Per Höllsberg

3.5k total citations
72 papers, 2.6k citations indexed

About

Per Höllsberg is a scholar working on Immunology, Oncology and Epidemiology. According to data from OpenAlex, Per Höllsberg has authored 72 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Immunology, 26 papers in Oncology and 25 papers in Epidemiology. Recurrent topics in Per Höllsberg's work include Cytomegalovirus and herpesvirus research (23 papers), Viral-associated cancers and disorders (19 papers) and T-cell and Retrovirus Studies (18 papers). Per Höllsberg is often cited by papers focused on Cytomegalovirus and herpesvirus research (23 papers), Viral-associated cancers and disorders (19 papers) and T-cell and Retrovirus Studies (18 papers). Per Höllsberg collaborates with scholars based in Denmark, United States and United Kingdom. Per Höllsberg's co-authors include David A. Hafler, S. Haahr, D A Hafler, Jeffrey S. Flier, Lisa H. Underhill, Bodil Øster, Anja Windhagen, H. J. Hansen, Emil Kofod‐Olsen and D Hafler and has published in prestigious journals such as New England Journal of Medicine, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Per Höllsberg

72 papers receiving 2.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Per Höllsberg Denmark 30 1.7k 568 557 505 482 72 2.6k
Shino Hanabuchi United States 33 3.8k 2.3× 326 0.6× 454 0.8× 748 1.5× 448 0.9× 50 5.1k
Christophe Pannetier France 33 3.9k 2.3× 528 0.9× 221 0.4× 967 1.9× 213 0.4× 55 5.1k
Isao Miyoshi Japan 26 3.1k 1.8× 486 0.9× 2.2k 4.0× 729 1.4× 2.1k 4.3× 144 4.5k
Keiko Nagata Japan 21 2.4k 1.4× 198 0.3× 1.6k 2.9× 659 1.3× 1.6k 3.3× 75 3.5k
Kimitaka Sagawa Japan 24 4.1k 2.4× 253 0.4× 2.1k 3.8× 448 0.9× 2.2k 4.6× 78 5.0k
D A Hafler United States 21 1.8k 1.1× 171 0.3× 133 0.2× 343 0.7× 116 0.2× 29 2.5k
Louis Gazzolo France 29 1.4k 0.8× 356 0.6× 809 1.5× 192 0.4× 755 1.6× 101 2.5k
Heiko Adler Germany 32 1.3k 0.8× 1.4k 2.5× 162 0.3× 877 1.7× 134 0.3× 75 3.1k
Mineki Saito Japan 23 1.4k 0.8× 226 0.4× 980 1.8× 63 0.1× 926 1.9× 98 2.0k
Miriam Siekevitz United States 16 1.9k 1.1× 205 0.4× 606 1.1× 211 0.4× 555 1.2× 22 2.6k

Countries citing papers authored by Per Höllsberg

Since Specialization
Citations

This map shows the geographic impact of Per Höllsberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Per Höllsberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Per Höllsberg more than expected).

Fields of papers citing papers by Per Höllsberg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Per Höllsberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Per Höllsberg. The network helps show where Per Höllsberg may publish in the future.

