Naofumi Yui

883 total citations
17 papers, 629 citations indexed

About

Naofumi Yui is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Nephrology. According to data from OpenAlex, Naofumi Yui has authored 17 papers receiving a total of 629 indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 5 papers in Pulmonary and Respiratory Medicine and 2 papers in Nephrology. Recurrent topics in Naofumi Yui's work include Ion Transport and Channel Regulation (13 papers), Electrolyte and hormonal disorders (5 papers) and Ion channel regulation and function (4 papers). Naofumi Yui is often cited by papers focused on Ion Transport and Channel Regulation (13 papers), Electrolyte and hormonal disorders (5 papers) and Ion channel regulation and function (4 papers). Naofumi Yui collaborates with scholars based in Japan, United States and Switzerland. Naofumi Yui's co-authors include Sei Sasaki, Shinichi Uchida, Dennis Brown, Tatemitsu Rai, Richard Bouley, Yumi Noda, Eisei Sohara, Hilary P. Stevenson, Jennifer King and Matthew J. Webber and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and The Journal of Physiology.

In The Last Decade

Naofumi Yui

17 papers receiving 620 citations

Peers

Countries citing papers authored by Naofumi Yui

Since Specialization

Citations

This map shows the geographic impact of Naofumi Yui's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Naofumi Yui with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Naofumi Yui more than expected).

Fields of papers citing papers by Naofumi Yui

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Naofumi Yui. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Naofumi Yui. The network helps show where Naofumi Yui may publish in the future.

Co-authorship network of co-authors of Naofumi Yui

This figure shows the co-authorship network connecting the top 25 collaborators of Naofumi Yui. A scholar is included among the top collaborators of Naofumi Yui based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Naofumi Yui. Naofumi Yui is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

