Nathan D. Okerlund

603 citations
7 papers · 420 indexed · h-index 6
Topics
Wnt/β-catenin signaling in development and cancer (3 papers)Autophagy in Disease and Therapy (2 papers)Cellular transport and secretion (2 papers)

In The Last Decade

Nathan D. Okerlund

7 papers receiving 419 citations

Peers

Nathan D. Okerlund
Comparison fields: 5 of 72
  • Molecular Biology 205
  • Cell Biology 137
  • Cellular and Molecular Neuroscience 125
  • Epidemiology 115
  • Genetics 90
Replace Xiaoting Wu with:
Xiaoting Wu China
Harry Samaroo United States
Asami Oguro‐Ando Japan
Frauke Ackermann Germany
Mengchen Ye United States
Marie Y. Davis United States
Sean E. Low United States
Laura Sancho United States
Maria De Risi Italy
Mazdak M. Bradberry United States
Nathan D. Okerlund relative to Xiaoting Wu China Xiaoting Wu's profile →
Citations per field
00.5×3.3×
Xiaoting Wu · 1×
Citations per year

Countries citing papers authored by Nathan D. Okerlund

Since Specialization
Citations

This map shows the geographic impact of Nathan D. Okerlund's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan D. Okerlund with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan D. Okerlund more than expected).

Fields of papers citing papers by Nathan D. Okerlund

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan D. Okerlund. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan D. Okerlund. The network helps show where Nathan D. Okerlund may publish in the future.

Co-authorship network of co-authors of Nathan D. Okerlund

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan D. Okerlund. A scholar is included among the top collaborators of Nathan D. Okerlund based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan D. Okerlund. Nathan D. Okerlund is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
#WorkIndexed citations
1 1
2 143
3 14
4 105
5 114
6 25
7 18

About Nathan D. Okerlund

Nathan D. Okerlund is a scholar working on Aging, Cell Biology and Electrochemistry, having authored 7 papers that have together received 420 indexed citations. Recurring topics across this work include Wnt/β-catenin signaling in development and cancer (3 papers), Autophagy in Disease and Therapy (2 papers) and Cellular transport and secretion (2 papers). The work is most often cited by research in Cell Biology (137 citations), Cellular and Molecular Neuroscience (125 citations) and Physiology (26 citations). Nathan D. Okerlund has collaborated with scholars based in United States, Germany and Netherlands. Frequent co-authors include Benjamin Cheyette, Sergio Leal‐Ortiz, Craig C. Garner, Clarissa L. Waites, Eckart D. Gundelfinger, Katharina Schneider, Sally A. Kim, Richard J. Reimer, Carolina Montenegro‐Venegas and Wilko D. Altrock. Their work appears in journals such as Neuron, Journal of Neuroscience and The EMBO Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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