Niloofar Ale‐Agha

1.7k total citations
31 papers, 1.3k citations indexed

About

Niloofar Ale‐Agha is a scholar working on Molecular Biology, Physiology and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Niloofar Ale‐Agha has authored 31 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 10 papers in Physiology and 6 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Niloofar Ale‐Agha's work include Connexins and lens biology (8 papers), Adipose Tissue and Metabolism (6 papers) and Air Quality and Health Impacts (5 papers). Niloofar Ale‐Agha is often cited by papers focused on Connexins and lens biology (8 papers), Adipose Tissue and Metabolism (6 papers) and Air Quality and Health Impacts (5 papers). Niloofar Ale‐Agha collaborates with scholars based in Germany, United States and Italy. Niloofar Ale‐Agha's co-authors include Judith Haendeler, Wilhelm Stahl, Joachim Altschmied, Helmut Sies, Joseph R. Burgoyne, Shinichi Oka, Philip Eaton, Maria Cristina Polidori, Lars‐Oliver Klotz and Iris Sawitza and has published in prestigious journals such as Journal of Biological Chemistry, PLoS ONE and Hepatology.

In The Last Decade

Niloofar Ale‐Agha

31 papers receiving 1.3k citations

Peers

Niloofar Ale‐Agha
Niloofar Ale‐Agha
Citations per year, relative to Niloofar Ale‐Agha Niloofar Ale‐Agha (= 1×) peers Noriko Fujiwara

Countries citing papers authored by Niloofar Ale‐Agha

Since Specialization
Citations

This map shows the geographic impact of Niloofar Ale‐Agha's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Niloofar Ale‐Agha with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Niloofar Ale‐Agha more than expected).

Fields of papers citing papers by Niloofar Ale‐Agha

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Niloofar Ale‐Agha. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Niloofar Ale‐Agha. The network helps show where Niloofar Ale‐Agha may publish in the future.

