Peter W. Halcrow

440 total citations
21 papers, 317 citations indexed

About

Peter W. Halcrow is a scholar working on Physiology, Cell Biology and Epidemiology. According to data from OpenAlex, Peter W. Halcrow has authored 21 papers receiving a total of 317 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Physiology, 10 papers in Cell Biology and 9 papers in Epidemiology. Recurrent topics in Peter W. Halcrow's work include Autophagy in Disease and Therapy (9 papers), Calcium signaling and nucleotide metabolism (9 papers) and HIV Research and Treatment (7 papers). Peter W. Halcrow is often cited by papers focused on Autophagy in Disease and Therapy (9 papers), Calcium signaling and nucleotide metabolism (9 papers) and HIV Research and Treatment (7 papers). Peter W. Halcrow collaborates with scholars based in United States, Netherlands and Saudi Arabia. Peter W. Halcrow's co-authors include Jonathan D. Geiger, Xuesong Chen, Nabab Khan, Zahra Afghah, Gaurav Datta, Joyce E. Ohm, Nicole Miller, Miranda L. Lynch, Nirmal Kumar and Olimpia Meucci and has published in prestigious journals such as Journal of Neuroscience, Scientific Reports and The FASEB Journal.

In The Last Decade

Peter W. Halcrow

20 papers receiving 317 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peter W. Halcrow United States 11 121 92 86 58 51 21 317
Piyali Saha India 11 163 1.3× 62 0.7× 56 0.7× 51 0.9× 70 1.4× 20 387
Judith Estévez‐Herrera Spain 13 194 1.6× 42 0.5× 58 0.7× 64 1.1× 79 1.5× 24 411
Alfred C. Chin United States 9 162 1.3× 12 0.1× 36 0.4× 62 1.1× 71 1.4× 19 323
Sonia Guha United States 11 215 1.8× 119 1.3× 112 1.3× 6 0.1× 66 1.3× 21 483
Honor L. Glenn United States 11 259 2.1× 29 0.3× 29 0.3× 10 0.2× 43 0.8× 26 412
Kanagaraj Subramanian United States 9 171 1.4× 49 0.5× 46 0.5× 14 0.2× 108 2.1× 11 308
Anoop Kumar G. Velikkakath India 5 183 1.5× 36 0.4× 223 2.6× 6 0.1× 118 2.3× 7 422
Qiaoxia Zheng China 6 146 1.2× 46 0.5× 100 1.2× 4 0.1× 86 1.7× 7 311
Joël Courageot France 12 164 1.4× 12 0.1× 35 0.4× 42 0.7× 86 1.7× 16 383
Eva Milanesi Italy 5 209 1.7× 24 0.3× 21 0.2× 21 0.4× 20 0.4× 6 282

Countries citing papers authored by Peter W. Halcrow

Since Specialization
Citations

This map shows the geographic impact of Peter W. Halcrow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter W. Halcrow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter W. Halcrow more than expected).

Fields of papers citing papers by Peter W. Halcrow

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter W. Halcrow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter W. Halcrow. The network helps show where Peter W. Halcrow may publish in the future.

