Naoko Tsukamoto
- Molecular Biology
- Public Health, Environmental and Occupational Health
- Endocrinology, Diabetes and Metabolism top 10%
- Reproductive Medicine top 10%
- Endocrine and Autonomic Systems top 10%
- Co-authors
- Fumio OtsukaHirofumi MakinoKenichi InagakiTomoko MiyoshiMasaya TakedaEri NakamuraYoshinori MatsumotoJirô Suzuki
- Topics
- Growth Hormone and Insulin-like Growth Factors (6 papers)Reproductive Biology and Fertility (5 papers)TGF-β signaling in diseases (4 papers)
- Partner nations
- JapanUnited States
In The Last Decade
Naoko Tsukamoto
18 papers receiving 398 citations
Peers
Comparison fields: 5 of 60
- Molecular Biology 200
- Public Health, Environmental and Occupational Health 119
- Endocrinology, Diabetes and Metabolism 98
- Reproductive Medicine 96
- Endocrine and Autonomic Systems 73
Countries citing papers authored by Naoko Tsukamoto
This map shows the geographic impact of Naoko Tsukamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naoko Tsukamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naoko Tsukamoto more than expected).
Fields of papers citing papers by Naoko Tsukamoto
This network shows the impact of papers produced by Naoko Tsukamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naoko Tsukamoto. The network helps show where Naoko Tsukamoto may publish in the future.
Co-authorship network of co-authors of Naoko Tsukamoto
This figure shows the co-authorship network connecting the top 25 collaborators of Naoko Tsukamoto. A scholar is included among the top collaborators of Naoko Tsukamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naoko Tsukamoto. Naoko Tsukamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 51 | |
| 2 | 26 | |
| 3 | 30 | |
| 4 | 31 | |
| 5 | 8 | |
| 6 | 11 | |
| 7 | 26 | |
| 8 | 17 | |
| 9 | 6 | |
| 10 | 17 | |
| 11 | 17 | |
| 12 | 40 | |
| 13 | 32 | |
| 14 | 35 | |
| 15 | 12 | |
| 16 | 39 | |
| 17 | 5 | |
| 18 | 2 |
About Naoko Tsukamoto
Naoko Tsukamoto is a scholar working on Behavioral Neuroscience, Endocrinology, Diabetes and Metabolism and Reproductive Medicine, having authored 18 papers that have together received 405 indexed citations. Recurring topics across this work include Growth Hormone and Insulin-like Growth Factors (6 papers), Reproductive Biology and Fertility (5 papers) and TGF-β signaling in diseases (4 papers). The work is most often cited by research in Reproductive Medicine (96 citations), Endocrine and Autonomic Systems (73 citations) and Endocrinology, Diabetes and Metabolism (98 citations). Naoko Tsukamoto has collaborated with scholars based in Japan and United States. Frequent co-authors include Fumio Otsuka, Hirofumi Makino, Kenichi Inagaki, Tomoko Miyoshi, Masaya Takeda, Eri Nakamura, Eri Nakamura, Yoshinori Matsumoto, Jirô Suzuki and Yasumasa Iwasaki. Their work appears in journals such as Endocrinology, Clinical Chemistry and Molecular and Cellular Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.