Ne N. Wu

1.2k total citations · 1 hit paper
16 papers, 899 citations indexed

About

Ne N. Wu is a scholar working on Molecular Biology, Epidemiology and Physiology. According to data from OpenAlex, Ne N. Wu has authored 16 papers receiving a total of 899 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 8 papers in Epidemiology and 4 papers in Physiology. Recurrent topics in Ne N. Wu's work include Autophagy in Disease and Therapy (8 papers), Mitochondrial Function and Pathology (6 papers) and Adipose Tissue and Metabolism (4 papers). Ne N. Wu is often cited by papers focused on Autophagy in Disease and Therapy (8 papers), Mitochondrial Function and Pathology (6 papers) and Adipose Tissue and Metabolism (4 papers). Ne N. Wu collaborates with scholars based in China, United States and Canada. Ne N. Wu's co-authors include Jun Ren, Yingmei Zhang, Yingmei Zhang, James R. Sowers, Shuyi Wang, Yaguang Bi, Dan Wang, Peijie Chen, Amir Ajoolabady and Haili Tian and has published in prestigious journals such as Physiological Reviews, British Journal of Pharmacology and Life Sciences.

In The Last Decade

Ne N. Wu

16 papers receiving 896 citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Obesity cardiomyopathy: evidence, mechanisms, and therapeutic implications

2021 237 citations Jun Ren, Ne N. Wu et al. Physiological Reviews profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Ne N. Wu China	14	461	226	224	177	94	16	899
Shigang Qiao China	20	398 0.9×	220 1.0×	174 0.8×	119 0.7×	93 1.0×	51	1.1k
Mahmoud Abdellatif Austria	18	384 0.8×	263 1.2×	297 1.3×	379 2.1×	90 1.0×	47	1.1k
Hiroaki Sunaga Japan	17	321 0.7×	210 0.9×	101 0.5×	243 1.4×	71 0.8×	33	799
Zhaohui Pei China	18	462 1.0×	200 0.9×	167 0.7×	102 0.6×	108 1.1×	38	889
Luiz Roberto Grassmann Bechara Brazil	20	632 1.4×	372 1.6×	164 0.7×	405 2.3×	47 0.5×	38	1.3k
Dov B. Ballak Netherlands	16	290 0.6×	153 0.7×	245 1.1×	187 1.1×	250 2.7×	25	901
Hideaki Nakatsuji Japan	19	400 0.9×	187 0.8×	376 1.7×	221 1.2×	129 1.4×	37	1.1k
Guangming Yang China	19	410 0.9×	89 0.4×	132 0.6×	177 1.0×	76 0.8×	55	843
Guangda Xiang China	19	432 0.9×	126 0.6×	221 1.0×	369 2.1×	108 1.1×	46	1.1k
Weijing Liu China	14	279 0.6×	118 0.5×	170 0.8×	127 0.7×	31 0.3×	41	703

Countries citing papers authored by Ne N. Wu

Since Specialization

Citations

This map shows the geographic impact of Ne N. Wu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ne N. Wu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ne N. Wu more than expected).

Fields of papers citing papers by Ne N. Wu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ne N. Wu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ne N. Wu. The network helps show where Ne N. Wu may publish in the future.

Co-authorship network of co-authors of Ne N. Wu

This figure shows the co-authorship network connecting the top 25 collaborators of Ne N. Wu. A scholar is included among the top collaborators of Ne N. Wu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ne N. Wu. Ne N. Wu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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16 of 16 papers shown

Wu, Min, Chen Zhao, Jie Lin, et al.. (2024). Ablation of Akt2 rescues chronic caloric restriction-provoked myocardial remodeling and dysfunction through a CDK1-mediated regulation of mitophagy. Life Sciences. 356. 123021–123021. 2 indexed citations

Bi, Yaguang, et al.. (2024). The E3 ubiquitin ligase MARCH2 protects against myocardial ischemia-reperfusion injury through inhibiting pyroptosis via negative regulation of PGAM5/MAVS/NLRP3 axis. Cell Discovery. 10(1). 24–24. 45 indexed citations

