Ola Awad

1.5k total citations
22 papers, 1.2k citations indexed

About

Ola Awad is a scholar working on Molecular Biology, Physiology and Cell Biology. According to data from OpenAlex, Ola Awad has authored 22 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Molecular Biology, 10 papers in Physiology and 8 papers in Cell Biology. Recurrent topics in Ola Awad's work include Lysosomal Storage Disorders Research (10 papers), Cellular transport and secretion (7 papers) and Mesenchymal stem cell research (6 papers). Ola Awad is often cited by papers focused on Lysosomal Storage Disorders Research (10 papers), Cellular transport and secretion (7 papers) and Mesenchymal stem cell research (6 papers). Ola Awad collaborates with scholars based in United States, Italy and Germany. Ola Awad's co-authors include Gina C. Schatteman, Ricardo A. Feldman, Chunhua Jiao, Leelamma M. Panicker, David M. Loeb, Martine Dunnwald, Elena Sivan‐Loukianova, Jackie R. Bickenbach, Diana Miller and Tea Soon Park and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and PLoS ONE.

In The Last Decade

Ola Awad

22 papers receiving 1.1k citations

Peers

Ola Awad
Pat Kumar United Kingdom
Caroline G. Humphries United States
Peter C. Stapor United States
Kouros Motamed United States
Pat Kumar United Kingdom
Ola Awad
Citations per year, relative to Ola Awad Ola Awad (= 1×) peers Pat Kumar

Countries citing papers authored by Ola Awad

Since Specialization
Citations

This map shows the geographic impact of Ola Awad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ola Awad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ola Awad more than expected).

Fields of papers citing papers by Ola Awad

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ola Awad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ola Awad. The network helps show where Ola Awad may publish in the future.

