Nicola Conran

3.2k total citations
105 papers, 2.0k citations indexed

About

Nicola Conran is a scholar working on Genetics, Hematology and Physiology. According to data from OpenAlex, Nicola Conran has authored 105 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 84 papers in Genetics, 60 papers in Hematology and 43 papers in Physiology. Recurrent topics in Nicola Conran's work include Hemoglobinopathies and Related Disorders (82 papers), Erythrocyte Function and Pathophysiology (37 papers) and Iron Metabolism and Disorders (25 papers). Nicola Conran is often cited by papers focused on Hemoglobinopathies and Related Disorders (82 papers), Erythrocyte Function and Pathophysiology (37 papers) and Iron Metabolism and Disorders (25 papers). Nicola Conran collaborates with scholars based in Brazil, United States and United Kingdom. Nicola Conran's co-authors include Fernando Ferreira Costa, Carla Fernanda Franco‐Penteado, John D. Belcher, Sara T. Olalla Saad, Andréia A. Canalli, Sara Teresinha Olalla Saad, Camila Bononi Almeida, Fabı́ola Traina, Carolina Lanaro and Lidiane S. Torres and has published in prestigious journals such as Blood, PLoS ONE and Scientific Reports.

In The Last Decade

Nicola Conran

100 papers receiving 2.0k citations

Peers

Nicola Conran

GENET

HEMAT

PHYSI

IMMUN

Jean‐Antoine Ribeil France

Kari Remes Finland

E. A. Rachmilewitz Israel

Çiğdem Altay Türkiye

I. Tatarsky Israel

Panagoula Κollia Greece

E. Fred Saunders Canada

Shigeki Ohtake Japan

Koichi Kamei Japan

Takayuki Saitoh Japan

Jean‐Antoine Ribeil France

Nicola Conran

976 ×0.7

GENET

871 ×0.8

HEMAT

307 ×0.5

PHYSI

701 ×1.7

277 ×1.4

IMMUN

Citations per year, relative to Nicola Conran Nicola Conran (= 1×) peers Jean‐Antoine Ribeil

Countries citing papers authored by Nicola Conran

Since Specialization

Citations

This map shows the geographic impact of Nicola Conran's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicola Conran with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicola Conran more than expected).

Fields of papers citing papers by Nicola Conran

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicola Conran. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicola Conran. The network helps show where Nicola Conran may publish in the future.

Co-authorship network of co-authors of Nicola Conran

This figure shows the co-authorship network connecting the top 25 collaborators of Nicola Conran. A scholar is included among the top collaborators of Nicola Conran based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicola Conran. Nicola Conran is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gushiken, Lucas Fernando Sérgio, Flávia C. Leonardo, Amanda Becerra, et al.. (2025). Caspase-1 activation drives vascular inflammatory processes and hypoperfusion in intravascular hemolysis. American Journal of Physiology-Heart and Circulatory Physiology. 329(4). H920–H938.

Gushiken, Lucas Fernando Sérgio, et al.. (2023). Molecular and cellular effects of in vivo chronic intravascular hemolysis and anti-inflammatory therapeutic approaches. Vascular Pharmacology. 150. 107176–107176. 7 indexed citations

Nader, Élie, Nicola Conran, Flávia C. Leonardo, et al.. (2023). Piezo1 activation augments sickling propensity and the adhesive properties of sickle red blood cells in a calcium‐dependent manner. British Journal of Haematology. 202(3). 657–668. 16 indexed citations

Sesti‐Costa, Renata, Fernando Ferreira Costa, & Nicola Conran. (2023). Role of Macrophages in Sickle Cell Disease Erythrophagocytosis and Erythropoiesis. International Journal of Molecular Sciences. 24(7). 6333–6333. 10 indexed citations

Silva, Juliete A.F., Flávia C. Leonardo, Carla Fernanda Franco‐Penteado, et al.. (2022). Reduced blood pressure in sickle cell disease is associated with decreased angiotensin converting enzyme (ACE) activity and is not modulated by ACE inhibition. PLoS ONE. 17(2). e0263424–e0263424. 5 indexed citations

