P. Reed Larsen
- Endocrinology, Diabetes and Metabolism top 0.05%
- Molecular Biology top 2%
- Genetics top 1%
- Physiology top 1%
- Pediatrics, Perinatology and Child Health top 1%
- Co-authors
- John W. HarneyAntônio C. BiancoDomenico SalvatoreGregory A. BrentMichael M. KaplanJack L. LeonardSung Woo KimDavid D. Moore
- Topics
- Thyroid Disorders and Treatments (87 papers)Growth Hormone and Insulin-like Growth Factors (25 papers)Thyroid Cancer Diagnosis and Treatment (16 papers)
- Partner nations
- United StatesItalyHungary
In The Last Decade
P. Reed Larsen
125 papers receiving 9.4k citations
Peers
Comparison fields: 5 of 136
- Endocrinology, Diabetes and Metabolism 6.4k
- Molecular Biology 3.0k
- Genetics 1.6k
- Physiology 1.5k
- Pediatrics, Perinatology and Child Health 860
Countries citing papers authored by P. Reed Larsen
This map shows the geographic impact of P. Reed Larsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P. Reed Larsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P. Reed Larsen more than expected).
Fields of papers citing papers by P. Reed Larsen
This network shows the impact of papers produced by P. Reed Larsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P. Reed Larsen. The network helps show where P. Reed Larsen may publish in the future.
Co-authorship network of co-authors of P. Reed Larsen
This figure shows the co-authorship network connecting the top 25 collaborators of P. Reed Larsen. A scholar is included among the top collaborators of P. Reed Larsen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with P. Reed Larsen. P. Reed Larsen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 16 | |
| 3 | 3 | |
| 4 | 111 | |
| 5 | 210 | |
| 6 | 19 | |
| 7 | 143 | |
| 8 | 91 | |
| 9 | 109 | |
| 10 | 49 | |
| 11 | 380 | |
| 12 | 105 | |
| 13 | 70 | |
| 14 | 128 | |
| 15 | 11 | |
| 16 | 384 | |
| 17 | 221 | |
| 18 | 75 | |
| 19 | 284 | |
| 20 | 38 |
About P. Reed Larsen
P. Reed Larsen is a scholar working on Endocrinology, Diabetes and Metabolism, Endocrine and Autonomic Systems and Physiology, having authored 125 papers that have together received 9.8k indexed citations. Recurring topics across this work include Thyroid Disorders and Treatments (87 papers), Growth Hormone and Insulin-like Growth Factors (25 papers) and Thyroid Cancer Diagnosis and Treatment (16 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (6.4k citations), Endocrine and Autonomic Systems (827 citations) and Physiology (1.5k citations). P. Reed Larsen has collaborated with scholars based in United States, Italy and Hungary. Frequent co-authors include John W. Harney, Antônio C. Bianco, Domenico Salvatore, Gregory A. Brent, Michael M. Kaplan, Jack L. Leonard, Sung Woo Kim, David D. Moore, Monica Dentice and Theo J. Visser. Their work appears in journals such as Nature, Science and New England Journal of Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.