Nadine Nagy

1.8k total citations
39 papers, 1.3k citations indexed

About

Nadine Nagy is a scholar working on Cell Biology, Molecular Biology and Surgery. According to data from OpenAlex, Nadine Nagy has authored 39 papers receiving a total of 1.3k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Cell Biology, 20 papers in Molecular Biology and 11 papers in Surgery. Recurrent topics in Nadine Nagy's work include Proteoglycans and glycosaminoglycans research (22 papers), Glycosylation and Glycoproteins Research (12 papers) and Pancreatic function and diabetes (9 papers). Nadine Nagy is often cited by papers focused on Proteoglycans and glycosaminoglycans research (22 papers), Glycosylation and Glycoproteins Research (12 papers) and Pancreatic function and diabetes (9 papers). Nadine Nagy collaborates with scholars based in United States, Germany and Australia. Nadine Nagy's co-authors include Paul L. Bollyky, Thomas N. Wight, Hedwich F. Kuipers, Adam Frymoyer, Heather D. Ishak, Gernot Kaber, Jenna Bollyky, Pamela Y. Johnson, Jens W. Fischer and Marika Bogdani and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Circulation.

In The Last Decade

Nadine Nagy

36 papers receiving 1.3k citations

Peers

Nadine Nagy

SURGE

IMMUN

GENET

Cristina Pontes Vicente Brazil

Thomas C. Wright United States

Minoru Haisa Japan

Toshiaki Yujiri Japan

Richard J. Fish Switzerland

Margaret B. Penno United States

Stefanie Löffek Germany

Liangdan Sun China

M Sporn United States

Jiehong Pan United States

Cristina Pontes Vicente Brazil

Nadine Nagy

193 ×0.4

390 ×0.8

130 ×0.5

SURGE

194 ×1.0

IMMUN

107 ×0.7

GENET

Citations per year, relative to Nadine Nagy Nadine Nagy (= 1×) peers Cristina Pontes Vicente

Countries citing papers authored by Nadine Nagy

Since Specialization

Citations

This map shows the geographic impact of Nadine Nagy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadine Nagy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadine Nagy more than expected).

Fields of papers citing papers by Nadine Nagy

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nadine Nagy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadine Nagy. The network helps show where Nadine Nagy may publish in the future.

Co-authorship network of co-authors of Nadine Nagy

This figure shows the co-authorship network connecting the top 25 collaborators of Nadine Nagy. A scholar is included among the top collaborators of Nadine Nagy based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadine Nagy. Nadine Nagy is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Barlow, Graham L., Christian M. Schürch, Salil S. Bhate, et al.. (2025). The extra-islet pancreas supports autoimmunity in human type 1 diabetes. eLife. 13. 2 indexed citations

Nagy, Nadine, Gernot Kaber, Darko Stefanovski, et al.. (2024). Hymecromone Promotes Longevity and Insulin Sensitivity in Mice. Cells. 13(20). 1727–1727.

Barlow, Graham L., Christian M. Schürch, Salil S. Bhate, et al.. (2024). The extra-islet pancreas supports autoimmunity in human type 1 diabetes. eLife. 13. 1 indexed citations

Nagy, Nadine, Gernot Kaber, Vivekananda Gupta Sunkari, et al.. (2023). Inhibition of hyaluronan synthesis prevents β-cell loss in obesity-associated type 2 diabetes. Matrix Biology. 123. 34–47. 5 indexed citations

Glassman, Caleb R., Leon Su, Hauke Winkelmann, et al.. (2021). Calibration of cell-intrinsic interleukin-2 response thresholds guides design of a regulatory T cell biased agonist. eLife. 10. 24 indexed citations

Bogdani, Marika, Charmaine J. Simeonovic, Nadine Nagy, et al.. (2021). Detection of Glycosaminoglycans in Pancreatic Islets and Lymphoid Tissues. Methods in molecular biology. 695–717.

Harten, Ingrid A., Gernot Kaber, Inkyung Kang, et al.. (2020). The synthesis and secretion of versican isoform V3 by mammalian cells: A role for N-linked glycosylation. Matrix Biology. 89. 27–42. 8 indexed citations

Nagy, Nadine, Gernot Kaber, Michael J. Kratochvil, et al.. (2020). Weekly injection of IL-2 using an injectable hydrogel reduces autoimmune diabetes incidence in NOD mice. Diabetologia. 64(1). 152–158. 12 indexed citations

Gebe, John A., Michel D. Gooden, Gail Workman, et al.. (2020). Modulation of hyaluronan synthases and involvement of T cell-derived hyaluronan in autoimmune responses to transplanted islets. SHILAP Revista de lepidopterología. 9. 100052–100052. 6 indexed citations

10.

Nagy, Nadine, Irina Gurevich, Hedwich F. Kuipers, et al.. (2019). 4-Methylumbelliferyl glucuronide contributes to hyaluronan synthesis inhibition. Journal of Biological Chemistry. 294(19). 7864–7877. 39 indexed citations

11.

Nagy, Nadine, et al.. (2018). Hyaluronan in immune dysregulation and autoimmune diseases. Matrix Biology. 78-79. 292–313. 69 indexed citations

12.

Nagy, Nadine, Vivekananda Gupta Sunkari, Gernot Kaber, et al.. (2018). Hyaluronan levels are increased systemically in human type 2 but not type 1 diabetes independently of glycemic control. Matrix Biology. 80. 46–58. 25 indexed citations

13.

Medina, Carlos, Nadine Nagy, & Paul L. Bollyky. (2018). Extracellular matrix and the maintenance and loss of peripheral immune tolerance in autoimmune insulitis. Current Opinion in Immunology. 55. 22–30. 15 indexed citations

14.

Homann, Susanne, Maria Grandoch, Nadine Nagy, et al.. (2017). Hyaluronan synthase 3 promotes plaque inflammation and atheroprogression. Matrix Biology. 66. 67–80. 30 indexed citations

15.

Suvorava, Tatsiana, Nadine Nagy, Stephanie Pick, et al.. (2015). Impact of eNOS-Dependent Oxidative Stress on Endothelial Function and Neointima Formation. Antioxidants and Redox Signaling. 23(9). 711–723. 30 indexed citations

16.

Nagy, Nadine, Hedwich F. Kuipers, Adam Frymoyer, et al.. (2015). 4-Methylumbelliferone Treatment and Hyaluronan Inhibition as a Therapeutic Strategy in Inflammation, Autoimmunity, and Cancer. Frontiers in Immunology. 6. 123–123. 222 indexed citations

17.

Bogdani, Marika, Charmaine J. Simeonovic, Nadine Nagy, et al.. (2014). The Detection of Glycosaminoglycans in Pancreatic Islets and Lymphoid Tissues. Methods in molecular biology. 1229. 413–430. 8 indexed citations

18.

Niedworok, Christian, Katharina Röck, Till Freudenberger, et al.. (2013). Inhibitory Role of the Small Leucine-Rich Proteoglycan Biglycan in Bladder Cancer. PLoS ONE. 8(11). e80084–e80084. 39 indexed citations

19.

Nagy, Nadine, Maria Grandoch, & Jens W. Fischer. (2011). Abstract 15194: Dabigatran Causes Remodelling of Plaque Extracellular-Matrix in Diabetogenic Low Density Lipoprotein-Receptor Knockout Mice. Circulation. 124. 1 indexed citations

20.

Nagy, Nadine, et al.. (2009). Cyclooxygenase inhibitors repress vascular hyaluronan‐synthesis in murine atherosclerosis and neointimal thickening. Journal of Cellular and Molecular Medicine. 13(9b). 3713–3719. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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