Wu, Ne N., Lifeng Wang, Lu Wang, et al.. (2023). Site-specific ubiquitination of VDAC1 restricts its oligomerization and mitochondrial DNA release in liver fibrosis. Experimental & Molecular Medicine. 55(1). 269–280. 37 indexed citations

Xu, Haixia, Wenjun Yu, Mingming Sun, et al.. (2023). Syntaxin17 contributes to obesity cardiomyopathy through promoting mitochondrial Ca2+ overload in a Parkin-MCUb-dependent manner. Metabolism. 143. 155551–155551. 23 indexed citations

Yu, Wei, Lin Wang, Weiying Ren, et al.. (2023). SGLT2 inhibitor empagliflozin alleviates cardiac remodeling and contractile anomalies in a FUNDC1-dependent manner in experimental Parkinson’s disease. Acta Pharmacologica Sinica. 45(1). 87–97. 21 indexed citations

Xu, Haixia, Xiang Wang, Wenjun Yu, et al.. (2023). Syntaxin 17 Protects Against Heart Failure Through Recruitment of CDK1 to Promote DRP1-Dependent Mitophagy. JACC Basic to Translational Science. 8(9). 1215–1239. 11 indexed citations

Chen, Lin, Zhiqiang Yin, Xing Qin, et al.. (2022). CD74 ablation rescues type 2 diabetes mellitus-induced cardiac remodeling and contractile dysfunction through pyroptosis-evoked regulation of ferroptosis. Pharmacological Research. 176. 106086–106086. 45 indexed citations

Wu, Ne N., Yaguang Bi, Amir Ajoolabady, et al.. (2022). Parkin Insufficiency Accentuates High-Fat Diet–Induced Cardiac Remodeling and Contractile Dysfunction Through VDAC1-Mediated Mitochondrial Ca2+ Overload. JACC Basic to Translational Science. 7(8). 779–796. 25 indexed citations

Sun, Shiqun, Wenjun Yu, Haixia Xu, et al.. (2022). TBC1D15-Drp1 interaction-mediated mitochondrial homeostasis confers cardioprotection against myocardial ischemia/reperfusion injury. Metabolism. 134. 155239–155239. 46 indexed citations

10.

Ajoolabady, Amir, Cynthia Lebeaupin, Ne N. Wu, Randal J. Kaufman, & Jun Ren. (2022). ER stress and inflammation crosstalk in obesity. Medicinal Research Reviews. 43(1). 5–30. 63 indexed citations

11.

Ren, Jun, Ne N. Wu, Shuyi Wang, James R. Sowers, & Yingmei Zhang. (2021). Obesity cardiomyopathy: evidence, mechanisms, and therapeutic implications. Physiological Reviews. 101(4). 1745–1807. 237 indexed citations breakdown →

12.

Gong, Yan, Guangwei Li, Jun Tao, et al.. (2020). Double knockout of Akt2 and AMPK accentuates high fat diet-induced cardiac anomalies through a cGAS-STING-mediated mechanism. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1866(10). 165855–165855. 55 indexed citations

13.

Yang, Mingjie, Shuyi Wang, Shouzhi Fu, et al.. (2020). Deletion of the E3 ubiquitin ligase, Parkin, exacerbates chronic alcohol intake‐induced cardiomyopathy through an Ambra1‐dependent mechanism. British Journal of Pharmacology. 178(4). 964–982. 22 indexed citations

14.

Wu, Ne N. & Jun Ren. (2019). Aldehyde Dehydrogenase 2 (ALDH2) and Aging: Is There a Sensible Link?. Advances in experimental medicine and biology. 1193. 237–253. 13 indexed citations

15.

Wu, Ne N., Yingmei Zhang, & Jun Ren. (2019). Mitophagy, Mitochondrial Dynamics, and Homeostasis in Cardiovascular Aging. Oxidative Medicine and Cellular Longevity. 2019. 1–15. 167 indexed citations

16.

Wu, Ne N., Haili Tian, Peijie Chen, et al.. (2019). Physical Exercise and Selective Autophagy: Benefit and Risk on Cardiovascular Health. Cells. 8(11). 1436–1436. 87 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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