Peili Gu

2.8k total citations
26 papers, 1.8k citations indexed

About

Peili Gu is a scholar working on Molecular Biology, Physiology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Peili Gu has authored 26 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 10 papers in Physiology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Peili Gu's work include Telomeres, Telomerase, and Senescence (10 papers), CRISPR and Genetic Engineering (9 papers) and DNA Repair Mechanisms (8 papers). Peili Gu is often cited by papers focused on Telomeres, Telomerase, and Senescence (10 papers), CRISPR and Genetic Engineering (9 papers) and DNA Repair Mechanisms (8 papers). Peili Gu collaborates with scholars based in United States, China and Hong Kong. Peili Gu's co-authors include Austin J. Cooney, Sandy Chang, Arthur Chung, David A. Wheeler, Ka Hou Chu, Xueping Xu, Siu‐Ming Chan, Damien Le Menuet, Jun Qin and Ma Wan and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Peili Gu

26 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Peili Gu United States 22 1.4k 423 371 267 194 26 1.8k
Ursula Weber United States 25 2.2k 1.5× 129 0.3× 509 1.4× 336 1.3× 264 1.4× 31 2.7k
Bernard F. Andruss United States 14 701 0.5× 79 0.2× 325 0.9× 197 0.7× 151 0.8× 23 1.5k
Stephen J. Duguay United States 16 603 0.4× 70 0.2× 115 0.3× 437 1.6× 98 0.5× 22 1.6k
Martine Simonelig France 29 2.4k 1.7× 43 0.1× 181 0.5× 296 1.1× 125 0.6× 53 2.8k
Yuanjun Zhao China 19 728 0.5× 231 0.5× 31 0.1× 96 0.4× 185 1.0× 79 1.2k
H.C. Ardley United Kingdom 17 939 0.7× 137 0.3× 160 0.4× 157 0.6× 111 0.6× 28 1.3k
Peizhang Xu United States 8 1.2k 0.9× 55 0.1× 235 0.6× 89 0.3× 135 0.7× 8 1.6k
Julia B. Cordero United Kingdom 22 892 0.6× 74 0.2× 393 1.1× 174 0.7× 587 3.0× 32 1.8k
Cordula Schulz United States 18 1.3k 0.9× 90 0.2× 390 1.1× 435 1.6× 340 1.8× 25 1.8k

Countries citing papers authored by Peili Gu

Since Specialization
Citations

This map shows the geographic impact of Peili Gu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peili Gu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peili Gu more than expected).

Fields of papers citing papers by Peili Gu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peili Gu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peili Gu. The network helps show where Peili Gu may publish in the future.

