Nattapon Panupinthu

3.7k total citations · 1 hit paper
28 papers, 3.0k citations indexed

About

Nattapon Panupinthu is a scholar working on Molecular Biology, Physiology and Cell Biology. According to data from OpenAlex, Nattapon Panupinthu has authored 28 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Molecular Biology, 10 papers in Physiology and 8 papers in Cell Biology. Recurrent topics in Nattapon Panupinthu's work include Adenosine and Purinergic Signaling (9 papers), Sphingolipid Metabolism and Signaling (8 papers) and Fibroblast Growth Factor Research (7 papers). Nattapon Panupinthu is often cited by papers focused on Adenosine and Purinergic Signaling (9 papers), Sphingolipid Metabolism and Signaling (8 papers) and Fibroblast Growth Factor Research (7 papers). Nattapon Panupinthu collaborates with scholars based in United States, Thailand and Canada. Nattapon Panupinthu's co-authors include Gordon B. Mills, Fa‐Xing Yu, G B Mills, Kun‐Liang Guan, Hairi Li, Xiang‐Dong Fu, Karen Tumaneng, Bin Zhao, Jenna L. Jewell and Jiagang Zhao and has published in prestigious journals such as Cell, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Nattapon Panupinthu

28 papers receiving 2.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Regulation of the Hippo-YAP Pathway by G-Protein-Coupled Receptor Signaling

2012 1.3k citations Fa‐Xing Yu, Bin Zhao et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Nattapon Panupinthu United States	20	1.7k	1.3k	508	478	280	28	3.0k
Shu‐Yong Lin China	22	1.9k 1.1×	350 0.3×	323 0.6×	195 0.4×	147 0.5×	35	2.6k
Carl Pelz United States	21	1.4k 0.8×	308 0.2×	412 0.8×	207 0.4×	211 0.8×	37	2.5k
Richard T. Waldron United States	32	2.0k 1.2×	484 0.4×	599 1.2×	127 0.3×	315 1.1×	63	3.2k
Steven W. Paugh United States	18	2.1k 1.2×	708 0.5×	185 0.4×	85 0.2×	536 1.9×	26	2.6k
Shobha Thangada United States	22	4.6k 2.7×	1.7k 1.3×	228 0.4×	139 0.3×	702 2.5×	26	5.2k
Masashi Isshiki Japan	17	2.7k 1.6×	391 0.3×	1.2k 2.4×	207 0.4×	404 1.4×	36	3.8k
Shawn G. Payne United States	20	2.6k 1.5×	800 0.6×	254 0.5×	79 0.2×	371 1.3×	24	2.9k
Randy S. Haun United States	28	1.3k 0.8×	387 0.3×	495 1.0×	165 0.3×	310 1.1×	52	2.2k
Anders Kvanta Sweden	31	1.6k 0.9×	242 0.2×	171 0.3×	150 0.3×	164 0.6×	104	3.6k
Graciela B. Sala‐Newby United Kingdom	30	1.5k 0.9×	316 0.2×	282 0.6×	129 0.3×	403 1.4×	61	2.6k

Countries citing papers authored by Nattapon Panupinthu

Since Specialization

Citations

This map shows the geographic impact of Nattapon Panupinthu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nattapon Panupinthu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nattapon Panupinthu more than expected).

Fields of papers citing papers by Nattapon Panupinthu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nattapon Panupinthu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nattapon Panupinthu. The network helps show where Nattapon Panupinthu may publish in the future.

Co-authorship network of co-authors of Nattapon Panupinthu

This figure shows the co-authorship network connecting the top 25 collaborators of Nattapon Panupinthu. A scholar is included among the top collaborators of Nattapon Panupinthu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nattapon Panupinthu. Nattapon Panupinthu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Suntornsaratoon, Panan, Narisorn Kitiyanant, Jarinthorn Teerapornpuntakit, et al.. (2024). Fibroblast growth factor-21 potentiates the stimulatory effects of 1,25-dihydroxyvitamin D3 on transepithelial calcium transport and TRPV6 Ca2+ channel expression. Biochemical and Biophysical Research Communications. 733. 150429–150429. 2 indexed citations

Thongbunchoo, Jirawan, et al.. (2023). Extracellular Fe2+ and Fe3+ modulate osteocytic viability, expression of SOST, RANKL and FGF23, and fluid flow-induced YAP1 nuclear translocation. Scientific Reports. 13(1). 21173–21173. 1 indexed citations

Panupinthu, Nattapon, et al.. (2022). Fe3+ opposes the 1,25(OH)2D3-induced calcium transport across intestinal epithelium-like Caco-2 monolayer in the presence or absence of ascorbic acid. PLoS ONE. 17(8). e0273267–e0273267. 7 indexed citations

Charoenphandhu, Narattaphol, Nattapon Panupinthu, Jarinthorn Teerapornpuntakit, et al.. (2022). Mild-intensity physical activity prevents cardiac and osseous iron deposition without affecting bone mechanical property or porosity in thalassemic mice. Scientific Reports. 12(1). 5959–5959. 2 indexed citations