#	Work	Indexed citations
1	LRBA signalosomes activate vasopressin‐induced AQP2 trafficking at recycling endosomes 2023 ·The Journal of Physiology ·(unknown), (unknown), Fumiaki Ando, (unknown), (unknown), Daisuke Oikawa, Naofumi Yui, Shintaro Mandai, Yutaro Mori, Koichiro Susa, Takayasu Mori, Eisei Sohara, Fuminori Tokunaga, Shinichi Uchida	4
2	Chlorpromazine Induces Basolateral Aquaporin-2 Accumulation via F-Actin Depolymerization and Blockade of Endocytosis in Renal Epithelial Cells 2020 ·Cells ·Richard Bouley, Naofumi Yui, (unknown), (unknown), Dennis Brown	21
3	AKAPs-PKA disruptors increase AQP2 activity independently of vasopressin in a model of nephrogenic diabetes insipidus 2018 ·Nature Communications ·Fumiaki Ando, Shuichi Mori, Naofumi Yui, Tetsuji Morimoto, Naohiro Nomura, Eisei Sohara, Tatemitsu Rai, Sei Sasaki, Yoshiaki Kondo, Hiroyuki Kagechika, Shinichi Uchida	34
4	Ezrin directly interacts with AQP2 and promotes its endocytosis 2017 ·Journal of Cell Science ·Wei Li, William Jin, Kenji Tsuji, (unknown), Naohiro Nomura, (unknown), Naofumi Yui, (unknown), Susanna Cotecchia, Teodor G. Păunescu, Dennis Brown, Hua Lu	30
5	Ser-261 phospho-regulation is involved in pS256 and pS269-mediated aquaporin-2 apical translocation 2017 ·Biochemical and Biophysical Research Communications ·Naofumi Yui, Fumiaki Ando, Sei Sasaki, Shinichi Uchida	13
6	The proteasome inhibitor bortezomib attenuates renal fibrosis in mice via the suppression of TGF-β1 2017 ·Scientific Reports ·Moko Zeniya, Takayasu Mori, Naofumi Yui, Naohiro Nomura, Shintaro Mandai, Kiyoshi Isobe, Motoko Chiga, Eisei Sohara, Tatemitsu Rai, Shinichi Uchida	34
7	Aquaporin-2 Ser-261 phosphorylation is regulated in combination with Ser-256 and Ser-269 phosphorylation 2016 ·Biochemical and Biophysical Research Communications ·Naofumi Yui, Sei Sasaki, Shinichi Uchida	15
8	Wnt5a induces renal AQP2 expression by activating calcineurin signalling pathway 2016 ·Nature Communications ·Fumiaki Ando, Eisei Sohara, Tetsuji Morimoto, Naofumi Yui, Naohiro Nomura, Eriko Kikuchi, Daiei Takahashi, Takayasu Mori, Alain Vandewalle, Tatemitsu Rai, Sei Sasaki, Yoshiaki Kondo, Shinichi Uchida	52
9	Bence-Jones Protein λ-type Multiple Myeloma Patient Withdrawn from Maintenance Hemodialysis after Long-term Bortezomib and Dexamethasone Therapy 2016 ·Internal Medicine ·(unknown), Naofumi Yui, Toshikage Nagao, Gaku Oshikawa, Mariko Negi, Soichiro Iimori, Tomokazu Okado, Tatemitsu Rai, Shinichi Uchida	1
10	Anaemia management and mortality risk in newly visiting patients with chronic kidney disease in Japan: The CKD‐ROUTE study 2015 ·Nephrology ·Soichiro Iimori, Shotaro Naito, Yumi Noda, Hidenori Nishida, (unknown), Naofumi Yui, Tomokazu Okado, Sei Sasaki, Shinichi Uchida, Tatemitsu Rai	46
11	Two Cases of Hemodialysis-associated Chronic Portal-systemic Shunt Encephalopathy (CPSE) with Opposite Changes in the Blood Ammonia Concentrations during Hemodialysis: A Case Report and Literature Review 2015 ·Internal Medicine ·(unknown), Tomokazu Okado, (unknown), Soichiro Iimori, Naofumi Yui, Eisei Sohara, Eiichiro Kanda, Tatemitsu Rai, Sei Sasaki, Shinichi Uchida	4
12	Treatment with 17-allylamino-17-demethoxygeldanamycin ameliorated symptoms of Bartter syndrome type IV caused by mutated Bsnd in mice 2013 ·Biochemical and Biophysical Research Communications ·Naohiro Nomura, Kazusaku Kamiya, Katsuhisa Ikeda, Naofumi Yui, Motoko Chiga, Eisei Sohara, (unknown), (unknown), (unknown)	11
13	Noncanonical Control of Vasopressin Receptor Type 2 Signaling by Retromer and Arrestin 2013 ·Journal of Biological Chemistry ·Timothy N. Feinstein, Naofumi Yui, Matthew J. Webber, Vanessa L. Wehbi, Hilary P. Stevenson, Jennifer King, Kenneth R. Hallows, Dennis Brown, Richard Bouley, Jean‐Pierre Vilardaga	157
14	Actin directly interacts with different membrane channel proteins and influences channel activities: AQP2 as a model 2013 ·Biochimica et Biophysica Acta (BBA) - Biomembranes ·Sei Sasaki, Naofumi Yui, Yumi Noda	59
15	AQP2 is necessary for vasopressin- and forskolin-mediated filamentous actin depolymerization in renal epithelial cells 2011 ·Biology Open ·Naofumi Yui, Hua Lu, Richard Bouley, Dennis Brown	31
16	Targeted disruption of the Wnk4 gene decreases phosphorylation of Na-Cl cotransporter, increases Na excretion and lowers blood pressure 2009 ·Human Molecular Genetics ·Akihito Ohta, Tatemitsu Rai, Naofumi Yui, Motoko Chiga, Sung‐Sen Yang, Shih‐Hua Lin, Eisei Sohara, Sei Sasaki, Shinichi Uchida	96
17	FAPP2 is required for aquaporin-2 apical sorting at trans-Golgi network in polarized MDCK cells 2009 ·American Journal of Physiology-Cell Physiology ·Naofumi Yui, (unknown), Eisei Sohara, Tatemitsu Rai, Akihito Ohta, Yumi Noda, Sei Sasaki, Shinichi Uchida	21

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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