Natsuko Nakamura
- Molecular Biology
- Ophthalmology top 5%
- Immunology
- Radiology, Nuclear Medicine and Imaging
- Cell Biology
- Co-authors
- Kazushige TsunodaBruce YongNobuko KawasakiToshisuke KawasakiMotohiro NonakaMegumi HonjoMakoto AiharaReiko Yamagishi
- Topics
- Retinal Diseases and Treatments (10 papers)Retinal Development and Disorders (9 papers)Glaucoma and retinal disorders (7 papers)
- Cited by
- OphthalmologyImmunologyNeurology
- Partner nations
- JapanUnited KingdomChina
In The Last Decade
Natsuko Nakamura
28 papers receiving 394 citations
Peers
Comparison fields: 5 of 60
- Molecular Biology 241
- Ophthalmology 158
- Immunology 103
- Radiology, Nuclear Medicine and Imaging 51
- Cell Biology 43
Countries citing papers authored by Natsuko Nakamura
This map shows the geographic impact of Natsuko Nakamura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natsuko Nakamura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natsuko Nakamura more than expected).
Fields of papers citing papers by Natsuko Nakamura
This network shows the impact of papers produced by Natsuko Nakamura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natsuko Nakamura. The network helps show where Natsuko Nakamura may publish in the future.
Co-authorship network of co-authors of Natsuko Nakamura
This figure shows the co-authorship network connecting the top 25 collaborators of Natsuko Nakamura. A scholar is included among the top collaborators of Natsuko Nakamura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natsuko Nakamura. Natsuko Nakamura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 4 | |
| 4 | 5 | |
| 5 | 2 | |
| 6 | Effects of topical TGF-β1, TGF-β2, ATX, and LPA on IOP elevation and regulation of the conventional aqueous humor outflow pathway. | 14 |
| 7 | 12 | |
| 8 | 2 | |
| 9 | 23 | |
| 10 | 26 | |
| 11 | 19 | |
| 12 | 21 | |
| 13 | 10 | |
| 14 | 23 | |
| 15 | 1 | |
| 16 | 19 | |
| 17 | 21 | |
| 18 | 73 | |
| 19 | 5 | |
| 20 | 2 |
About Natsuko Nakamura
Natsuko Nakamura is a scholar working on Ophthalmology, Cell Biology and Cellular and Molecular Neuroscience, having authored 29 papers that have together received 399 indexed citations. Recurring topics across this work include Retinal Diseases and Treatments (10 papers), Retinal Development and Disorders (9 papers) and Glaucoma and retinal disorders (7 papers). The work is most often cited by research in Ophthalmology (158 citations), Immunology (103 citations) and Neurology (32 citations). Natsuko Nakamura has collaborated with scholars based in Japan, United Kingdom and China. Frequent co-authors include Kazushige Tsunoda, Bruce Yong, Nobuko Kawasaki, Toshisuke Kawasaki, Motohiro Nonaka, Megumi Honjo, Makoto Aihara, Reiko Yamagishi, Makoto Baba and Shinji Asano. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.