Pedro Aparício

2.0k total citations
46 papers, 1.6k citations indexed

About

Pedro Aparício is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Pedro Aparício has authored 46 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Immunology, 9 papers in Molecular Biology and 9 papers in Oncology. Recurrent topics in Pedro Aparício's work include T-cell and B-cell Immunology (27 papers), Immune Cell Function and Interaction (20 papers) and Immunotherapy and Immune Responses (9 papers). Pedro Aparício is often cited by papers focused on T-cell and B-cell Immunology (27 papers), Immune Cell Function and Interaction (20 papers) and Immunotherapy and Immune Responses (9 papers). Pedro Aparício collaborates with scholars based in Spain, France and United States. Pedro Aparício's co-authors include José A. Łópez de Castro, Pascual Parrilla, Miguel López‐Botet, Trinidad Hernández‐Caselles, Francisco Sánchez‐Madrid, Carlos Martı́nez-A, Alberto Baroja‐Mazo, Dolores Jaraquemada, José Yélamos and Gonzalo Rubio and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Pedro Aparício

46 papers receiving 1.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pedro Aparício Spain 25 1.0k 386 380 144 143 46 1.6k
G J Freeman United States 21 1.9k 1.9× 485 1.3× 443 1.2× 183 1.3× 197 1.4× 25 2.6k
Y. Nishimura Japan 24 941 0.9× 286 0.7× 483 1.3× 132 0.9× 132 0.9× 43 1.9k
Patricia J. Noel United States 12 2.1k 2.0× 637 1.7× 428 1.1× 92 0.6× 84 0.6× 14 2.5k
Marylou G. Gibson United States 6 1.3k 1.3× 253 0.7× 229 0.6× 144 1.0× 190 1.3× 8 1.9k
H Yagita Japan 17 706 0.7× 148 0.4× 303 0.8× 130 0.9× 107 0.7× 26 1.1k
Janet E. Buhlmann United States 13 1.4k 1.4× 473 1.2× 342 0.9× 85 0.6× 75 0.5× 21 2.0k
Claude Gimmi United States 14 1.4k 1.4× 450 1.2× 407 1.1× 172 1.2× 234 1.6× 30 1.9k
Dass S. Vinay United States 24 1.7k 1.7× 797 2.1× 302 0.8× 77 0.5× 96 0.7× 44 2.3k
Georg Tiefenthaler Germany 18 983 1.0× 225 0.6× 400 1.1× 153 1.1× 404 2.8× 29 1.6k
Lesley Rhee United States 11 2.9k 2.9× 424 1.1× 291 0.8× 116 0.8× 156 1.1× 13 3.5k

Countries citing papers authored by Pedro Aparício

Since Specialization
Citations

This map shows the geographic impact of Pedro Aparício's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pedro Aparício with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pedro Aparício more than expected).

Fields of papers citing papers by Pedro Aparício

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pedro Aparício. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pedro Aparício. The network helps show where Pedro Aparício may publish in the future.

