Nicholas Doerr

843 total citations
15 papers, 393 citations indexed

About

Nicholas Doerr is a scholar working on Genetics, Pathology and Forensic Medicine and Immunology. According to data from OpenAlex, Nicholas Doerr has authored 15 papers receiving a total of 393 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Genetics, 6 papers in Pathology and Forensic Medicine and 5 papers in Immunology. Recurrent topics in Nicholas Doerr's work include Genetic and Kidney Cyst Diseases (7 papers), Immune Cell Function and Interaction (5 papers) and Biomedical Research and Pathophysiology (4 papers). Nicholas Doerr is often cited by papers focused on Genetic and Kidney Cyst Diseases (7 papers), Immune Cell Function and Interaction (5 papers) and Biomedical Research and Pathophysiology (4 papers). Nicholas Doerr collaborates with scholars based in United States, Austria and Greece. Nicholas Doerr's co-authors include Thomas Weimbs, Jonathan M. Shillingford, Shivakumar Vasanth, Michael Kinter, Terry Watnick, Sambuddho Mukherjee, George Kollias, Georg Schett, Jochen Zwerina and Silvia Hayer and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Nicholas Doerr

15 papers receiving 391 citations

Peers

Countries citing papers authored by Nicholas Doerr

Since Specialization

Citations

This map shows the geographic impact of Nicholas Doerr's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicholas Doerr with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicholas Doerr more than expected).

Fields of papers citing papers by Nicholas Doerr

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicholas Doerr. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicholas Doerr. The network helps show where Nicholas Doerr may publish in the future.

Co-authorship network of co-authors of Nicholas Doerr

This figure shows the co-authorship network connecting the top 25 collaborators of Nicholas Doerr. A scholar is included among the top collaborators of Nicholas Doerr based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicholas Doerr. Nicholas Doerr is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

