Nina Schultz

508 total citations
18 papers, 346 citations indexed

About

Nina Schultz is a scholar working on Physiology, Molecular Biology and Neurology. According to data from OpenAlex, Nina Schultz has authored 18 papers receiving a total of 346 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Physiology, 6 papers in Molecular Biology and 6 papers in Neurology. Recurrent topics in Nina Schultz's work include Alzheimer's disease research and treatments (15 papers), Barrier Structure and Function Studies (4 papers) and Dementia and Cognitive Impairment Research (4 papers). Nina Schultz is often cited by papers focused on Alzheimer's disease research and treatments (15 papers), Barrier Structure and Function Studies (4 papers) and Dementia and Cognitive Impairment Research (4 papers). Nina Schultz collaborates with scholars based in Sweden, United States and Netherlands. Nina Schultz's co-authors include Malin Wennström, Elin Byman, Malin Fex, Henrietta M. Nielsen, Lennart Minthon, Anders Olofsson, Oskar Hansson, Kristoffer Brännström, Simon Moussaud and Shorena Janelidze and has published in prestigious journals such as PLoS ONE, International Journal of Molecular Sciences and Journal of Cerebral Blood Flow & Metabolism.

In The Last Decade

Nina Schultz

18 papers receiving 346 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nina Schultz Sweden 12 169 133 84 44 44 18 346
Elin Byman Sweden 10 119 0.7× 93 0.7× 61 0.7× 28 0.6× 20 0.5× 10 253
Felecia M. Marottoli United States 8 188 1.1× 114 0.9× 135 1.6× 48 1.1× 50 1.1× 12 392
Susana Carmona Portugal 7 172 1.0× 146 1.1× 133 1.6× 69 1.6× 39 0.9× 18 387
Eli C. Levin United States 6 154 0.9× 140 1.1× 146 1.7× 50 1.1× 15 0.3× 7 397
Xiao Nie Sweden 9 194 1.1× 160 1.2× 118 1.4× 87 2.0× 10 0.2× 11 478
Ori Liraz Israel 10 308 1.8× 76 0.6× 169 2.0× 105 2.4× 35 0.8× 15 470
Helen K. Warwick United Kingdom 4 249 1.5× 89 0.7× 218 2.6× 138 3.1× 30 0.7× 5 477
Brandon C. Farmer United States 8 220 1.3× 77 0.6× 249 3.0× 58 1.3× 34 0.8× 9 491
Thorsten Pflanzner Germany 8 180 1.1× 114 0.9× 136 1.6× 45 1.0× 25 0.6× 9 416

Countries citing papers authored by Nina Schultz

Since Specialization
Citations

This map shows the geographic impact of Nina Schultz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nina Schultz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nina Schultz more than expected).

Fields of papers citing papers by Nina Schultz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nina Schultz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nina Schultz. The network helps show where Nina Schultz may publish in the future.

