Noam Levaot

729 total citations
20 papers, 529 citations indexed

About

Noam Levaot is a scholar working on Molecular Biology, Oncology and Genetics. According to data from OpenAlex, Noam Levaot has authored 20 papers receiving a total of 529 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 8 papers in Oncology and 3 papers in Genetics. Recurrent topics in Noam Levaot's work include Bone Metabolism and Diseases (9 papers), Bone health and treatments (8 papers) and S100 Proteins and Annexins (4 papers). Noam Levaot is often cited by papers focused on Bone Metabolism and Diseases (9 papers), Bone health and treatments (8 papers) and S100 Proteins and Annexins (4 papers). Noam Levaot collaborates with scholars based in Israel, United States and Germany. Noam Levaot's co-authors include Michal Hershfinkel, Andrew J. Scotter, Salima Janmohamed, Robert Rottapel, José La Rose, Ioannis D. Dimitriou, Paul D. Simoncic, Paul M. Krzyzanowski, Yasuyoshi Ueki and Shengqing Gu and has published in prestigious journals such as Cell, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Noam Levaot

17 papers receiving 525 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Noam Levaot Israel 12 304 173 94 60 51 20 529
Sandra Parenti Italy 14 228 0.8× 75 0.4× 51 0.5× 26 0.4× 37 0.7× 27 430
Stephen H. Schilling United States 8 404 1.3× 108 0.6× 32 0.3× 86 1.4× 20 0.4× 10 555
Meiheng Yang United States 10 339 1.1× 129 0.7× 35 0.4× 59 1.0× 69 1.4× 18 503
Brian J. Grindel United States 13 273 0.9× 78 0.5× 32 0.3× 36 0.6× 15 0.3× 21 486
K. Sundquist Finland 9 589 1.9× 269 1.6× 49 0.5× 131 2.2× 46 0.9× 16 734
Eeva-Kaisa Karhukorpi Finland 6 500 1.6× 350 2.0× 49 0.5× 134 2.2× 32 0.6× 8 733
Haizi Cheng United States 13 218 0.7× 49 0.3× 57 0.6× 59 1.0× 7 0.1× 25 444
Norihiro Ishida‐Kitagawa Japan 11 227 0.7× 63 0.4× 65 0.7× 34 0.6× 11 0.2× 19 360
Jinjie Wu China 14 161 0.5× 64 0.4× 171 1.8× 20 0.3× 14 0.3× 22 537

Countries citing papers authored by Noam Levaot

Since Specialization
Citations

This map shows the geographic impact of Noam Levaot's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Noam Levaot with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Noam Levaot more than expected).

Fields of papers citing papers by Noam Levaot

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Noam Levaot. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Noam Levaot. The network helps show where Noam Levaot may publish in the future.

Co-authorship network of co-authors of Noam Levaot

This figure shows the co-authorship network connecting the top 25 collaborators of Noam Levaot. A scholar is included among the top collaborators of Noam Levaot based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Noam Levaot. Noam Levaot is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Loboda, Jure, Emmanuelle Merquiol, Boris Turk, et al.. (2025). Development and Application of Small Molecule–Peptide Conjugates as Cathepsin K-Specific Covalent Irreversible Inhibitors in Human Osteoclast and Lung Cancer. JACS Au. 5(3). 1104–1120. 4 indexed citations
2.
Stepensky, David, et al.. (2024). Preventing osteoporotic bone loss in mice by promoting balanced bone remodeling through M-CSFRGD, a dual antagonist to c-FMS and αvβ3 receptors. International Journal of Biological Macromolecules. 282(Pt 2). 136821–136821.
3.
4.
Halpérin, Daniel, Yuval Yogev, Ohad Wormser, et al.. (2023). SMARCA4 mutation causes human otosclerosis and a similar phenotype in mice. Journal of Medical Genetics. 61(2). 117–124.
5.
Papo, Niv, et al.. (2022). Competitive blocking of LRP4–sclerostin binding interface strongly promotes bone anabolic functions. Cellular and Molecular Life Sciences. 79(2). 113–113. 11 indexed citations
6.
Meirow, Yaron, Daniele Colombo, Kerem Ben‐Meir, et al.. (2022). Specific inflammatory osteoclast precursors induced during chronic inflammation give rise to highly active osteoclasts associated with inflammatory bone loss. Bone Research. 10(1). 36–36. 29 indexed citations
7.
Levaot, Noam, et al.. (2020). Positive Outcomes of Denosumab Treatment in 2 Patients With Cherubism. Journal of Oral and Maxillofacial Surgery. 78(12). 2226–2234. 7 indexed citations
8.
Levaot, Noam, et al.. (2019). Reconstruction of the ovary microenvironment utilizing macroporous scaffold with affinity-bound growth factors. Biomaterials. 205. 11–22. 36 indexed citations
9.
Einav, Yulia, et al.. (2018). A dual-specific macrophage colony-stimulating factor antagonist of c-FMS and αvβ3 integrin for osteoporosis therapy. PLoS Biology. 16(8). e2002979–e2002979. 24 indexed citations
10.
Dahan, Idit, et al.. (2018). Unraveling the transcriptional regulation of TWIST1 in limb development. PLoS Genetics. 14(10). e1007738–e1007738. 21 indexed citations
11.
Omar, Ibrahim Che, et al.. (2018). Schlafen2 mutation in mice causes an osteopetrotic phenotype due to a decrease in the number of osteoclast progenitors. Scientific Reports. 8(1). 13005–13005. 12 indexed citations
12.
Schmidt, Felix N., Hanifeh Khayyeri, Sahar Hiram‐Bab, et al.. (2018). Perturbed bone composition and integrity with disorganized osteoblast function in zinc receptor/Gpr39‐deficient mice. The FASEB Journal. 32(5). 2507–2518. 21 indexed citations
13.
Levaot, Noam & Michal Hershfinkel. (2018). How cellular Zn2+ signaling drives physiological functions. Cell Calcium. 75. 53–63. 76 indexed citations
14.
Levaot, Noam, et al.. (2017). Dual‐specificity tyrosine phosphorylation‐regulated kinase 2 regulates osteoclast fusion in a cell heterotypic manner. Journal of Cellular Physiology. 233(1). 617–629. 6 indexed citations
15.
Ilić, Stefan, Anat Shahar, Barak Akabayov, et al.. (2017). Engineering a monomeric variant of macrophage colony-stimulating factor (M-CSF) that antagonizes the c-FMS receptor. Biochemical Journal. 474(15). 2601–2617. 9 indexed citations
16.
Levaot, Noam, et al.. (2016). GPR39 is negatively regulating osteoblast differentiation and bone formation during aging. Bone Abstracts. 1 indexed citations
17.
18.
Levaot, Noam, et al.. (2015). Combinatorial and Computational Approaches to Identify Interactions of Macrophage Colony-stimulating Factor (M-CSF) and Its Receptor c-FMS. Journal of Biological Chemistry. 290(43). 26180–26193. 18 indexed citations
19.
Levaot, Noam, Oleksandr Voytyuk, Ioannis D. Dimitriou, et al.. (2011). Loss of Tankyrase-Mediated Destruction of 3BP2 Is the Underlying Pathogenic Mechanism of Cherubism. Cell. 147(6). 1324–1339. 150 indexed citations
20.
Levaot, Noam, Paul D. Simoncic, Ioannis D. Dimitriou, et al.. (2011). 3BP2-deficient mice are osteoporotic with impaired osteoblast and osteoclast functions. Journal of Clinical Investigation. 121(8). 3244–3257. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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