Natalia Gromak

5.2k total citations · 1 hit paper
31 papers, 3.6k citations indexed

About

Natalia Gromak is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cancer Research. According to data from OpenAlex, Natalia Gromak has authored 31 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Molecular Biology, 6 papers in Cellular and Molecular Neuroscience and 5 papers in Cancer Research. Recurrent topics in Natalia Gromak's work include RNA Research and Splicing (17 papers), DNA Repair Mechanisms (13 papers) and RNA modifications and cancer (11 papers). Natalia Gromak is often cited by papers focused on RNA Research and Splicing (17 papers), DNA Repair Mechanisms (13 papers) and RNA modifications and cancer (11 papers). Natalia Gromak collaborates with scholars based in United Kingdom, United States and France. Natalia Gromak's co-authors include Nicholas Proudfoot, Matthias F. Groh, Konstantina Skourti-Stathaki, Steven West, Agnese Cristini, Maiken Søndergaard Kristiansen, Michele MP Lufino, Richard Wade‐Martins, Monica Ballarino and Francesca Pagano and has published in prestigious journals such as Nature, Science and Nucleic Acids Research.

In The Last Decade

Natalia Gromak

31 papers receiving 3.6k citations

Hit Papers

Human Senataxin Resolves ... 2011 2026 2016 2021 2011 200 400 600

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Natalia Gromak United Kingdom 22 3.4k 566 304 287 219 31 3.6k
Kathi Zarnack Germany 29 3.6k 1.1× 756 1.3× 126 0.4× 91 0.3× 238 1.1× 73 3.9k
Christopher C. Ebmeier United States 21 2.9k 0.9× 224 0.4× 368 1.2× 264 0.9× 401 1.8× 53 3.4k
Behnam Nabet United States 22 3.1k 0.9× 356 0.6× 179 0.6× 619 2.2× 174 0.8× 41 3.6k
Warren A. Whyte United States 6 3.3k 1.0× 537 0.9× 410 1.3× 240 0.8× 199 0.9× 7 3.7k
Konstantina Skourti-Stathaki United Kingdom 9 1.9k 0.6× 233 0.4× 211 0.7× 177 0.6× 338 1.5× 11 2.1k
Lawryn H. Kasper United States 22 2.7k 0.8× 387 0.7× 320 1.1× 501 1.7× 64 0.3× 26 3.6k
Jonathan D. Chesnut United States 22 2.1k 0.6× 242 0.4× 492 1.6× 170 0.6× 154 0.7× 38 2.4k
Tatiana García‐Muse Spain 16 3.1k 0.9× 357 0.6× 368 1.2× 549 1.9× 348 1.6× 19 3.3k
Saı̈d Sif United States 36 5.4k 1.6× 403 0.7× 549 1.8× 542 1.9× 164 0.7× 58 6.0k

Countries citing papers authored by Natalia Gromak

Since Specialization
Citations

This map shows the geographic impact of Natalia Gromak's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalia Gromak with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalia Gromak more than expected).

Fields of papers citing papers by Natalia Gromak

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Natalia Gromak. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalia Gromak. The network helps show where Natalia Gromak may publish in the future.

