P. L. Kramer

2.8k total citations
41 papers, 1.9k citations indexed

About

P. L. Kramer is a scholar working on Neurology, Cellular and Molecular Neuroscience and Molecular Biology. According to data from OpenAlex, P. L. Kramer has authored 41 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Neurology, 19 papers in Cellular and Molecular Neuroscience and 11 papers in Molecular Biology. Recurrent topics in P. L. Kramer's work include Neurological disorders and treatments (19 papers), Genetic Neurodegenerative Diseases (13 papers) and Hereditary Neurological Disorders (10 papers). P. L. Kramer is often cited by papers focused on Neurological disorders and treatments (19 papers), Genetic Neurodegenerative Diseases (13 papers) and Hereditary Neurological Disorders (10 papers). P. L. Kramer collaborates with scholars based in United States, Germany and Sweden. P. L. Kramer's co-authors include Laurie J. Ozelius, Susan Bressman, D. de Leon, Neil Risch, Mitchell F. Brin, M. Litt, Stanley Fahn, Christine Klein, Deborah Raymond and Irene H. Maumenee and has published in prestigious journals such as Nature Genetics, Neurology and Annals of Neurology.

In The Last Decade

P. L. Kramer

41 papers receiving 1.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
P. L. Kramer United States	23	1.0k	837	574	323	292	41	1.9k
Friedrich Asmus Germany	26	1.3k 1.3×	753 0.9×	348 0.6×	359 1.1×	290 1.0×	50	2.0k
A. Perretti Italy	26	529 0.5×	670 0.8×	440 0.8×	98 0.3×	29 0.1×	61	1.4k
S Tsuji Japan	16	697 0.7×	1.0k 1.2×	931 1.6×	164 0.5×	19 0.1×	39	1.7k
Setsuko Suemune Japan	19	188 0.2×	730 0.9×	293 0.5×	96 0.3×	32 0.1×	30	1.5k
Mohamed Lehar United States	19	294 0.3×	324 0.4×	590 1.0×	41 0.1×	104 0.4×	34	1.4k
Christine R. Matheson United States	13	194 0.2×	1.8k 2.1×	684 1.2×	108 0.3×	46 0.2×	15	2.3k
Atle Melberg Sweden	25	285 0.3×	432 0.5×	1.4k 2.4×	239 0.7×	30 0.1×	44	2.1k
Aasef G. Shaikh United States	23	766 0.7×	532 0.6×	224 0.4×	48 0.1×	159 0.5×	114	1.7k
Haruhiko Naito Japan	14	608 0.6×	1.2k 1.5×	1.0k 1.8×	227 0.7×	11 0.0×	50	1.6k
Dominic G. O’Donovan United Kingdom	16	268 0.3×	280 0.3×	401 0.7×	65 0.2×	38 0.1×	54	1.4k

Countries citing papers authored by P. L. Kramer

Since Specialization

Citations

This map shows the geographic impact of P. L. Kramer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P. L. Kramer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P. L. Kramer more than expected).

Fields of papers citing papers by P. L. Kramer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by P. L. Kramer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P. L. Kramer. The network helps show where P. L. Kramer may publish in the future.

Co-authorship network of co-authors of P. L. Kramer

This figure shows the co-authorship network connecting the top 25 collaborators of P. L. Kramer. A scholar is included among the top collaborators of P. L. Kramer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with P. L. Kramer. P. L. Kramer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Pasutto, Francesca, Kate E. Keller, Nicole Weisschuh, et al.. (2011). Variants in ASB10 are associated with open-angle glaucoma. Human Molecular Genetics. 21(6). 1336–1349. 62 indexed citations

Schmidt, Alexander, H.‐C. Jabusch, Eckart Altenmüller, et al.. (2006). Dominantly transmitted focal dystonia in families of patients with musician’s cramp. Neurology. 67(4). 691–693. 43 indexed citations

Samples, J. R., et al.. (2004). GLC1G: Mapping a new POAG locus on chormosome 5. Investigative Ophthalmology & Visual Science. 45(13). 4622–4622. 1 indexed citations