Wongdee, Kannikar, et al.. (2021). Excessive salt consumption causes systemic calcium mishandling and worsens microarchitecture and strength of long bones in rats. Scientific Reports. 11(1). 1850–1850. 20 indexed citations

Teerapornpuntakit, Jarinthorn, et al.. (2021). CFTR-mediated anion secretion in parathyroid hormone-treated Caco-2 cells is associated with PKA and PI3K phosphorylation but not intracellular pH changes or Na+/K+-ATPase abundance. Biochemistry and Biophysics Reports. 27. 101054–101054. 2 indexed citations

Wongdee, Kannikar, Nattapon Panupinthu, Jirawan Thongbunchoo, et al.. (2018). Prolonged exposure to 1,25(OH)2D3 and high ionized calcium induces FGF-23 production in intestinal epithelium-like Caco-2 monolayer: A local negative feedback for preventing excessive calcium transport. Archives of Biochemistry and Biophysics. 640. 10–16. 19 indexed citations

Wang, Chao, Chao Gu, Kang Jin Jeong, et al.. (2017). YAP/TAZ-Mediated Upregulation of GAB2 Leads to Increased Sensitivity to Growth Factor–Induced Activation of the PI3K Pathway. Cancer Research. 77(7). 1637–1648. 43 indexed citations

Cheung, Lydia W.T., Shuangxing Yu, Dong Zhang, et al.. (2014). Naturally Occurring Neomorphic PIK3R1 Mutations Activate the MAPK Pathway, Dictating Therapeutic Response to MAPK Pathway Inhibitors. Cancer Cell. 26(4). 479–494. 71 indexed citations

10.

Hartman, Zachary C., Graham M. Poage, Petra den Hollander, et al.. (2013). Growth of Triple-Negative Breast Cancer Cells Relies upon Coordinate Autocrine Expression of the Proinflammatory Cytokines IL-6 and IL-8. Cancer Research. 73(11). 3470–3480. 341 indexed citations

11.

Panupinthu, Nattapon, Mihai Gagea, Yiling Lu, et al.. (2013). Self-reinforcing loop of amphiregulin and Y-box binding protein-1 contributes to poor outcomes in ovarian cancer. Oncogene. 33(22). 2846–2856. 45 indexed citations

12.

Jeong, Kang Jin, Kyung Hwa Cho, Nattapon Panupinthu, et al.. (2012). EGFR mediates LPA‐induced proteolytic enzyme expression and ovarian cancer invasion: Inhibition by resveratrol. Molecular Oncology. 7(1). 121–129. 30 indexed citations

13.

Yu, Fa‐Xing, Bin Zhao, Nattapon Panupinthu, et al.. (2012). Regulation of the Hippo-YAP Pathway by G-Protein-Coupled Receptor Signaling. Cell. 150(4). 780–791. 1282 indexed citations breakdown →

14.

Patil, Renukadevi, Jianxiong Liu, Yaohong Wang, et al.. (2011). Benzyl and Naphthalene Methylphosphonic Acid Inhibitors of Autotaxin with Anti‐invasive and Anti‐metastatic Activity. ChemMedChem. 6(5). 922–935. 51 indexed citations

15.

Lü, Yan, Wěi Li, Yuko Fujiwara, et al.. (2010). Synthesis and pharmacological evaluation of the stereoisomers of 3-carba cyclic-phosphatidic acid. Bioorganic & Medicinal Chemistry Letters. 20(24). 7525–7528. 20 indexed citations

16.

Panupinthu, Nattapon, et al.. (2010). Lysophosphatidic acid production and action: critical new players in breast cancer initiation and progression. British Journal of Cancer. 102(6). 941–946. 107 indexed citations

17.

Jeong, Kangjin, Nattapon Panupinthu, Jeongwoo Han, et al.. (2010). Lysophosphatidic acid augments human hepatocellular carcinoma cell invasion through LPA1 receptor and MMP-9 expression. Oncogene. 30(11). 1351–1359. 121 indexed citations

18.

Grol, Matthew W., Nattapon Panupinthu, Jasminka Korčok, Stephen M. Sims, & S. Jeffrey Dixon. (2009). Expression, signaling, and function of P2X7 receptors in bone. Purinergic Signalling. 5(2). 205–221. 88 indexed citations

19.

Dixon, S. Jeffrey, Rong-Guo Yu, Nattapon Panupinthu, & John X. Wilson. (2004). Activation of P2 nucleotide receptors stimulates acid efflux from astrocytes. Glia. 47(4). 367–376. 32 indexed citations

20.

Ke, Hua Zhu, Hong Qi, Qing Zhang, et al.. (2003). Deletion of the P2X₇Nucleotide Receptor Reveals Its Regulatory Roles in Bone Formation and Resorption. Molecular Endocrinology. 17(7). 1356–1367. 229 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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