Co-authorship network of co-authors of Nina Schultz

This figure shows the co-authorship network connecting the top 25 collaborators of Nina Schultz. A scholar is included among the top collaborators of Nina Schultz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nina Schultz. Nina Schultz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Nuñez‐Diaz, Cristina, Emelie Andersson, Nina Schultz, et al.. (2024). The fluorescent ligand bTVBT2 reveals increased p-tau uptake by retinal microglia in Alzheimer’s disease patients and AppNL−F/NL−F mice. Alzheimer s Research & Therapy. 16(1). 4–4. 7 indexed citations
2.
Wennström, Malin, Nina Schultz, Geidy E. Serrano, et al.. (2024). The Relationship between p-tau217, p-tau231, and p-tau205 in the Human Brain Is Affected by the Cellular Environment and Alzheimer’s Disease Pathology. Cells. 13(4). 331–331. 4 indexed citations
3.
Nuñez‐Diaz, Cristina, et al.. (2023). Contraction of human brain vascular pericytes in response to islet amyloid polypeptide is reversed by pramlintide. Molecular Brain. 16(1). 25–25. 5 indexed citations
4.
Andersson, Emelie, Nina Schultz, Takashi Saito, et al.. (2023). Cerebral Aβ deposition precedes reduced cerebrospinal fluid and serum Aβ42/Aβ40 ratios in the AppNL−F/NL−F knock-in mouse model of Alzheimer’s disease. Alzheimer s Research & Therapy. 15(1). 64–64. 11 indexed citations
5.
Roth, Bodil, Nina Schultz, Cristina Nuñez‐Diaz, et al.. (2023). Plasma IAPP-Autoantibody Levels in Alzheimer’s Disease Patients Are Affected by APOE4 Status. International Journal of Molecular Sciences. 24(4). 3776–3776. 3 indexed citations
6.
Argouarch, Andrea R., Nina Schultz, Andrew C. Yang, et al.. (2022). Postmortem Human Dura Mater Cells Exhibit Phenotypic, Transcriptomic and Genetic Abnormalities that Impact their Use for Disease Modeling. Stem Cell Reviews and Reports. 18(8). 3050–3065. 3 indexed citations
7.
Gobom, Johan, Andréa L. Benedet, Niklas Mattsson, et al.. (2022). Antibody-free measurement of cerebrospinal fluid tau phosphorylation across the Alzheimer’s disease continuum. Molecular Neurodegeneration. 17(1). 81–81. 25 indexed citations
8.
Treccani, Giulia, Nina Schultz, David P. Herzog, et al.. (2020). Hippocampal NG2+ pericytes in chronically stressed rats and depressed patients: a quantitative study. Stress. 24(3). 353–358. 12 indexed citations
9.
Gharibyan, Anna L., et al.. (2020). Apolipoprotein E Interferes with IAPP Aggregation and Protects Pericytes from IAPP-Induced Toxicity. Biomolecules. 10(1). 134–134. 16 indexed citations
10.
Schultz, Nina, et al.. (2019). Levels of Retinal Amyloid-β Correlate with Levels of Retinal IAPP and Hippocampal Amyloid-β in Neuropathologically Evaluated Individuals. Journal of Alzheimer s Disease. 73(3). 1201–1209. 35 indexed citations
11.
Byman, Elin, et al.. (2019). A Potential Role for α-Amylase in Amyloid-β-Induced Astrocytic Glycogenolysis and Activation. Journal of Alzheimer s Disease. 68(1). 205–217. 15 indexed citations
12.
Schultz, Nina, Shorena Janelidze, Elin Byman, et al.. (2019). Levels of islet amyloid polypeptide in cerebrospinal fluid and plasma from patients with Alzheimer’s disease. PLoS ONE. 14(6). e0218561–e0218561. 18 indexed citations
13.
Schultz, Nina, Elin Byman, & Malin Wennström. (2018). Levels of retinal IAPP are altered in Alzheimer's disease patients and correlate with vascular changes and hippocampal IAPP levels. Neurobiology of Aging. 69. 94–101. 17 indexed citations
14.
Byman, Elin, et al.. (2018). Brain alpha‐amylase: a novel energy regulator important in Alzheimer disease?. Brain Pathology. 28(6). 920–932. 29 indexed citations
15.
Schultz, Nina, Kristoffer Brännström, Elin Byman, et al.. (2018). Amyloid‐beta 1‐40 is associated with alterations in NG2+ pericyte population ex vivo and in vitro. Aging Cell. 17(3). e12728–e12728. 53 indexed citations
16.
Schultz, Nina, Elin Byman, Malin Fex, & Malin Wennström. (2016). Amylin alters human brain pericyte viability and NG2 expression. Journal of Cerebral Blood Flow & Metabolism. 37(4). 1470–1482. 56 indexed citations
17.
Schultz, Nina, Henrietta M. Nielsen, Lennart Minthon, & Malin Wennström. (2014). Involvement of Matrix Metalloproteinase-9 in Amyloid-β 1–42–Induced Shedding of the Pericyte Proteoglycan NG2. Journal of Neuropathology & Experimental Neurology. 73(7). 684–692. 34 indexed citations
18.
Rs, Dyer, et al.. (1979). Hippocampal afterdischarges and their post-ictal sequelae in rats: effects of carbon monoxide hypoxia.. PubMed. 1(1). 21–5. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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