Co-authorship network of co-authors of Niloofar Ale‐Agha

This figure shows the co-authorship network connecting the top 25 collaborators of Niloofar Ale‐Agha. A scholar is included among the top collaborators of Niloofar Ale‐Agha based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Niloofar Ale‐Agha. Niloofar Ale‐Agha is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Ale‐Agha, Niloofar, et al.. (2024). The Adhesion GPCR ADGRL2/LPHN2 Can Protect Against Cellular and Organismal Dysfunction. Cells. 13(22). 1826–1826. 2 indexed citations
2.
Dyballa‐Rukes, Nadine, et al.. (2023). Caffeine Inhibits Oxidative Stress- and Low Dose Endotoxemia-Induced Senescence—Role of Thioredoxin-1. Antioxidants. 12(6). 1244–1244. 8 indexed citations
3.
Brinkmann, Vanessa, Margherita Romeo, Lucie Larigot, et al.. (2022). Aryl Hydrocarbon Receptor-Dependent and -Independent Pathways Mediate Curcumin Anti-Aging Effects. Antioxidants. 11(4). 613–613. 8 indexed citations
4.
Hundhausen, Christian, Joachim Altschmied, Niloofar Ale‐Agha, et al.. (2021). Endothelial hyaluronan synthase 3 aggravates acute colitis in an experimental model of inflammatory bowel disease. Matrix Biology. 102. 20–36. 5 indexed citations
5.
Brinkmann, Vanessa, Niloofar Ale‐Agha, Judith Haendeler, & Natascia Ventura. (2020). The Aryl Hydrocarbon Receptor (AhR) in the Aging Process: Another Puzzling Role for This Highly Conserved Transcription Factor. Frontiers in Physiology. 10. 1561–1561. 56 indexed citations
6.
Ale‐Agha, Niloofar, Christine Goy, Nadine Dyballa‐Rukes, et al.. (2018). Induction of a senescent like phenotype and loss of gap junctional intercellular communication by carbon nanoparticle exposure of lung epithelial cells. Experimental Gerontology. 117. 106–112. 9 indexed citations
7.
Dyballa‐Rukes, Nadine, Anna Eckers, Niloofar Ale‐Agha, et al.. (2016). The Anti-Apoptotic Properties of APEX1 in the Endothelium Require the First 20 Amino Acids and Converge on Thioredoxin-1. Antioxidants and Redox Signaling. 26(12). 616–629. 10 indexed citations
8.
Eckers, Anna, Sascha Jakob, Christian Heiß, et al.. (2016). The aryl hydrocarbon receptor promotes aging phenotypes across species. Scientific Reports. 6(1). 19618–19618. 63 indexed citations
9.
Goy, Christine, Joachim Altschmied, Sascha Jakob, et al.. (2014). The imbalanced redox status in senescent endothelial cells is due to dysregulated Thioredoxin-1 and NADPH oxidase 4. Experimental Gerontology. 56. 45–52. 22 indexed citations
10.
Ale‐Agha, Niloofar, Nadine Dyballa‐Rukes, Sascha Jakob, Joachim Altschmied, & Judith Haendeler. (2014). Cellular functions of the dual-targeted catalytic subunit of telomerase, telomerase reverse transcriptase — Potential role in senescence and aging. Experimental Gerontology. 56. 189–193. 36 indexed citations
11.
Sydlik, Ulrich, Niloofar Ale‐Agha, Andreas Bilstein, et al.. (2014). Signalling-Dependent Adverse Health Effects of Carbon Nanoparticles Are Prevented by the Compatible Solute Mannosylglycerate (Firoin) In Vitro and In Vivo. PLoS ONE. 9(11). e111485–e111485. 13 indexed citations
12.
Burgoyne, Joseph R., Shinichi Oka, Niloofar Ale‐Agha, & Philip Eaton. (2012). Hydrogen Peroxide Sensing and Signaling by Protein Kinases in the Cardiovascular System. Antioxidants and Redox Signaling. 18(9). 1042–1052. 164 indexed citations
13.
Ale‐Agha, Niloofar, Catrin Albrecht, & Lars‐Oliver Klotz. (2010). Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to carbon or silica-based nanoparticles. Biological Chemistry. 391(11). 1333–9. 15 indexed citations
14.
Ale‐Agha, Niloofar, Catrin Albrecht, & Lars‐Oliver Klotz. (2009). Loss of gap junctional intercellular communication in rat lung epithelial cells exposed to quartz particles. Biochemical and Biophysical Research Communications. 390(1). 44–47. 8 indexed citations
15.
Ale‐Agha, Niloofar, Stefanie Galbán, Kotb Abdelmohsen, et al.. (2009). HuR regulates gap junctional intercellular communication by controlling β-catenin levels and adherens junction integrity #. Hepatology. 50(5). 1567–1576. 39 indexed citations
16.
Grether‐Beck, Susanne, et al.. (2009). Role of HuR and p38MAPK in Ultraviolet B-induced Post-transcriptional Regulation of COX-2 Expression in the Human Keratinocyte Cell Line HaCaT. Journal of Biological Chemistry. 285(6). 3896–3904. 50 indexed citations
17.
Abdelmohsen, Kotb, Niloofar Ale‐Agha, Juliane I. Beier, et al.. (2007). Epidermal growth factor- and stress-induced loss of gap junctional communication is mediated by ERK-1/ERK-2 but not ERK-5 in rat liver epithelial cells. Biochemical and Biophysical Research Communications. 364(2). 313–317. 13 indexed citations
18.
Bellei, Barbara, Arianna Mastrofrancesco, Stefania Briganti, et al.. (2007). Ultraviolet A induced modulation of gap junctional intercellular communication by P38 MAPK activation in human keratinocytes. Experimental Dermatology. 17(2). 115–124. 23 indexed citations
19.
Aust, Olivier, et al.. (2003). Lycopene oxidation product enhances gap junctional communication. Food and Chemical Toxicology. 41(10). 1399–1407. 113 indexed citations
20.
Stahl, Wilhelm, Niloofar Ale‐Agha, & Maria Cristina Polidori. (2002). Non-Antioxidant Properties of Carotenoids. Biological Chemistry. 383(3-4). 553–558. 100 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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