Co-authorship network of co-authors of Natalia Gromak

This figure shows the co-authorship network connecting the top 25 collaborators of Natalia Gromak. A scholar is included among the top collaborators of Natalia Gromak based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natalia Gromak. Natalia Gromak is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Xhemalçe, Blerta, Kyle M. Miller, & Natalia Gromak. (2024). Epitranscriptome in action: RNA modifications in the DNA damage response. Molecular Cell. 84(19). 3610–3626. 7 indexed citations
2.
Wang, Eric T., Catherine H. Freudenreich, Natalia Gromak, et al.. (2023). What repeat expansion disorders can teach us about the Central Dogma. Molecular Cell. 83(3). 324–329. 2 indexed citations
3.
Cristini, Agnese, Michael Tellier, Laura-Oana Albulescu, et al.. (2022). RNase H2, mutated in Aicardi‐Goutières syndrome, resolves co-transcriptional R-loops to prevent DNA breaks and inflammation. Nature Communications. 13(1). 2961–2961. 54 indexed citations
4.
Gromak, Natalia, et al.. (2022). R-Loop Immunoprecipitation: A Method to Detect R-Loop Interacting Factors. Methods in molecular biology. 2528. 215–237. 5 indexed citations
5.
Ramachandran, Shaliny, Jon Griffin, Iosifina P. Foskolou, et al.. (2021). Hypoxia-induced SETX links replication stress with the unfolded protein response. Nature Communications. 12(1). 3686–3686. 30 indexed citations
6.
Cristini, Agnese, Giulia Ricci, Sébastien Britton, et al.. (2019). Dual Processing of R-Loops and Topoisomerase I Induces Transcription-Dependent DNA Double-Strand Breaks. Cell Reports. 28(12). 3167–3181.e6. 119 indexed citations
7.
Cristini, Agnese, Matthias F. Groh, Maiken Søndergaard Kristiansen, & Natalia Gromak. (2018). RNA/DNA Hybrid Interactome Identifies DXH9 as a Molecular Player in Transcriptional Termination and R-Loop-Associated DNA Damage. Cell Reports. 23(6). 1891–1905. 289 indexed citations
8.
Gromak, Natalia, et al.. (2018). Large XPF-dependent deletions following misrepair of a DNA double strand break are prevented by the RNA:DNA helicase Senataxin. Scientific Reports. 8(1). 3850–3850. 24 indexed citations
9.
Kotsantis, Panagiotis, Sarah Irmscher, Rebecca M. Jones, et al.. (2016). Increased global transcription activity as a mechanism of replication stress in cancer. Nature Communications. 7(1). 13087–13087. 226 indexed citations
10.
Groh, Matthias F., Laura-Oana Albulescu, Agnese Cristini, & Natalia Gromak. (2016). Senataxin: Genome Guardian at the Interface of Transcription and Neurodegeneration. Journal of Molecular Biology. 429(21). 3181–3195. 101 indexed citations
11.
Groh, Matthias F., Michele MP Lufino, Richard Wade‐Martins, & Natalia Gromak. (2014). R-loops Associated with Triplet Repeat Expansions Promote Gene Silencing in Friedreich Ataxia and Fragile X Syndrome. PLoS Genetics. 10(5). e1004318–e1004318. 269 indexed citations
12.
Groh, Matthias F. & Natalia Gromak. (2014). Out of Balance: R-loops in Human Disease. PLoS Genetics. 10(9). e1004630–e1004630. 145 indexed citations
13.
Gromak, Natalia, Martin Dienstbier, Sara Macías, et al.. (2013). Drosha Regulates Gene Expression Independently of RNA Cleavage Function. Cell Reports. 5(6). 1499–1510. 54 indexed citations
14.
Webby, Celia J., Alexander Wolf, Natalia Gromak, et al.. (2009). Jmjd6 Catalyses Lysyl-Hydroxylation of U2AF65, a Protein Associated with RNA Splicing. Science. 325(5936). 90–93. 315 indexed citations
15.
Dye, Michael J., Natalia Gromak, & Nicholas Proudfoot. (2006). Exon Tethering in Transcription by RNA Polymerase II. Molecular Cell. 21(6). 849–859. 122 indexed citations
16.
Dye, Michael J., Natalia Gromak, Dirk Haussecker, Steven West, & Nicholas Proudfoot. (2006). Turnover and Function of Noncoding RNA Polymerase II Transcripts. Cold Spring Harbor Symposia on Quantitative Biology. 71(0). 275–284. 3 indexed citations
17.
West, Steven, Natalia Gromak, & Nicholas Proudfoot. (2004). Human 5′ → 3′ exonuclease Xrn2 promotes transcription termination at co-transcriptional cleavage sites. Nature. 432(7016). 522–525. 373 indexed citations
18.
Gromak, Natalia, et al.. (2003). Antagonistic regulation of α-actinin alternative splicing by CELF proteins and polypyrimidine tract binding protein. RNA. 9(4). 443–456. 91 indexed citations
19.
Gromak, Natalia. (2003). The PTB interacting protein raver1 regulates  -tropomyosin alternative splicing. The EMBO Journal. 22(23). 6356–6364. 94 indexed citations
20.
Gromak, Natalia. (2002). A splicing silencer that regulates smooth muscle specific alternative splicing is active in multiple cell types. Nucleic Acids Research. 30(16). 3548–3557. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026