Silva, Juliete A.F., et al.. (2017). Role for Platelets in Mediating Neutrophil Recruitment in TNF-α Induced Vaso-Occlusive Processes in Animals with Sickle Cell Anemia. Blood. 130. 540. 1 indexed citations

Colella, Marina Pereira, Bárbara A. Santana, Nicola Conran, et al.. (2017). Telomere length correlates with disease severity and inflammation in sickle cell disease. Revista Brasileira de Hematologia e Hemoterapia. 39(2). 140–145. 16 indexed citations

Pericole, Fernando Vieira, et al.. (2016). Inflammasome-Dependent IL-1β Release from Neutrophils in Human Sickle Cell Anemia. Blood. 128(22). 854–854. 5 indexed citations

Colella, Marina Pereira, Erich Vinícius De Paula, João Agostinho Machado‐Neto, et al.. (2015). Elevated hypercoagulability markers in hemoglobin SC disease. Haematologica. 100(4). 466–471. 28 indexed citations

10.

Conran, Nicola & Camila Bononi Almeida. (2015). Hemolytic vascular inflammation: an update. Revista Brasileira de Hematologia e Hemoterapia. 38(1). 55–57. 8 indexed citations

11.

Dominical, Venina Marcela, et al.. (2014). In vitro microfluidic model for the study of vaso-occlusive processes. Experimental Hematology. 43(3). 223–228. 19 indexed citations

12.

Albuquerque, Dulcinéia Martins de, et al.. (2014). In vitro and in vivo anti-angiogenic effects of hydroxyurea. Microvascular Research. 94. 106–113. 29 indexed citations

13.

Garrido, Vanessa T., Venina Marcela Dominical, Fabı́ola Traina, et al.. (2012). Elevated plasma levels and platelet‐associated expression of the pro‐thrombotic and pro‐inflammatory protein, T NFSF14 (LIGHT), in sickle cell disease. British Journal of Haematology. 158(6). 788–797. 25 indexed citations

14.

Fernandes, Luís Gustavo Romani, et al.. (2010). Role of the Mac-1 and VLA-4 integrins, and concomitant Th2-cytokine production, in nitric oxide modulated eosinophil migration from bone marrow to lungs in allergic mice. International Immunopharmacology. 11(2). 204–211. 13 indexed citations

15.

Conran, Nicola & Fernando Ferreira Costa. (2009). Hemoglobin disorders and endothelial cell interactions. Clinical Biochemistry. 42(18). 1824–1838. 48 indexed citations

16.

Canalli, Andréia A., et al.. (2008). Increased adhesive properties of neutrophils in sickle cell disease may be reversed by pharmacological nitric oxide donation. Haematologica. 93(4). 605–609. 59 indexed citations

17.

Lima, Carmen Sílvia Passos, Carla Fernanda Franco‐Penteado, Andréia A. Canalli, et al.. (2006). Expressions of the VLA-4, LFA-1 and Mac-1 integrins in eosinophil migration in a case of chronic eosinophilic leukaemia. Leukemia Research. 31(5). 695–697. 1 indexed citations

18.

Canalli, Andréia A., Fabı́ola Traina, Dulcinéia Martins de Albuquerque, et al.. (2006). Therapy with hydroxyurea is associated with reduced adhesion molecule gene and protein expression in sickle red cells with a concomitant reduction in adhesive properties. European Journal Of Haematology. 78(2). 144–151. 42 indexed citations

19.

Canalli, Andréia A., Nicola Conran, André Fattori, Sara T. Olalla Saad, & Fernando Ferreira Costa. (2004). Increased adhesive properties of eosinophils in sickle cell disease. Experimental Hematology. 32(8). 728–734. 19 indexed citations

20.

Conran, Nicola, et al.. (2004). Increased soluble guanylate cyclase activity in the red blood cells of sickle cell patients. British Journal of Haematology. 124(4). 547–554. 46 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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