Nadine Waldschmitt

5.4k total citations
17 papers, 763 citations indexed

About

Nadine Waldschmitt is a scholar working on Molecular Biology, Epidemiology and Genetics. According to data from OpenAlex, Nadine Waldschmitt has authored 17 papers receiving a total of 763 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 6 papers in Epidemiology and 6 papers in Genetics. Recurrent topics in Nadine Waldschmitt's work include Inflammatory Bowel Disease (6 papers), Gut microbiota and health (5 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Nadine Waldschmitt is often cited by papers focused on Inflammatory Bowel Disease (6 papers), Gut microbiota and health (5 papers) and Endoplasmic Reticulum Stress and Disease (3 papers). Nadine Waldschmitt collaborates with scholars based in Germany, France and Switzerland. Nadine Waldschmitt's co-authors include Dirk Haller, Eva Rath, Emanuel Berger, Pieter Giesbertz, Amira Metwaly, Sevana Khaloian, Mathias Chamaillard, Matthieu Allez, Mathias Heikenwälder and Klaus‐Peter Janssen and has published in prestigious journals such as Nature Communications, Gastroenterology and Cancer Research.

In The Last Decade

Nadine Waldschmitt

16 papers receiving 754 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nadine Waldschmitt Germany 11 447 162 153 122 112 17 763
Lukas Niederreiter Austria 11 264 0.6× 126 0.8× 192 1.3× 65 0.5× 240 2.1× 17 677
Mikihiro Fujiya Japan 13 354 0.8× 171 1.1× 60 0.4× 156 1.3× 104 0.9× 60 790
Zili Lei China 18 540 1.2× 128 0.8× 79 0.5× 108 0.9× 77 0.7× 48 948
Suzana D. Savkovic United States 20 564 1.3× 156 1.0× 234 1.5× 90 0.7× 75 0.7× 39 1.2k
Lei Hou China 18 491 1.1× 252 1.6× 233 1.5× 61 0.5× 147 1.3× 46 1.1k
Bara Sarraj United States 12 345 0.8× 188 1.2× 303 2.0× 63 0.5× 46 0.4× 12 896
Simone Spieckermann Germany 5 310 0.7× 123 0.8× 176 1.2× 93 0.8× 70 0.6× 7 758
Shien Hu United States 17 664 1.5× 119 0.7× 54 0.4× 139 1.1× 84 0.8× 25 1.0k
Lien Van den Bossche Belgium 7 299 0.7× 169 1.0× 121 0.8× 135 1.1× 80 0.7× 14 615
Yonatan Herzig Israel 5 786 1.8× 95 0.6× 270 1.8× 54 0.4× 87 0.8× 6 1.0k

Countries citing papers authored by Nadine Waldschmitt

Since Specialization
Citations

This map shows the geographic impact of Nadine Waldschmitt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadine Waldschmitt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadine Waldschmitt more than expected).

Fields of papers citing papers by Nadine Waldschmitt

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadine Waldschmitt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadine Waldschmitt. The network helps show where Nadine Waldschmitt may publish in the future.

