Ototaka Higashi
- Co-authors
- Yoko KikuchiYoshiyuki HondaTsuneo ArakawaKuniaki NarisawaYasushi SatoTsunenobu TamuraTakashi MizunoTadashi Hayashi
- Topics
- Erythrocyte Function and Pathophysiology (5 papers)Acute Myeloid Leukemia Research (3 papers)Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers)
- Journals
- Archives of Disease in ChildhoodCancer Genetics and CytogeneticsThe Tohoku Journal of Experimental Medicine
- Partner nations
- Japan
In The Last Decade
Ototaka Higashi
25 papers receiving 342 citations
Peers
Comparison fields: 5 of 65
- Hematology 120
- Molecular Biology 101
- Immunology 86
- Rheumatology 65
- Genetics 62
Countries citing papers authored by Ototaka Higashi
This map shows the geographic impact of Ototaka Higashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ototaka Higashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ototaka Higashi more than expected).
Fields of papers citing papers by Ototaka Higashi
This network shows the impact of papers produced by Ototaka Higashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ototaka Higashi. The network helps show where Ototaka Higashi may publish in the future.
Co-authorship network of co-authors of Ototaka Higashi
This figure shows the co-authorship network connecting the top 25 collaborators of Ototaka Higashi. A scholar is included among the top collaborators of Ototaka Higashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ototaka Higashi. Ototaka Higashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 2 | |
| 2 | [Serotonergic disturbance in hereditary progressive dystonia--clinical effects of tetrahydrobiopterin and 5-hydroxytryptophan]. | 2 |
| 3 | 1 | |
| 4 | 5 | |
| 5 | 3 | |
| 6 | [Chédiak-Higashi syndrome]. | 0 |
| 7 | 41 | |
| 8 | [H2O2-induced platelet aggregation (author's transl)]. | 1 |
| 9 | 5 | |
| 10 | 12 | |
| 11 | 3 | |
| 12 | 2 | |
| 13 | 1 | |
| 14 | 15 | |
| 15 | 26 | |
| 16 | 54 | |
| 17 | 12 | |
| 18 | 155 | |
| 19 | 10 | |
| 20 | 1 |
About Ototaka Higashi
Ototaka Higashi is a scholar working on Hematology, Internal Medicine and Biochemistry, having authored 30 papers that have together received 408 indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (5 papers), Acute Myeloid Leukemia Research (3 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (3 papers). The work is most often cited by research in Hematology (120 citations), Clinical Biochemistry (53 citations) and Biochemistry (35 citations). Ototaka Higashi has collaborated with scholars based in Japan. Frequent co-authors include Yoko Kikuchi, Yoshiyuki Honda, Tsuneo Arakawa, Kuniaki Narisawa, Yasushi Sato, Tsunenobu Tamura, Takashi Mizuno, Tadashi Hayashi, Yoshiro Wada and Ryoichi Satodate. Their work appears in journals such as Archives of Disease in Childhood, Cancer Genetics and Cytogenetics and The Tohoku Journal of Experimental Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.