Co-authorship network of co-authors of Per Höllsberg

This figure shows the co-authorship network connecting the top 25 collaborators of Per Höllsberg. A scholar is included among the top collaborators of Per Höllsberg based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Per Höllsberg. Per Höllsberg is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schleimann, Mariane Høgsbjerg, et al.. (2014). The DR6 protein from human herpesvirus-6B induces p53-independent cell cycle arrest in G2/M. Virology. 452-453. 254–263. 7 indexed citations
2.
Höllsberg, Per, et al.. (2013). Synergy between vitamin D3 and toll-like receptor agonists regulates human dendritic cell response during maturation. 4 indexed citations
3.
Höllsberg, Per, et al.. (2013). Seminal shedding of human herpesviruses. Virology Journal. 10(1). 226–226. 23 indexed citations
4.
Höllsberg, Per, et al.. (2013). Synergy between Vitamin D3and Toll-Like Receptor Agonists Regulates Human Dendritic Cell Response during Maturation. SHILAP Revista de lepidopterología. 2013. 1–8. 34 indexed citations
5.
Kofod‐Olsen, Emil, et al.. (2012). Human Herpesvirus-6A/B Binds to Spermatozoa Acrosome and Is the Most Prevalent Herpesvirus in Semen from Sperm Donors. PLoS ONE. 7(11). e48810–e48810. 25 indexed citations
6.
Bjerg, Lasse, et al.. (2012). Altered frequency of T regulatory cells is associated with disability status in relapsing–remitting multiple sclerosis patients. Journal of Neuroimmunology. 249(1-2). 76–82. 33 indexed citations
7.
Höllsberg, Per, et al.. (2009). Human herpesvirus-6B induces expression of the human endogenous retrovirus K18-encoded superantigen. Journal of Clinical Virology. 46(1). 15–19. 51 indexed citations
8.
Hvas, Christian Lodberg, Jens Kelsen, Jørgen Agnholt, et al.. (2007). Crohn's disease intestinal CD4+ T cells have impaired interleukin-10 production which is not restored by probiotic bacteria. Scandinavian Journal of Gastroenterology. 42(5). 592–601. 15 indexed citations
9.
Øster, Bodil, et al.. (2006). Human herpesvirus 6B inhibits cell proliferation by a p53-independent pathway. Journal of Clinical Virology. 37. S63–S68. 14 indexed citations
10.
11.
Øster, Bodil & Per Höllsberg. (2002). A sensitive quantification of HHV-6B by real-time PCR. Biological Procedures Online. 4(1). 88–93. 7 indexed citations
12.
Bieganowska, Katarzyna, Per Höllsberg, Guy J. Buckle, et al.. (1999). Direct Analysis of Viral-Specific CD8+ T Cells with Soluble HLA-A2/Tax11-19 Tetramer Complexes in Patients with Human T Cell Lymphotropic Virus-Associated Myelopathy. The Journal of Immunology. 162(3). 1765–1771. 114 indexed citations
13.
Greenfield, Edward, Edward D. Howard, Timothy Paradis, et al.. (1997). B7.2 expressed by T cells does not induce CD28-mediated costimulatory activity but retains CTLA4 binding: implications for induction of antitumor immunity to T cell tumors. The Journal of Immunology. 158(5). 2025–2034. 70 indexed citations
14.
Höllsberg, Per, Christian Schölz, David E. Anderson, et al.. (1997). Expression of a hypoglycosylated form of CD86 (B7-2) on human T cells with altered binding properties to CD28 and CTLA-4. The Journal of Immunology. 159(10). 4799–4805. 56 indexed citations
15.
Anderson, David E., et al.. (1997). Weak peptide agonists reveal functional differences in B7-1 and B7-2 costimulation of human T cell clones. The Journal of Immunology. 159(4). 1669–1675. 20 indexed citations
16.
Höllsberg, Per. (1997). Pathogenesis of chronic progressive myelopathy associated with human T-cell lymphotropic virus type I. Acta Neurologica Scandinavica. 95. 86–93. 25 indexed citations
17.
Schölz, Christian, G J Freeman, Edward Greenfield, D A Hafler, & Per Höllsberg. (1996). Activation of human T cell lymphotropic virus type I-infected T cells is independent of B7 costimulation. The Journal of Immunology. 157(7). 2932–2938. 10 indexed citations
18.
Zoubak, Serguei, Jennifer Richardson, A. V. Rynditch, et al.. (1994). Regional specificity of HTLV-I pro viral integration in the human genome. Gene. 143(2). 155–163. 34 indexed citations
19.
Flier, Jeffrey S., Lisa H. Underhill, Per Höllsberg, & David A. Hafler. (1993). Pathogenesis of Diseases Induced by Human Lymphotropic Virus Type I Infection. New England Journal of Medicine. 328(16). 1173–1182. 259 indexed citations
20.
Höllsberg, Per, et al.. (1989). Search for a retrovirus in long-term cultured cerebrospinal fluid cells and peripheral blood mononuclear cells from patients with multiple sclerosis. Acta Neurologica Scandinavica. 80(6). 603–609. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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