Bressman, Susan, Chiara Sabatti, Deborah Raymond, et al.. (2000). The DYT1 phenotype and guidelines for diagnostic testing. Neurology. 54(9). 1746–1753. 242 indexed citations

Zareparsi, Sepideh, Karin Wirdefeldt, Catherine E Burgess, et al.. (1999). Exclusion of dominant mutations within the FTDP-17 locus on chromosome 17 for Parkinson's disease. Neuroscience Letters. 272(2). 140–142. 2 indexed citations

Klein, Christine, et al.. (1998). Clinical and genetic evaluation of a family with a mixed dystonia phenotype from south tyrol. Annals of Neurology. 44(3). 394–398. 12 indexed citations

Almasy, Laura, Susan Bressman, Deborah Raymond, et al.. (1997). Idiopathic torsion dystonia linked to chromosome 8 in two mennonite families. Annals of Neurology. 42(4). 670–673. 116 indexed citations

Litt, M., et al.. (1997). Autosomal Dominant Cerulean Cataract Is Associated with a Chain Termination Mutation in the Human -Crystallin Gene CRYBB2. Human Molecular Genetics. 6(5). 665–668. 190 indexed citations

Wirtz, Mary K., J. R. Samples, P. L. Kramer, et al.. (1996). Weill-Marchesani syndrome - possible linkage of the autosomal dominant form to 15q21.1. American Journal of Medical Genetics. 65(1). 68–75. 31 indexed citations

10.

Kramer, P. L., et al.. (1996). A Second Gene for Cerulean Cataracts Maps to the β Crystallin Region on Chromosome 22. Genomics. 35(3). 539–542. 48 indexed citations

11.

Holmgren, Gösta, Laurie J. Ozelius, Lars Forsgren, et al.. (1995). Adult onset idiopathic torsion dystonia is excluded from the DYT 1 region (9q34) in a Swedish family.. Journal of Neurology Neurosurgery & Psychiatry. 59(2). 178–181. 30 indexed citations

12.

Kramer, P. L., David E. Root, & S. Gancher. (1994). A gene for nystagmus-associated episodic ataxia maps to chromosome 19p. The American Journal of Human Genetics. 55(3). 536–47. 4 indexed citations

13.

Wahlström, J., Laurie J. Ozelius, P. L. Kramer, et al.. (1994). The gene for familial dystonia with myoclonic jerks responsive to alcohol is not located on the distal end of 9q. Clinical Genetics. 45(2). 88–92. 11 indexed citations

14.

Bressman, Susan, Gary A. Heiman, Torbjoern G. Nygaard, et al.. (1994). A study of idiopathic torsion dystonia in a non‐Jewish family. Neurology. 44(2). 283–283. 54 indexed citations

15.

Kramer, P. L., Gary A. Heiman, Thomas Gasser, et al.. (1994). The DYT1 gene on 9q34 is responsible for most cases of early limb-onset idiopathic torsion dystonia in non-Jews.. PubMed. 55(3). 468–75. 89 indexed citations

16.

Litt, M., P. L. Kramer, D.L. Browne, et al.. (1994). A gene for episodic ataxia/myokymia maps to chromosome 12p13.. PubMed. 55(4). 702–9. 69 indexed citations

17.

Kramer, P. L., X.Y. Hauge, J L Weber, et al.. (1993). A microsatellite-based index map of human chromosome 11. Human Molecular Genetics. 2(7). 909–913. 31 indexed citations

18.

Bowcock, A., Robert I. Barnes, R. White, et al.. (1992). The CEPH consortium linkage map of human chromosome 15q. Genomics. 14(4). 833–840. 1 indexed citations

19.

Price, R. Arlen, P. L. Kramer, David L. Pauls, & Kenneth K. Kídd. (1989). Estimation of segregation and linkage parameters in simulated data. II. Simultaneous estimation with one linked marker.. PubMed. 45(1). 95–105. 4 indexed citations

20.

Kramer, P. L., Laurie J. Ozelius, James F. Gusella, et al.. (1987). Exclusion of autosomal dominant dystonia gene from large regions of chromosomes 11p, 13q, and 21q by multi‐point linkage analysis. Genetic Epidemiology. 4(5). 377–386. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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