Natasha Kamal

504 citations
20 papers · 311 indexed · h-index 9
Topics
Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers)Pediatric Hepatobiliary Diseases and Treatments (3 papers)Autoimmune and Inflammatory Disorders (3 papers)

In The Last Decade

Natasha Kamal

20 papers receiving 308 citations

Peers

Natasha Kamal
Comparison fields: 5 of 54
  • Immunology 131
  • Surgery 103
  • Epidemiology 93
  • Genetics 87
  • Molecular Biology 45
Replace Cindy S. Bauer with:
Cindy S. Bauer United States
Christina Boros Australia
AJ Cant United Kingdom
Abolhassan Farhoudi Iran
Willem S. Lexmond Netherlands
M. A. Sattar Kuwait
Hack-Ki Kim South Korea
Lizbeth Blancas‐Galicia Mexico
Elisabeth E. Nibbelke Netherlands
Samantha Zullow United States
Natasha Kamal relative to Cindy S. Bauer United States Cindy S. Bauer's profile →
Citations per field
00.5×2.8×
Cindy S. Bauer · 1×
Citations per year

Countries citing papers authored by Natasha Kamal

Since Specialization
Citations

This map shows the geographic impact of Natasha Kamal's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natasha Kamal with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natasha Kamal more than expected).

Fields of papers citing papers by Natasha Kamal

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Natasha Kamal. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natasha Kamal. The network helps show where Natasha Kamal may publish in the future.

Co-authorship network of co-authors of Natasha Kamal

This figure shows the co-authorship network connecting the top 25 collaborators of Natasha Kamal. A scholar is included among the top collaborators of Natasha Kamal based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natasha Kamal. Natasha Kamal is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 7
2 4
3 23
4 7
5 2
6 12
7 1
8 4
9 36
10 1
11 36
12 1
13 20
14 31
15 40
16 7
17 2
18 1
19 10
20 66

About Natasha Kamal

Natasha Kamal is a scholar working on Hepatology, Immunology and Epidemiology, having authored 20 papers that have together received 311 indexed citations. Recurring topics across this work include Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers), Pediatric Hepatobiliary Diseases and Treatments (3 papers) and Autoimmune and Inflammatory Disorders (3 papers). The work is most often cited by research in Immunology (131 citations), Dermatology (33 citations) and Genetics (87 citations). Natasha Kamal has collaborated with scholars based in United States, Australia and France. Frequent co-authors include Christopher Koh, Pallavi Surana, Theo Heller, Harry L. Malech, Steven M. Holland, John I. Gallin, Martha Quezado, Benjamin L. Cohen, Meghna Alimchandani and Éric Delaporte. Their work appears in journals such as Gastroenterology, Clinical Infectious Diseases and The American Journal of Gastroenterology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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