Paul van der Saag

455 citations
6 papers · 357 · h-index 6

Impact in

Papers in

    • Estrogen and related hormone effects 5
    • Cancer Risks and Factors 2
    • HER2/EGFR in Cancer Research 1
    • Cytokine Signaling Pathways and Interactions 1

Paul van der Saag

6 papers receiving 351 citations

Peers

Paul van der Saag
Comparison fields: 5 of 65
  • Pathology and Forensic Medicine 156
  • Biochemistry 48
  • Genetics 189
  • Health, Toxicology and Mutagenesis 39
  • Cancer Research 40
Replace Kai Smolnikar with:
Kai Smolnikar Germany
Mawussi Adjakly France
Nam-Hee Kang South Korea
Min Ji Bak United States
Tseng Cc Japan
Aisha Dugani Libya
B. A. J. EVANS United Kingdom
Xiayu Wu China
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Citations per field
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Citations per year

Countries citing papers authored by Paul van der Saag

Since Specialization
Citations

This map shows the geographic impact of Paul van der Saag's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul van der Saag with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul van der Saag more than expected).

Fields of papers citing papers by Paul van der Saag

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Paul van der Saag. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul van der Saag. The network helps show where Paul van der Saag may publish in the future.

Co-authors

The 25 scholars most cited alongside Paul van der Saag, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Paul van der Saag Line = papers co-authored together Paul van der Saag links everyone, so they are left out of the graph.

All Works

6 of 6 papers shown
#Work
1 2008106
2 2007106
3 200885
4 201133
5 199914
6 200413

About Paul van der Saag

Paul van der Saag is a scholar working on Genetics, Oncology, Molecular Biology, Pathology and Forensic Medicine and Endocrine and Autonomic Systems, having authored 6 papers that have together received 357 indexed citations. Recurring topics across this work include Estrogen and related hormone effects (5 papers), Phytoestrogen effects and research (3 papers), Retinoids in leukemia and cellular processes (2 papers), Cancer Risks and Factors (2 papers), Connexins and lens biology (1 paper), HER2/EGFR in Cancer Research (1 paper), Cytokine Signaling Pathways and Interactions (1 paper) and Bone Metabolism and Diseases (1 paper). The work is most often cited by research in Pathology and Forensic Medicine (156 citations), Biochemistry (48 citations), Genetics (189 citations), Health, Toxicology and Mutagenesis (39 citations) and Cancer Research (40 citations). Paul van der Saag has collaborated with scholars based in Netherlands, Sweden and Germany. Frequent co-authors include Albertinka J. Murk, Anders Ström, Ivonne M.C.M. Rietjens, Jacques Vervoort, Sari Mäkelä, Anastasios Damdimopoulos, Ingemar Pongratz, Hans van den Berg, Jan-Ακε Gustafsson and Jan Jaehrling. Their work appears in journals such as Toxicological Sciences, Journal of Nutrition, Archives of Biochemistry and Biophysics, Bone and The Journal of Steroid Biochemistry and Molecular Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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