Co-authorship network of co-authors of Peili Gu

This figure shows the co-authorship network connecting the top 25 collaborators of Peili Gu. A scholar is included among the top collaborators of Peili Gu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peili Gu. Peili Gu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cai, Tiantian, et al.. (2024). Polymorphisms of B-lymphocyte-associated genes CD20 and FCRL5 are associated with susceptibility to autoimmune thyroid diseases. Human Immunology. 85(6). 111165–111165. 1 indexed citations
2.
Gu, Peili, Shuting Jia, Valerie M. Tesmer, et al.. (2021). Distinct functions of POT1 proteins contribute to the regulation of telomerase recruitment to telomeres. Nature Communications. 12(1). 5514–5514. 27 indexed citations
3.
Rai, Rekha, et al.. (2019). The Replisome Mediates A-NHEJ Repair of Telomeres Lacking POT1-TPP1 Independently of MRN Function. Cell Reports. 29(11). 3708–3725.e5. 11 indexed citations
4.
Chen, Cong, Peili Gu, Jian Wu, et al.. (2017). Structural insights into POT1-TPP1 interaction and POT1 C-terminal mutations in human cancer. Nature Communications. 8(1). 14929–14929. 75 indexed citations
5.
6.
Gu, Peili, Wei Deng, Ming Lei, & Sandy Chang. (2013). Single strand DNA binding proteins 1 and 2 protect newly replicated telomeres. Cell Research. 23(5). 705–719. 27 indexed citations
7.
Wan, Bingbing, Jinhu Yin, Kent Horvath, et al.. (2013). SLX4 Assembles a Telomere Maintenance Toolkit by Bridging Multiple Endonucleases with Telomeres. Cell Reports. 4(5). 861–869. 96 indexed citations
8.
Gu, Peili, Jin‐Na Min, Yang Wang, et al.. (2012). CTC1 deletion results in defective telomere replication, leading to catastrophic telomere loss and stem cell exhaustion. The EMBO Journal. 31(10). 2309–2321. 128 indexed citations
9.
Xu, Xuping, Peili Gu, David M. Lonard, et al.. (2011). miRNA Regulatory Circuits in ES Cells Differentiation: A Chemical Kinetics Modeling Approach. PLoS ONE. 6(10). e23263–e23263. 4 indexed citations
10.
Lam, Yung C., Shamima Akhter, Peili Gu, et al.. (2010). SNMIB/Apollo protects leading‐strand telomeres against NHEJ‐mediated repair. The EMBO Journal. 29(13). 2230–2241. 98 indexed citations
11.
Gu, Peili, Jeffrey G. Reid, Xiaolian Gao, et al.. (2008). Novel MicroRNA Candidates and miRNA-mRNA Pairs in Embryonic Stem (ES) Cells. PLoS ONE. 3(7). e2548–e2548. 42 indexed citations
12.
Gu, Peili, et al.. (2007). Nuclear Receptors in Regulation of Mouse ES Cell Pluripotency and Differentiation. PPAR Research. 2007. 1–10. 44 indexed citations
13.
Raviscioni, Michele, et al.. (2005). Correlated Evolutionary Pressure at Interacting Transcription Factors and DNA Response Elements Can Guide the Rational Engineering of DNA Binding Specificity. Journal of Molecular Biology. 350(3). 402–415. 22 indexed citations
14.
Gu, Peili, Xueping Xu, Ryan T. Wagner, et al.. (2005). Evolutionary Trace-based Peptides Identify a Novel Asymmetric Interaction That Mediates Oligomerization in Nuclear Receptors. Journal of Biological Chemistry. 280(36). 31818–31829. 27 indexed citations
15.
Gu, Peili, Bryan Goodwin, Arthur Chung, et al.. (2005). Orphan Nuclear Receptor LRH-1 Is Required To Maintain Oct4 Expression at the Epiblast Stage of Embryonic Development. Molecular and Cellular Biology. 25(9). 3492–3505. 244 indexed citations
16.
Chan, Siu‐Ming, Peili Gu, Ka Hou Chu, & Stephen S. Tobe. (2003). Crustacean neuropeptide genes of the CHH/MIH/GIH family: implications from molecular studies. General and Comparative Endocrinology. 134(3). 214–219. 126 indexed citations
17.
Lan, Zi‐Jian, Peili Gu, Xueping Xu, & Austin J. Cooney. (2003). Expression of the Orphan Nuclear Receptor, Germ Cell Nuclear Factor, in Mouse Gonads and Preimplantation Embryos1. Biology of Reproduction. 68(1). 282–289. 40 indexed citations
18.
Gu, Peili, Y.I.N.S. Gunawardene, Bkc Chow, Jian He, & Siu‐Ming Chan. (2002). Characterization of a novel cellular retinoic acid/retinol binding protein from shrimp: expression of the recombinant protein for immunohistochemical detection and binding assay. Gene. 288(1-2). 77–84. 24 indexed citations
19.
20.
Chen, Xiao‐Guang, et al.. (1998). PCR cloning and expression of the molt-inhibiting hormone gene for the crab (Charybdis feriatus). Gene. 224(1-2). 23–33. 51 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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