Perry M. Kim

1.0k total citations
13 papers, 822 citations indexed

About

Perry M. Kim is a scholar working on Cancer Research, Molecular Biology and Pediatrics, Perinatology and Child Health. According to data from OpenAlex, Perry M. Kim has authored 13 papers receiving a total of 822 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Cancer Research, 8 papers in Molecular Biology and 2 papers in Pediatrics, Perinatology and Child Health. Recurrent topics in Perry M. Kim's work include Carcinogens and Genotoxicity Assessment (8 papers), DNA Repair Mechanisms (5 papers) and Pluripotent Stem Cells Research (2 papers). Perry M. Kim is often cited by papers focused on Carcinogens and Genotoxicity Assessment (8 papers), DNA Repair Mechanisms (5 papers) and Pluripotent Stem Cells Research (2 papers). Perry M. Kim collaborates with scholars based in Canada and United States. Perry M. Kim's co-authors include Peter G. Wells, Louise M. Winn, Rebecca R. Laposa, Christopher J.B. Nicol, Jac A. Nickoloff, Lei Huang, William F. Morgan, Peter I. Mackenzie, Fay K. Kessler and Antony J. Hansen and has published in prestigious journals such as Nucleic Acids Research, Molecular and Cellular Biology and Cancer Research.

In The Last Decade

Perry M. Kim

13 papers receiving 805 citations

Peers

Perry M. Kim
Agneta Blanck Sweden
Y. Barra France
Gabriela Balogh United States
Joshua C. Kwekel United States
Leena Strauss Finland
Ranjit K. Thirumaran United States
Tadashi Ogishima Japan
Matthew J. van Zwieten United States
Stephanie J. Webb United States
Agneta Blanck Sweden
Perry M. Kim
Citations per year, relative to Perry M. Kim Perry M. Kim (= 1×) peers Agneta Blanck

Countries citing papers authored by Perry M. Kim

Since Specialization
Citations

This map shows the geographic impact of Perry M. Kim's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Perry M. Kim with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Perry M. Kim more than expected).

Fields of papers citing papers by Perry M. Kim

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Perry M. Kim. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Perry M. Kim. The network helps show where Perry M. Kim may publish in the future.

Co-authorship network of co-authors of Perry M. Kim

This figure shows the co-authorship network connecting the top 25 collaborators of Perry M. Kim. A scholar is included among the top collaborators of Perry M. Kim based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Perry M. Kim. Perry M. Kim is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Huang, Lei, Perry M. Kim, Jac A. Nickoloff, & William F. Morgan. (2007). Targeted and Nontargeted Effects of Low-Dose Ionizing Radiation on Delayed Genomic Instability in Human Cells. Cancer Research. 67(3). 1099–1104. 70 indexed citations
2.
3.
Huang, Lei, Leslie E. Smith, Perry M. Kim, et al.. (2004). Ionizing Radiation Induces Delayed Hyperrecombination in Mammalian Cells. Molecular and Cellular Biology. 24(11). 5060–5068. 38 indexed citations
4.
Wells, Peter G., Peter I. Mackenzie, Jayanta Roy Chowdhury, et al.. (2004). GLUCURONIDATION AND THE UDP-GLUCURONOSYLTRANSFERASES IN HEALTH AND DISEASE. Drug Metabolism and Disposition. 32(3). 281–290. 186 indexed citations
5.
Kim, Perry M., et al.. (2004). TCDD-induced homologous recombination: the role of the Ah receptor versus oxidative DNA damage. Mutation Research/Genetic Toxicology and Environmental Mutagenesis. 563(1). 71–79. 12 indexed citations
6.
Winn, Louise M., Perry M. Kim, & Jac A. Nickoloff. (2003). Oxidative Stress-Induced Homologous Recombination As a Novel Mechanism for Phenytoin-Initiated Toxicity. Journal of Pharmacology and Experimental Therapeutics. 306(2). 523–527. 29 indexed citations
7.
8.
Kim, Perry M.. (2001). Overexpression of human RAD51 and RAD52 reduces double-strand break-induced homologous recombination in mammalian cells. Nucleic Acids Research. 29(21). 4352–4360. 85 indexed citations
9.
Winn, Louise M., Perry M. Kim, & Peter G. Wells. (1998). Investigation of the Tobacco-Specific Carcinogen 4-(Methylnitrosamino)-1-(3-Pyridyl)-1-Butanone for In Vivo and In Vitro Murine Embryopathy and Embryonic ras Mutations. Journal of Pharmacology and Experimental Therapeutics. 287(3). 1128–1135. 4 indexed citations
10.
Winn, Louise M., Perry M. Kim, & Peter G. Wells. (1998). Investigation of the tobacco-specific carcinogen 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone for in vivo and iIn vitro murine embryopathy and embryonic ras mutations.. PubMed. 287(3). 1128–35. 6 indexed citations
11.
Wells, Peter G., et al.. (1997). Oxidative damage in chemical teratogenesis. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 396(1-2). 65–78. 211 indexed citations
12.
Kim, Perry M., et al.. (1997). Peroxidase-Dependent Bioactivation and Oxidation of DNA and Protein in Benzo[a]Pyrene-Initiated Micronucleus Formation. Free Radical Biology and Medicine. 23(4). 579–596. 35 indexed citations
13.
Kim, Perry M. & Peter G. Wells. (1996). Genoprotection by UDP-glucuronosyltransferases in peroxidase-dependent, reactive oxygen species-mediated micronucleus initiation by the carcinogens 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone and benzo[a]pyrene.. PubMed. 56(7). 1526–32. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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