Pedro P. Costa

414 total citations
12 papers, 337 citations indexed

About

Pedro P. Costa is a scholar working on Molecular Biology, Cell Biology and Oncology. According to data from OpenAlex, Pedro P. Costa has authored 12 papers receiving a total of 337 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 6 papers in Cell Biology and 4 papers in Oncology. Recurrent topics in Pedro P. Costa's work include Amyloidosis: Diagnosis, Treatment, Outcomes (11 papers), Protein Kinase Regulation and GTPase Signaling (4 papers) and Cellular transport and secretion (4 papers). Pedro P. Costa is often cited by papers focused on Amyloidosis: Diagnosis, Treatment, Outcomes (11 papers), Protein Kinase Regulation and GTPase Signaling (4 papers) and Cellular transport and secretion (4 papers). Pedro P. Costa collaborates with scholars based in Portugal, United States and Japan. Pedro P. Costa's co-authors include Maria João Saraiva, DeWitt S. Goodman, Hirokazu Furuya, Yoshiyuki Sakaki, Hiroyuki Sasaki, Steven Birken, Katsuji Yoshioka, Ikuo Goto, Mary Ann Gawinowicz and Isabel L. Alves and has published in prestigious journals such as Neurology, Biochemistry and Biochemical and Biophysical Research Communications.

In The Last Decade

Pedro P. Costa

12 papers receiving 326 citations

Peers

Pedro P. Costa
H M Kronenberg United States
S. Okubo Japan
Shen Kiat Lim Singapore
Jibin Zeng Canada
Amy J. Lambert United States
Weiwen Xie United States
Ulf Schulze Germany
Pedro P. Costa
Citations per year, relative to Pedro P. Costa Pedro P. Costa (= 1×) peers Thibault Lalu

Countries citing papers authored by Pedro P. Costa

Since Specialization
Citations

This map shows the geographic impact of Pedro P. Costa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pedro P. Costa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pedro P. Costa more than expected).

Fields of papers citing papers by Pedro P. Costa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pedro P. Costa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pedro P. Costa. The network helps show where Pedro P. Costa may publish in the future.

Co-authorship network of co-authors of Pedro P. Costa

This figure shows the co-authorship network connecting the top 25 collaborators of Pedro P. Costa. A scholar is included among the top collaborators of Pedro P. Costa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pedro P. Costa. Pedro P. Costa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Munar-Qués, Miguel, et al.. (1997). Familial amyloidotic polyneuropathy. TTR Met 30 in Majorca (Spain). Amyloid. 4(3). 181–186. 10 indexed citations
2.
Almeida, Maria Rosário, et al.. (1993). Transthyretin ALA 71: A new transthyretin variant in a Spanish family with familial amyloidotic polyneuropathy. Human Mutation. 2(5). 420–421. 14 indexed citations
3.
Almeida, Maria Rosário, Alessandra Ferlini, Antonino Forabosco, et al.. (1992). Two transthyretin variants (TTR Ala-49 and TTR Gln-89) in two sicilian kindreds with hereditary amyloidosis. Human Mutation. 1(3). 211–215. 36 indexed citations
4.
Furuya, Hirokazu, Maria João Saraiva, Mary Ann Gawinowicz, et al.. (1991). Production of recombinant human transthyretin with biological activities toward the understanding of the molecular basis of familial amyloidotic polyneuropathy (FAP). Biochemistry. 30(9). 2415–2421. 74 indexed citations
5.
Altland, Klaus, et al.. (1991). Characterization of a basic transthyretin variant - TTR Arg 102 - in the German population. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1097(3). 224–226. 15 indexed citations
6.
Yoshioka, Katsuji, Hirokazu Furuya, Hiroyuki Sasaki, et al.. (1989). Haplotype analysis of familial amyloidotic polyneuropathy. Human Genetics. 82(1). 9–13. 69 indexed citations
7.
Furuya, Hirokazu, Masamitsu Nakazato, Maria João Saraiva, et al.. (1989). Tetramer formation of a variant type human transthyretin (prealbumin) produced byEscherichia coli expression system. Biochemical and Biophysical Research Communications. 163(2). 851–859. 11 indexed citations
8.
Saraiva, Maria João, Pedro P. Costa, Maria Rosário Almeida, et al.. (1988). Familial amyloidotic polyneuropathy: transthyretin (prealbumin) variants in kindreds of Italian origin. Human Genetics. 80(4). 341–343. 8 indexed citations
9.
Costa, Pedro P. & Maria João Saraiva. (1988). Familial Amyloidotic Polyneuropathy: Screening of the Population at Risk in Portugal. 425–428. 3 indexed citations
10.
Leite, Isabel Cristina Gonçalves, et al.. (1988). Familial Amyloid Polyneuropathy (Portuguese Type): Study of Asymptomatic Carriers. 429–434. 1 indexed citations
11.
Saraiva, Maria João, Pedro P. Costa, & DeWitt S. Goodman. (1986). Genetic expression of a transthyretin mutation in typical and late‐onset Portuguese families with familial amyloidotic polyneuropathy. Neurology. 36(11). 1413–1413. 48 indexed citations
12.
Saraiva, Maria João, Steven Birken, Pedro P. Costa, & DeWitt S. Goodman. (1984). Family Studies of the Genetic Abnormality in Transthyretin (Prealbumin) in Portuguese Patients with Familial Amyloidotic Poly neuropathya. Annals of the New York Academy of Sciences. 435(1). 86–100. 48 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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