Olaf Witt

1.1k total citations
25 papers, 523 citations indexed

About

Olaf Witt is a scholar working on Molecular Biology, Neurology and Genetics. According to data from OpenAlex, Olaf Witt has authored 25 papers receiving a total of 523 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Neurology and 5 papers in Genetics. Recurrent topics in Olaf Witt's work include Histone Deacetylase Inhibitors Research (7 papers), Neuroblastoma Research and Treatments (6 papers) and Glioma Diagnosis and Treatment (3 papers). Olaf Witt is often cited by papers focused on Histone Deacetylase Inhibitors Research (7 papers), Neuroblastoma Research and Treatments (6 papers) and Glioma Diagnosis and Treatment (3 papers). Olaf Witt collaborates with scholars based in Germany, United States and France. Olaf Witt's co-authors include Ina Oehme, Arnulf Pekrun, Hedwig E. Deubzer, Jindřich Činátl, Hans Wilhelm Doerr, Martin Michaelis, Florian Rothweiler, Wolfgang Tillmann, Jörg Kreuter and Susanne Barth and has published in prestigious journals such as Journal of Clinical Oncology, Cancer Research and Biochemical Journal.

In The Last Decade

Olaf Witt

23 papers receiving 514 citations

Peers

Olaf Witt
Marlene C. Gerner Austria
Smita Matkar United States
Megan L. Goodall United States
Diana C. West United States
Shadi Zahedi United States
R Kancherla United States
Xiaohong Song China
Manon Verwijs-Janssen Netherlands
Franziska Wachter United States
Chiara Palladino Italy
Marlene C. Gerner Austria
Olaf Witt
Citations per year, relative to Olaf Witt Olaf Witt (= 1×) peers Marlene C. Gerner

Countries citing papers authored by Olaf Witt

Since Specialization
Citations

This map shows the geographic impact of Olaf Witt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Olaf Witt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Olaf Witt more than expected).

Fields of papers citing papers by Olaf Witt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Olaf Witt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Olaf Witt. The network helps show where Olaf Witt may publish in the future.