Co-authorship network of co-authors of Peter W. Halcrow

This figure shows the co-authorship network connecting the top 25 collaborators of Peter W. Halcrow. A scholar is included among the top collaborators of Peter W. Halcrow based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter W. Halcrow. Peter W. Halcrow is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Halcrow, Peter W., et al.. (2024). HERV-K (HML-2) Envelope Protein Induces Mitochondrial Depolarization and Neurotoxicity via Endolysosome Iron Dyshomeostasis. Journal of Neuroscience. 44(14). e0826232024–e0826232024. 6 indexed citations
4.
Afghah, Zahra, et al.. (2024). Involvement of Endolysosomes and Aurora Kinase A in the Regulation of Amyloid β Protein Levels in Neurons. International Journal of Molecular Sciences. 25(11). 6200–6200. 2 indexed citations
5.
Halcrow, Peter W., et al.. (2023). Amyloid Beta Protein (Aβ1-42) induced Endolysosome Iron Dyshomeostasis caused increases in Reactive Oxygen Species, Mitochondrial Depolarization, and Neurotoxicity. Journal of Pharmacology and Experimental Therapeutics. 385. 130–130. 2 indexed citations
6.
Halcrow, Peter W., Nirmal Kumar, Zahra Afghah, et al.. (2022). Heterogeneity of ferrous iron‐containing endolysosomes and effects of endolysosome iron on endolysosome numbers, sizes, and localization patterns. Journal of Neurochemistry. 161(1). 69–83. 12 indexed citations
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Khan, Nabab, et al.. (2022). HIV‐1 Tat endocytosis and retention in endolysosomes affects HIV‐1 Tat‐induced LTR transactivation in astrocytes. The FASEB Journal. 36(3). e22184–e22184. 11 indexed citations
10.
Halcrow, Peter W., Miranda L. Lynch, Jonathan D. Geiger, & Joyce E. Ohm. (2021). Role of endolysosome function in iron metabolism and brain carcinogenesis. Seminars in Cancer Biology. 76. 74–85. 29 indexed citations
11.
Halcrow, Peter W., Jonathan D. Geiger, & Xuesong Chen. (2021). Overcoming Chemoresistance: Altering pH of Cellular Compartments by Chloroquine and Hydroxychloroquine. Frontiers in Cell and Developmental Biology. 9. 627639–627639. 45 indexed citations
12.
Halcrow, Peter W., Nabab Khan, Zahra Afghah, et al.. (2021). HIV-1 gp120-Induced Endolysosome de-Acidification Leads to Efflux of Endolysosome Iron, and Increases in Mitochondrial Iron and Reactive Oxygen Species. Journal of Neuroimmune Pharmacology. 17(1-2). 181–194. 30 indexed citations
13.
Khan, Nabab, et al.. (2021). Endolysosome iron restricts Tat-mediated HIV-1 LTR transactivation by increasing HIV-1 Tat oligomerization and β-catenin expression. Journal of NeuroVirology. 27(5). 755–773. 5 indexed citations
14.
Datta, Gaurav, Nicole Miller, Peter W. Halcrow, et al.. (2021). SARS-CoV-2 S1 Protein Induces Endolysosome Dysfunction and Neuritic Dystrophy. Frontiers in Cellular Neuroscience. 15. 10 indexed citations
15.
Khan, Nabab, Peter W. Halcrow, Zahra Afghah, et al.. (2020). Two‐pore channels regulate Tat endolysosome escape and Tat‐mediated HIV‐1 LTR transactivation. The FASEB Journal. 34(3). 4147–4162. 34 indexed citations
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Khan, Nabab, et al.. (2019). BK channels regulate extracellular Tat-mediated HIV-1 LTR transactivation. Scientific Reports. 9(1). 12285–12285. 29 indexed citations
17.
Halcrow, Peter W., et al.. (2019). Readily Releasable Stores of Calcium in Neuronal Endolysosomes: Physiological and Pathophysiological Relevance. Advances in experimental medicine and biology. 1131. 681–697. 9 indexed citations
18.
Irollo, Elena, et al.. (2019). Morphine-Induced Modulation of Endolysosomal Iron Mediates Upregulation of Ferritin Heavy Chain in Cortical Neurons. eNeuro. 6(4). ENEURO.0237–19.2019. 21 indexed citations
19.
Halcrow, Peter W., Nabab Khan, Gaurav Datta, et al.. (2019). Importance of measuring endolysosome, cytosolic, and extracellular pH in understanding the pathogenesis of and possible treatments for glioblastoma multiforme. Cancer Reports. 2(6). 24 indexed citations
20.
Halcrow, Peter W., et al.. (2016). Molecular Changes Associated With Tumor Initiation and Progression of Soft Tissue Sarcomas. Progress in molecular biology and translational science. 144. 323–380. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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