Co-authorship network of co-authors of Ola Awad

This figure shows the co-authorship network connecting the top 25 collaborators of Ola Awad. A scholar is included among the top collaborators of Ola Awad based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ola Awad. Ola Awad is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sarkar, Chinmoy, et al.. (2023). Deregulation of mTORC1-TFEB axis in human iPSC model of GBA1-associated Parkinson’s disease. Frontiers in Neuroscience. 17. 1152503–1152503. 13 indexed citations
2.
Jones, Jace W., Maureen A. Kane, Ola Awad, et al.. (2021). Elevated Glucosylsphingosine in Gaucher Disease induced Pluripotent Stem Cell Neurons Deregulates Lysosomal Compartment through Mammalian Target of Rapamycin Complex 1. Stem Cells Translational Medicine. 10(7). 1081–1094. 26 indexed citations
3.
Brown, Robert A., Tami J. Kingsbury, Marlene A. Jacobson, et al.. (2019). mTOR hyperactivity mediates lysosomal dysfunction in Gaucher's disease iPSC-neuronal cells. Disease Models & Mechanisms. 12(10). 51 indexed citations
4.
Awad, Ola, et al.. (2017). Mechanism of autophagy-lysosomal pathway dysfunction in Gaucher disease iPSC neurons (P1.151). Neurology. 88(16_supplement). 1 indexed citations
5.
Awad, Ola, Leelamma M. Panicker, Robert A. Brown, et al.. (2017). Altered Differentiation Potential of Gaucher’s Disease iPSC Neuronal Progenitors due to Wnt/β-Catenin Downregulation. Stem Cell Reports. 9(6). 1853–1867. 40 indexed citations
6.
Westbroek, Wendy, Ricardo A. Feldman, Ola Awad, et al.. (2016). A peptide-linked recombinant glucocerebrosidase for targeted neuronal delivery: Design, production, and assessment. Journal of Biotechnology. 221. 1–12. 22 indexed citations
7.
Awad, Ola, Chinmoy Sarkar, Leelamma M. Panicker, et al.. (2015). Altered TFEB-mediated lysosomal biogenesis in Gaucher disease iPSC-derived neuronal cells. Human Molecular Genetics. 24(20). 5775–5788. 98 indexed citations
8.
Park, Tea Soon, Diana Miller, Leelamma M. Panicker, et al.. (2015). Gaucher Disease-Induced Pluripotent Stem Cells Display Decreased Erythroid Potential and Aberrant Myelopoiesis. Stem Cells Translational Medicine. 4(8). 878–886. 20 indexed citations
9.
Trucco, Matteo, Ola Awad, Breelyn A. Wilky, et al.. (2013). A Novel Chordoma Xenograft Allows In Vivo Drug Testing and Reveals the Importance of NF-κB Signaling in Chordoma Biology. PLoS ONE. 8(11). e79950–e79950. 19 indexed citations
10.
Panicker, Leelamma M., Diana Miller, Tea Soon Park, et al.. (2012). Induced pluripotent stem cell model recapitulates pathologic hallmarks of Gaucher disease. Proceedings of the National Academy of Sciences. 109(44). 18054–18059. 95 indexed citations
11.
McCarty, Gregory, Ola Awad, & David M. Loeb. (2011). WT1 Protein Directly Regulates Expression of Vascular Endothelial Growth Factor and Is a Mediator of Tumor Response to Hypoxia. Journal of Biological Chemistry. 286(51). 43634–43643. 50 indexed citations
12.
Awad, Ola, Jason T. Yustein, Preeti Shah, et al.. (2010). High ALDH Activity Identifies Chemotherapy-Resistant Ewing's Sarcoma Stem Cells That Retain Sensitivity to EWS-FLI1 Inhibition. PLoS ONE. 5(11). e13943–e13943. 121 indexed citations
13.
Schatteman, Gina C., Ola Awad, Chunlin Wang, et al.. (2010). Lin− Cells Mediate Tissue Repair by Regulating MCP-1/CCL-2. American Journal Of Pathology. 177(4). 2002–2010. 6 indexed citations
14.
Stark, W J, Rakesh K. Goyal, Ola Awad, Eduardo Luis De Vito, & Anthony C. Kouzis. (2006). The safety and efficacy of combined phacoemulsification and trabeculectomy with releasable sutures. British Journal of Ophthalmology. 90(2). 146–149. 7 indexed citations
15.
Awad, Ola, Eduard I Dedkov, Chunhua Jiao, et al.. (2006). Differential Healing Activities of CD34 + and CD14 + Endothelial Cell Progenitors. Arteriosclerosis Thrombosis and Vascular Biology. 26(4). 758–764. 90 indexed citations
16.
Awad, Ola, Chunhua Jiao, Ning Ma, Martine Dunnwald, & Gina C. Schatteman. (2005). Obese Diabetic Mouse Environment Differentially Affects Primitive and Monocytic Endothelial Cell Progenitors. Stem Cells. 23(4). 575–583. 77 indexed citations
17.
Schatteman, Gina C. & Ola Awad. (2003). In Vivo and in Vitro Properties of CD34+ and CD14+ Endothelial Cell Precursors. Advances in experimental medicine and biology. 522. 9–16. 21 indexed citations
18.
Schatteman, Gina C. & Ola Awad. (2003). Hemangioblasts, angioblasts, and adult endothelial cell progenitors. The Anatomical Record Part A Discoveries in Molecular Cellular and Evolutionary Biology. 276A(1). 13–21. 76 indexed citations
19.
Sivan‐Loukianova, Elena, et al.. (2003). CD34+ Blood Cells Accelerate Vascularization and Healing of Diabetic Mouse Skin Wounds. Journal of Vascular Research. 40(4). 368–377. 144 indexed citations
20.
Awad, Ola, et al.. (2003). Lepr db Diabetic Mouse Bone Marrow Cells Inhibit Skin Wound Vascularization but Promote Wound Healing. Circulation Research. 92(11). 1247–1253. 69 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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