#	Work	Indexed citations
1	Cleavage fragments of the C-terminal tail of polycystin-1 are regulated by oxidative stress and induce mitochondrial dysfunction 2023 ·Journal of Biological Chemistry ·(unknown), (unknown), Guangyi Liu, Eiko Nishiuchi, Nicholas Doerr, (unknown), (unknown), (unknown), Thomas Weimbs	7
2	Restoration of atypical protein kinase C ζ function in autosomal dominant polycystic kidney disease ameliorates disease progression 2022 ·Proceedings of the National Academy of Sciences ·(unknown), (unknown), Nicholas Doerr, (unknown), (unknown), (unknown), Yidi Wang, Markus M. Rinschen, (unknown), Oliver Wessely, Thomas Weimbs	3
3	Abstract LB143: BNZ-2, a dual specific IL15/IL21 inhibitor, rescues humanized NOG-IL15 transgenic mice from intestinal acute graft versus host disease without disrupting NK and CD8 T cell engraftment 2021 ·Cancer Research ·(unknown), Nicholas Doerr, Laith Q. Al‐Mawsawi, (unknown), Adrian J. Giovannone, Nazli Azimi	1
4	Identification of a γc Receptor Antagonist That Prevents Reprogramming of Human Tissue-resident Cytotoxic T Cells by IL15 and IL21 2019 ·Gastroenterology ·Cezary Ciszewski, Valentina Discepolo, Alain Pacis, Nicholas Doerr, Olivier Tastet, Toufic Mayassi, Mariantonia Maglio, Asjad Basheer, Laith Q. Al‐Mawsawi, Peter H.R. Green, Renata Auricchio, Riccardo Troncone, Thomas A. Waldmann, Nazli Azimi, Yutaka Tagaya, Luis B. Barreiro, Bana Jabrì	25
5	Results From a First‐in‐Human Study of BNZ‐1, a Selective Multicytokine Inhibitor Targeting Members of the Common Gamma (γc) Family of Cytokines 2019 ·The Journal of Clinical Pharmacology ·Paul Frohna, (unknown), Nicholas Doerr, Asjad Basheer, Laith Q. Al‐Mawsawi, (unknown), Juan Carlos Zapata, Xiaorong Wu, Thomas A. Waldmann, Nazli Azimi, Yutaka Tagaya	16
6	Identification of targets of IL-13 and STAT6 signaling in polycystic kidney disease 2018 ·American Journal of Physiology-Renal Physiology ·Erin E. Olsan, (unknown), Jacob A. Torres, Nicholas Doerr, Thomas Weimbs	18
7	B-102 Results from a First-In-Human study with BNZ-1, a novel, selective inhibitor of IL-2, IL-9, and IL-15 at the common gamma-chain receptor, in clinical development for the treatment of HAM/TSP and T-cell malignancies 2018 ·JAIDS Journal of Acquired Immune Deficiency Syndromes ·Paul Frohna, Yutaka Tagaya, (unknown), Nicholas Doerr, Asjad Basheer, Laith Q. Al‐Mawsawi, (unknown), Juan Carlos Zapata	1
8	Results from a First-in-Human Study with Bnz-1: A Novel Peptide Inhibitor of IL-2, IL-9 and IL-15 for the Treatment of T-Cell Malignancies That Safely and Selectively Decreases Regulatory T-Cells, Natural Killer Cells, and CD8+ Central Memory T-Cells 2017 ·Blood ·Paul Frohna, Yutaka Tagaya, (unknown), Nicholas Doerr, Asjad Basheer, Laith Q. Al‐Mawsawi, (unknown), Juan Carlos Zapata, (unknown), Nazli Azimi	5
9	Regulation of Polycystin-1 Function by Calmodulin Binding 2016 ·PLoS ONE ·Nicholas Doerr, Yidi Wang, (unknown), Guangyi Liu, (unknown), В. З. Плетнев, Tengis S. Pavlov, Alexander Staruschenko, Ashraf M. Mohieldin, Maki Takahashi, Surya M. Nauli, Thomas Weimbs	17
10	Bicc1 Polymerization Regulates the Localization and Silencing of Bound mRNA 2015 ·Molecular and Cellular Biology ·Benjamin Rothé, Lucía C. Leal-Esteban, (unknown), (unknown), Nicholas Doerr, Thomas Weimbs, Justyna Iwaszkiewicz, Daniel B. Constam	23
11	The Cleaved Cytoplasmic Tail of Polycystin-1 Regulates Src-Dependent STAT3 Activation 2014 ·Journal of the American Society of Nephrology ·(unknown), Xuewen Song, Xiaofang Wang, Markus M. Rinschen, Nicholas Doerr, Wells B. LaRivière, Bernhard Schermer, York Pei, Vicente E. Torres, Thomas Weimbs	53
12	Polycystin-1 regulates STAT activity by a dual mechanism 2011 ·Proceedings of the National Academy of Sciences ·(unknown), Jonathan M. Shillingford, Shivakumar Vasanth, Nicholas Doerr, Sambuddho Mukherjee, Michael Kinter, Terry Watnick, Thomas Weimbs	103
13	Repair of Local Bone Erosions and Reversal of Systemic Bone Loss Upon Therapy with Anti-Tumor Necrosis Factor in Combination with Osteoprotegerin or Parathyroid Hormone in Tumor Necrosis Factor-Mediated Arthritis 2004 ·American Journal Of Pathology ·Kurt Redlich, Birgit Görtz, Silvia Hayer, Jochen Zwerina, Nicholas Doerr, Paul J. Kostenuik, Helga Bergmeister, George Kollias, G Steiner, Josef S. Smolen, Georg Schett	115
14	Repair of local bone erosions and reversal of systemic bone loss upon therapy with anti-TNF in combination with OPG or PTH in TNF-mediated arthritis 2004 ·Arthritis Research ·Kurt Redlich, Birgit Görtz, Silvia Hayer, Jochen Zwerina, Nicholas Doerr, Paul J. Kostenuik, George Kollias, G Steiner, (unknown), Georg Schett	4
15	[On the biogenesis of isoflavone. Part 2. On the mechanism of migration]. 1960 ·PubMed ·Hans Grisebach, Nicholas Doerr	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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