Co-authorship network of co-authors of Olaf Witt

This figure shows the co-authorship network connecting the top 25 collaborators of Olaf Witt. A scholar is included among the top collaborators of Olaf Witt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Olaf Witt. Olaf Witt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Nguyen, Duy, Sara Najafi, Norman Mack, et al.. (2024). Synergy of retinoic acid and BH3 mimetics in MYC(N)-driven embryonal nervous system tumours. British Journal of Cancer. 131(4). 763–777. 1 indexed citations
2.
Schneider, Annika, Camilla Bjørn Jensen, Amirhossein Sakhteman, et al.. (2024). 210P Therapeutic implications of phosphoproteomics in molecular cancer diagnostics. Annals of Oncology. 35. S300–S301. 1 indexed citations
3.
Chiang, Jason, Angela N. Viaene, Philipp Sievers, et al.. (2024). A multi-institutional series of a novel, recurrent TRIM24::MET fusion-driven infant-type hemispheric glioma reveals significant clinico-pathological heterogeneity. Acta Neuropathologica Communications. 12(1). 101–101.
4.
Berker, Yannick, Heike Peterziel, Petra Fiesel, et al.. (2022). Patient-by-Patient Deep Transfer Learning for Drug-Response Profiling Using Confocal Fluorescence Microscopy of Pediatric Patient-Derived Tumor-Cell Spheroids. IEEE Transactions on Medical Imaging. 41(12). 3981–3999. 5 indexed citations
5.
Ridinger, Johannes, Damir Krunic, Sara Najafi, et al.. (2021). Broad-Spectrum HDAC Inhibitors Promote Autophagy through FOXO Transcription Factors in Neuroblastoma. Cells. 10(5). 1001–1001. 25 indexed citations
6.
Morgen, Michael, Raphael R. Steimbach, Peter Sehr, et al.. (2020). Design and Synthesis of Dihydroxamic Acids as HDAC6/8/10 Inhibitors. ChemMedChem. 15(13). 1163–1174. 27 indexed citations
7.
Kolbinger, Fiona R., E Koeneke, Johannes Ridinger, et al.. (2018). The HDAC6/8/10 inhibitor TH34 induces DNA damage-mediated cell death in human high-grade neuroblastoma cell lines. Archives of Toxicology. 92(8). 2649–2664. 28 indexed citations
8.
Meyners, Christian, et al.. (2018). The enzyme activity of histone deacetylase 8 is modulated by a redox-switch. Redox Biology. 20. 60–67. 40 indexed citations
9.
Oehme, Ina, Marco Lodrini, Nathan Brady, & Olaf Witt. (2013). Histone deacetylase 10-promoted autophagy as a druggable point of interference to improve the treatment response of advanced neuroblastomas. Autophagy. 9(12). 2163–2165. 17 indexed citations
10.
Schmidt, René, Andreas Faldum, Olaf Witt, & Joachim Gerß. (2013). Adaptive designs with arbitrary dependence structure. Biometrical Journal. 56(1). 86–106. 7 indexed citations
11.
Rosenberger, Albert, Svantje Tauber, Christian Dullin, et al.. (2009). Antitumor Effects of a Combined 5-Aza-2′Deoxycytidine and Valproic Acid Treatment on Rhabdomyosarcoma and Medulloblastoma in Ptch Mutant Mice. Cancer Research. 69(3). 887–895. 85 indexed citations
12.
Michaelis, Martin, Malte Kleinschmidt, Susanne Barth, et al.. (2009). Anti-cancer effects of artesunate in a panel of chemoresistant neuroblastoma cell lines. Biochemical Pharmacology. 79(2). 130–136. 95 indexed citations
13.
Michaelis, Martin, Nora Hinsch, Florian Rothweiler, et al.. (2008). Cisplatin-Resistant Neuroblastoma Cells Express Enhanced Levels of Epidermal Growth Factor Receptor (EGFR) and Are Sensitive to Treatment with EGFR-Specific Toxins. Clinical Cancer Research. 14(20). 6531–6537. 45 indexed citations
14.
Wegener, Dennis, Christian Hildmann, Daniel Riester, et al.. (2008). Identification of novel small-molecule histone deacetylase inhibitors by medium-throughput screening using a fluorigenic assay. Biochemical Journal. 413(1). 143–150. 14 indexed citations
15.
Witt, Olaf, et al.. (2002). Induction of fetal hemoglobin synthesis by valproate: Modulation of MAPkinase pathways. American Journal of Hematology. 71(1). 45–46. 35 indexed citations
16.
Erdlenbruch, Bernhard, Arnulf Pekrun, Holger Schiffmann, Olaf Witt, & M. Lakomek. (2002). Accidental cisplatin overdose in a child: Reversal of acute renal failure with sodium thiosulfate. Medical and Pediatric Oncology. 38(5). 349–352. 16 indexed citations
17.
18.
Witt, Olaf, et al.. (2000). Tributyrin plusall-trans-retinoic acid efficiently induces fetal hemoglobin expression in human erythroleukemia cells. American Journal of Hematology. 64(4). 319–321. 13 indexed citations
19.
Jäckel, Martin C., Olaf Witt, Stefan Eber, Helmut Eiffert, & Rainer Laskawi. (1999). Die postoperative antibiotische Therapie von zervikalen Lymphadenitiden durch nichttuberkulöse, atypische Mykobakterien*. Laryngo-Rhino-Otologie. 78(8). 450–454. 2 indexed citations
20.
Witt, Olaf, et al.. (1999). Severe cerebral venous sinus thrombosis and dural arteriovenous fistula in an infant with protein S deficiency. Child s Nervous System. 15(2-3). 128–130. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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2026