Nozomu Inamoto
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- Renin-Angiotensin System Studies 4
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- Hormonal Regulation and Hypertension 10
- Diet, Metabolism, and Disease 3
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- Ion Transport and Channel Regulation 3
- Receptor Mechanisms and Signaling 3
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- Genetic Associations and Epidemiology 3
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- Liver Disease Diagnosis and Treatment 2
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- Lipoproteins and Cardiovascular Health 2
Nozomu Inamoto
21 papers receiving 827 citations
Peers
Comparison fields: 5 of 64
- Cardiology and Cardiovascular Medicine 346
- Endocrinology, Diabetes and Metabolism 236
- Molecular Biology 372
- Genetics 119
- Nephrology 28
Countries citing papers authored by Nozomu Inamoto
This map shows the geographic impact of Nozomu Inamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nozomu Inamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nozomu Inamoto more than expected).
Fields of papers citing papers by Nozomu Inamoto
This network shows the impact of papers produced by Nozomu Inamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nozomu Inamoto. The network helps show where Nozomu Inamoto may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Nozomu Inamoto, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2007 | 118 | |
| 2 | 2006 | 25 | |
| 3 | 2006 | 39 | |
| 4 | 日本人集団における高血圧とCYBA,CLCNKB,およびKCNMB1の機能的遺伝子多型性の間の関連分析:吹田の研究 | 2005 | 50 |
| 5 | 2005 | 27 | |
| 6 | 2004 | 20 | |
| 7 | 2004 | 16 | |
| 8 | 2004 | 71 | |
| 9 | 2004 | 45 | |
| 10 | 2004 | 47 | |
| 11 | 2004 | 19 | |
| 12 | 2004 | 72 | |
| 13 | 2004 | 44 | |
| 14 | 2003 | 8 | |
| 15 | 2003 | 60 | |
| 16 | 2002 | 27 | |
| 17 | 2001 | 30 | |
| 18 | 2001 | 22 | |
| 19 | 2001 | 32 | |
| 20 | Effect of HgCl2 on rat kidney cells in primary culture. | 1976 | 13 |
About Nozomu Inamoto
Nozomu Inamoto is a scholar working on Endocrinology, Diabetes and Metabolism, Cardiology and Cardiovascular Medicine, Biochemistry, Biochemistry and Molecular Biology, having authored 21 papers that have together received 851 indexed citations. Recurring topics across this work include Hormonal Regulation and Hypertension (10 papers), Renin-Angiotensin System Studies (4 papers), Genetic Associations and Epidemiology (3 papers), Ion Transport and Channel Regulation (3 papers), Diet, Metabolism, and Disease (3 papers), Receptor Mechanisms and Signaling (3 papers), Liver Disease Diagnosis and Treatment (2 papers) and Lipoproteins and Cardiovascular Health (2 papers). The work is most often cited by research in Cardiology and Cardiovascular Medicine (346 citations), Endocrinology, Diabetes and Metabolism (236 citations), Molecular Biology (372 citations), Genetics (119 citations) and Nephrology (28 citations). Nozomu Inamoto has collaborated with scholars based in Japan, United States and Vietnam. Frequent co-authors include Yoshihiro Kokubo, Hitonobu Tomoike, Naoharu Iwai, Toshifumi Mannami, Keisuke Shioji, Naomi Tago, Hiroaki Naraba, Toshio Ogihara, Kei Kamide and Yuhei Kawano. Their work appears in journals such as Journal of Hypertension, Hypertension Research, Stroke, Atherosclerosis and Journal of the American College of Cardiology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.