE antibodies. Dependence on host skin mast cells.. The Journal of Immunology. 154(10). 5080–5092. 13 indexed citations
15.
Paliwal, Vipin, Alan M. Friedman, W Ptak, & Philip W. Askenase. (1994). Monoclonal, antigen-specific, T cell contrasuppressor factor expresses determinants of TCR alpha-chain (not necessarily TCR beta -chain), having a molecular mass of about 40 kDa.. The Journal of Immunology. 152(6). 2811–2820. 17 indexed citations
16.
Millet, Isabelle, Nicholas R. Ferreri, Rajani Ramabhadran, & Philip W. Askenase. (1992). IL-3 dependence of a Thy-1lo, B220+, IL-3 receptor-positive antigen-specific DTH-initiating clone. Cellular Immunology. 143(1). 154–169. 1 indexed citations
17.
Parmentier, H.K., et al.. (1989). Identification and Partial Characterization of a T-Cell-Derived Antigen-Binding Factor from Mice Infected with the Intestinal Helminth <i>Trichinella spiralis</i>. International Archives of Allergy and Immunology. 90(3). 237–247. 4 indexed citations
19.
Godfrey, Henry P., Mildred E. Phillips, & Philip W. Askenase. (1983). Histopathology of Delayed-Onset Hypersensitivities in Contact-Sensitive Guinea Pigs. International Archives of Allergy and Immunology. 70(1). 50–58. 8 indexed citations
20.
Askenase, Philip W., W Ptak, Robert W. Rosenstein, & R K Gershon. (1982). Transfer of an antigen specific immediate hypersensitivity-like reaction with an antigen binding factor produced by T cells. Journal of Allergy and Clinical Immunology. 69(1). 147–147. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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