Parash Parajuli

595 total citations
22 papers, 433 citations indexed

About

Parash Parajuli is a scholar working on Molecular Biology, Oncology and Organic Chemistry. According to data from OpenAlex, Parash Parajuli has authored 22 papers receiving a total of 433 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 6 papers in Oncology and 4 papers in Organic Chemistry. Recurrent topics in Parash Parajuli's work include Pancreatic and Hepatic Oncology Research (5 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Free Radicals and Antioxidants (4 papers). Parash Parajuli is often cited by papers focused on Pancreatic and Hepatic Oncology Research (5 papers), Cancer, Hypoxia, and Metabolism (4 papers) and Free Radicals and Antioxidants (4 papers). Parash Parajuli collaborates with scholars based in United States, France and Australia. Parash Parajuli's co-authors include Paul W. Sylvester, Roshan V. Tiwari, Mohammed S. Razzaque, Azeddine Atfi, Céline Prunier, Fathy A. Behery, Khalid A. El Sayed, Mohamed R. Akl, Keli Xu and Purba Singh and has published in prestigious journals such as The Journal of Cell Biology, The EMBO Journal and Cancer Research.

In The Last Decade

Parash Parajuli

21 papers receiving 428 citations

Peers

Parash Parajuli

BIOCH

ONCOL

PHYSI

A. C. S. A. Herrera Brazil

Shuhei Yamada Japan

Seema Noor Germany

Soumya Sasi United States

Diego Romero‐Perez United States

Richa Tiwary United States

Zhirong Wang China

Zhiping Guo China

Baolei Zhao China

Haixiang Su China

A. C. S. A. Herrera Brazil

Parash Parajuli

207 ×0.9

59 ×0.6

BIOCH

108 ×1.1

154 ×2.1

ONCOL

49 ×0.7

PHYSI

Citations per year, relative to Parash Parajuli Parash Parajuli (= 1×) peers A. C. S. A. Herrera

Countries citing papers authored by Parash Parajuli

Since Specialization

Citations

This map shows the geographic impact of Parash Parajuli's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Parash Parajuli with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Parash Parajuli more than expected).

Fields of papers citing papers by Parash Parajuli

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Parash Parajuli. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Parash Parajuli. The network helps show where Parash Parajuli may publish in the future.

Co-authorship network of co-authors of Parash Parajuli

This figure shows the co-authorship network connecting the top 25 collaborators of Parash Parajuli. A scholar is included among the top collaborators of Parash Parajuli based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Parash Parajuli. Parash Parajuli is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Hurwitz, Eric, Parash Parajuli, Céline Prunier, et al.. (2023). Antagonism between Prdm16 and Smad4 specifies the trajectory and progression of pancreatic cancer. The Journal of Cell Biology. 222(4). 10 indexed citations

Parajuli, Parash, et al.. (2023). Deciphering epithelial-to-mesenchymal transition in pancreatic cancer. Advances in cancer research. 159. 37–73. 4 indexed citations

Ramakrishnan, Gopalakrishnan, Parash Parajuli, Eric Hurwitz, et al.. (2022). NF1 loss of function as an alternative initiating event in pancreatic ductal adenocarcinoma. Cell Reports. 41(6). 111623–111623. 4 indexed citations

Parajuli, Parash, et al.. (2021). Cancer-Mediated Muscle Cachexia: Etiology and Clinical Management. Trends in Endocrinology and Metabolism. 32(6). 382–402. 38 indexed citations

Parajuli, Parash, et al.. (2020). Pancreatic cancer triggers diabetes through TGF-β–mediated selective depletion of islet β-cells. Life Science Alliance. 3(6). e201900573–e201900573. 20 indexed citations

Parajuli, Parash, Purba Singh, Zhe Wang, et al.. (2019). TGIF 1 functions as a tumor suppressor in pancreatic ductal adenocarcinoma. The EMBO Journal. 38(13). e101067–e101067. 21 indexed citations

Parajuli, Parash, Audrey Loumaye, Purba Singh, et al.. (2018). Twist1 Activation in Muscle Progenitor Cells Causes Muscle Loss Akin to Cancer Cachexia. Developmental Cell. 45(6). 712–725.e6. 46 indexed citations

Tiwari, Roshan V., Parash Parajuli, & Paul W. Sylvester. (2015). Synergistic anticancer effects of combined γ-tocotrienol and oridonin treatment is associated with the induction of autophagy. Molecular and Cellular Biochemistry. 408(1-2). 123–137. 35 indexed citations

Parajuli, Parash, Roshan V. Tiwari, & Paul W. Sylvester. (2015). Anticancer Effects of γ-Tocotrienol Are Associated with a Suppression in Aerobic Glycolysis. Biological and Pharmaceutical Bulletin. 38(9). 1352–1360. 18 indexed citations

10.

Tiwari, Roshan V., Parash Parajuli, & Paul W. Sylvester. (2015). γ-Tocotrienol-induced endoplasmic reticulum stress and autophagy act concurrently to promote breast cancer cell death. Biochemistry and Cell Biology. 93(4). 306–320. 39 indexed citations

11.

Parajuli, Parash, et al.. (2014). Anticancer Effects of Combined γ-Tocotrienol and PPARγ-Antagonist Treatment are Associated with a Suppression in Adipogenic Factor Expression. Journal of Pharmacy and Nutrition Sciences. 4(1). 43–56. 2 indexed citations

12.

Parajuli, Parash, et al.. (2014). Anticancer Effects of Combined Î³-Tocotrienol and PPARÎ³ Antagonist Treatment are Associated with a Suppression in Adipogenic Factor Expression. 4(1). 43–56. 2 indexed citations

13.

Parajuli, Parash, Fathy A. Behery, Alaadin Alayoubi, et al.. (2014). Oxazine derivatives of γ- and δ-tocotrienol display enhanced anticancer activity in vivo.. PubMed. 34(6). 2715–26. 28 indexed citations

14.

Parajuli, Parash, Fathy A. Behery, Alaadin Alayoubi, et al.. (2014). δ-Tocotrienol Oxazine Derivative Antagonizes Mammary Tumor Cell Compensatory Response to CoCl₂-Induced Hypoxia. BioMed Research International. 2014. 1–13. 9 indexed citations

15.

Tiwari, Roshan V., Parash Parajuli, & Paul W. Sylvester. (2013). γ-Tocotrienol-induced autophagy in malignant mammary cancer cells. Experimental Biology and Medicine. 239(1). 33–44. 48 indexed citations

16.

Parajuli, Parash, et al.. (2013). Abstract P3-03-11: Oxazine derivatives of g- and D- tocotrienols display potent anticancer effects in vivo. Cancer Research. 73(24_Supplement). P3–3. 3 indexed citations

17.

Sylvester, Paul W., et al.. (2013). Potential role of tocotrienols in the treatment and prevention of breast cancer. BioFactors. 40(1). 49–58. 36 indexed citations

18.

MacNutt, Meaghan J., et al.. (2012). Acclimatisation in trekkers with and without recent exposure to high altitude. European Journal of Applied Physiology. 112(9). 3287–3294. 10 indexed citations

19.

Behery, Fathy A., et al.. (2012). Optimization of tocotrienols as antiproliferative and antimigratory leads. European Journal of Medicinal Chemistry. 59. 329–341. 14 indexed citations

20.

Khanal, Pratik, et al.. (2010). Substance Use among Medical Students in Kathmandu Valley. Journal of Nepal Medical Association. 50(180). 267–72. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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