Co-authorship network of co-authors of Nadine Waldschmitt

This figure shows the co-authorship network connecting the top 25 collaborators of Nadine Waldschmitt. A scholar is included among the top collaborators of Nadine Waldschmitt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadine Waldschmitt. Nadine Waldschmitt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Chauvin, Camille, Katarina Radulović, Olivier Boulard, et al.. (2023). Loss of NOD2 in macrophages improves colitis and tumorigenesis in a lysozyme-dependent manner. Frontiers in Immunology. 14. 1252979–1252979.
2.
Chauvin, Camille, Daniel Álvarez-Simón, Katarina Radulović, et al.. (2023). NOD2 in monocytes negatively regulates macrophage development through TNFalpha. Frontiers in Immunology. 14. 1181823–1181823. 6 indexed citations
3.
Álvarez-Simón, Daniel, Katarina Radulović, Myriam Delacre, et al.. (2021). 6th European Congress of Immunology, 1–4 September 2021, Virtual meeting. European Journal of Immunology. 51(S1). 1–448. 1 indexed citations
4.
Khaloian, Sevana, Eva Rath, Nassim Hammoudi, et al.. (2020). Mitochondrial impairment drives intestinal stem cell transition into dysfunctional Paneth cells predicting Crohn’s disease recurrence. Gut. 69(11). 1939–1951. 136 indexed citations
5.
Metwaly, Amira, Andreas Dunkel, Nadine Waldschmitt, et al.. (2020). Integrated microbiota and metabolite profiles link Crohn’s disease to sulfur metabolism. Nature Communications. 11(1). 4322–4322. 91 indexed citations
6.
Schülke, K., Anna E. Groebner, Nadine Waldschmitt, et al.. (2020). Initiation of Conceptus Elongation Coincides with an Endometrium Basic Fibroblast Growth Factor (FGF2) Protein Increase in Heifers. International Journal of Molecular Sciences. 21(5). 1584–1584. 7 indexed citations
7.
Ćalasan, Jelena, Nadine Waldschmitt, Amira Metwaly, et al.. (2019). Tu1858 – Segmented Filamentous Bacteria Induce Alternative Th17 Differentiation and Ileo-Colonic Crohn's Disease-Like Inflammation. Gastroenterology. 156(6). S–1149. 2 indexed citations
8.
Coleman, Olivia I., Nadine Waldschmitt, Eva Rath, et al.. (2018). Activated ATF6 Induces Intestinal Dysbiosis and Innate Immune Response to Promote Colorectal Tumorigenesis. Gastroenterology. 155(5). 1539–1552.e12. 87 indexed citations
9.
Waldschmitt, Nadine, Amira Metwaly, Sandra E. Fischer, & Dirk Haller. (2018). Microbial Signatures as a Predictive Tool in IBD—Pearls and Pitfalls. Inflammatory Bowel Diseases. 24(6). 1123–1132. 8 indexed citations
10.
Lamas, Bruno, Marie‐Laure Michel, Nadine Waldschmitt, et al.. (2017). Card9 mediates susceptibility to intestinal pathogens through microbiota modulation and control of bacterial virulence. Gut. 67(10). 1836–1844. 30 indexed citations
11.
Zietek, Tamara, Nadine Waldschmitt, & Eva Rath. (2017). Role of Incretin Hormones in Bowel Diseases. Endocrine development. 32. 49–73. 11 indexed citations
12.
Radulović, Katarina, Sylvain Normand, Ateequr Rehman, et al.. (2017). A dietary flavone confers communicable protection against colitis through NLRP6 signaling independently of inflammasome activation. Mucosal Immunology. 11(3). 811–819. 55 indexed citations
13.
Berger, Emanuel, Eva Rath, Detian Yuan, et al.. (2016). Mitochondrial function controls intestinal epithelial stemness and proliferation. Nature Communications. 7(1). 13171–13171. 149 indexed citations
14.
Pitt, Jonathan M., Marie Vétizou, Nadine Waldschmitt, et al.. (2016). Fine-Tuning Cancer Immunotherapy: Optimizing the Gut Microbiome. Cancer Research. 76(16). 4602–4607. 87 indexed citations
15.
Waldschmitt, Nadine, Emanuel Berger, Eva Rath, et al.. (2014). C/EBP homologous protein inhibits tissue repair in response to gut injury and is inversely regulated with chronic inflammation. Mucosal Immunology. 7(6). 1452–1466. 26 indexed citations
16.
17.
Ulbrich, Susanne E., Thomas Fröhlich, K. Schülke, et al.. (2009). Evidence for Estrogen-Dependent Uterine Serpin (SERPINA14) Expression During Estrus in the Bovine Endometrial Glandular Epithelium and Lumen1. Biology of Reproduction. 81(4). 795–805. 47 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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