Co-authorship network of co-authors of Pedro Aparício

This figure shows the co-authorship network connecting the top 25 collaborators of Pedro Aparício. A scholar is included among the top collaborators of Pedro Aparício based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pedro Aparício. Pedro Aparício is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Bedora-Faure, Marie, Chloé Lescale, Carlos Martínez, et al.. (2019). Coordinated signals from the DNA repair enzymes PARP-1 and PARP-2 promotes B-cell development and function. Cell Death and Differentiation. 26(12). 2667–2681. 34 indexed citations
2.
Baroja‐Mazo, Alberto, Beatriz Revilla‐Nuin, L. Martínez‐Alarcón, et al.. (2018). Extracellular adenosine reversibly inhibits the activation of human regulatory T cells and negatively influences the achievement of the operational tolerance in liver transplantation. American Journal of Transplantation. 19(1). 48–61. 22 indexed citations
3.
Martínez, Carlos, Cristina Baró, Alberto Baroja‐Mazo, et al.. (2010). Loss of poly(ADP-ribose) polymerase-2 leads to rapid development of spontaneous T-cell lymphomas in p53-deficient mice. Oncogene. 29(19). 2877–2883. 48 indexed citations
4.
Pons, J.A., Beatriz Revilla‐Nuin, Alberto Baroja‐Mazo, et al.. (2008). FoxP3 in Peripheral Blood Is Associated With Operational Tolerance in Liver Transplant Patients During Immunosuppression Withdrawal. Transplantation. 86(10). 1370–1378. 142 indexed citations
5.
Aguado, Enrique, Mario Martínez‐Florensa, & Pedro Aparício. (2006). Activation of T lymphocytes and the role of the adapter LAT. Transplant Immunology. 17(1). 23–26. 15 indexed citations
6.
Yélamos, José, A.J. Buendía, Nieves Ortega, et al.. (2004). Genetic and pharmacological inhibition of poly(ADP-ribose) polymerase-1 interferes in the chlamydial life cycle. Biochemical and Biophysical Research Communications. 324(2). 840–848. 3 indexed citations
7.
Caparrós, Esther, et al.. (2003). Aggregation of MHC class I molecules on a CD8+ α β T cell clone specifically inhibits non‐antigen‐specific lysis of target cells. European Journal of Immunology. 34(1). 47–55. 3 indexed citations
8.
Montoya, Marı́a C., David Sancho, G. Bonello, et al.. (2002). Role of ICAM-3 in the initial interaction of T lymphocytes and APCs. Nature Immunology. 3(2). 159–168. 132 indexed citations
9.
Padmanabhan, S., et al.. (2001). Domain Architecture of a High Mobility Group A-type Bacterial Transcriptional Factor. Journal of Biological Chemistry. 276(45). 41566–41575. 34 indexed citations
10.
Bonilla, Sonia, et al.. (2001). From hematopoietic stem cells to neural stem cells. The International Journal of Developmental Biology. 45(S1). S67–S68. 1 indexed citations
11.
Bellón, Teresa, Manuel Llano, Alfredo Minguela, et al.. (1999). Triggering of Effector Functions on a CD8+ T Cell Clone Upon the Aggregation of an Activatory CD94/kp39 Heterodimer. The Journal of Immunology. 162(7). 3996–4002. 57 indexed citations
12.
Martínez‐Esparza, María, Celia Jiménez‐Cervantes, Friedrich Beermann, et al.. (1997). Transforming Growth Factor-β1 Inhibits Basal Melanogenesis in B16/F10 Mouse Melanoma Cells by Increasing the Rate of Degradation of Tyrosinase and Tyrosinase-related Protein-1. Journal of Biological Chemistry. 272(7). 3967–3972. 68 indexed citations
13.
14.
Hernández‐Caselles, Trinidad, Gonzalo Rubio, Miguel R. Campanero, et al.. (1993). ICAM‐3, the third LFA‐1 counterreceptor, is a co‐stimulatory molecule for both resting and activated T lymphocytes. European Journal of Immunology. 23(11). 2799–2806. 90 indexed citations
15.
Regueiro, José R., Paloma Pérez‐Aciego, Pedro Aparício, et al.. (1990). Low IgG2 and polysaccharide response in a T cell receptor expression defect. European Journal of Immunology. 20(11). 2411–2416. 16 indexed citations
16.
Aparício, Pedro, et al.. (1989). Isolation and characterization of (gamma, delta) CD4+ T cell clones derived from human fetal liver cells.. The Journal of Experimental Medicine. 170(3). 1009–1014. 29 indexed citations
17.
Aparício, Pedro, et al.. (1989). Differential Growth Requirements and Effector Functions of α/β and γ/δ Human T Cells. Immunological Reviews. 111(1). 5–33. 19 indexed citations
18.
Jaraquemada, Dolores, Begoña Galocha, Pedro Aparício, et al.. (1988). Modulation on immunogenicity by HLA‐B27 subtype polymorphism. European Journal of Immunology. 18(12). 1945–1950. 9 indexed citations
19.
Aparício, Pedro, Dolores Jaraquemada, & José A. Łópez de Castro. (1987). Alloreactive cytolytic T cell clones with dual recognition of HLA-B27 and HLA-DR2 antigens. Selective involvement of CD8 in their class I--directed cytotoxicity.. The Journal of Experimental Medicine. 165(2). 428–443. 45 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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