Nadine A. Defranoux
- Co-authors
- Matthias WablHans‐Dieter VolkJacqueline VerdièreMauricette GaisnePiotr P. SłonimskiCynthia L. StokesBernard GuiardWanying Li
- Topics
- Rheumatoid Arthritis Research and Therapies (12 papers)Autoimmune and Inflammatory Disorders Research (4 papers)Cytokine Signaling Pathways and Interactions (3 papers)
- Cited by
- RheumatologyImmunologyHematology
- Journals
- Annual Review of ImmunologyJournal of Molecular BiologyJournal of Bone and Mineral Research
- Partner nations
- United StatesUnited KingdomJapan
In The Last Decade
Nadine A. Defranoux
19 papers receiving 358 citations
Peers
Comparison fields: 5 of 78
- Molecular Biology 184
- Rheumatology 116
- Immunology 92
- Radiology, Nuclear Medicine and Imaging 40
- Oncology 39
Countries citing papers authored by Nadine A. Defranoux
This map shows the geographic impact of Nadine A. Defranoux's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nadine A. Defranoux with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nadine A. Defranoux more than expected).
Fields of papers citing papers by Nadine A. Defranoux
This network shows the impact of papers produced by Nadine A. Defranoux. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nadine A. Defranoux. The network helps show where Nadine A. Defranoux may publish in the future.
Co-authorship network of co-authors of Nadine A. Defranoux
This figure shows the co-authorship network connecting the top 25 collaborators of Nadine A. Defranoux. A scholar is included among the top collaborators of Nadine A. Defranoux based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nadine A. Defranoux. Nadine A. Defranoux is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 26 | |
| 3 | 12 | |
| 4 | 46 | |
| 5 | 3 | |
| 6 | 18 | |
| 7 | 5 | |
| 8 | 2 | |
| 9 | 2 | |
| 10 | 1 | |
| 11 | 1 | |
| 12 | 1 | |
| 13 | 28 | |
| 14 | 41 | |
| 15 | 2 | |
| 16 | 22 | |
| 17 | 22 | |
| 18 | 93 | |
| 19 | 54 |
About Nadine A. Defranoux
Nadine A. Defranoux is a scholar working on Rheumatology, Hematology and Genetics, having authored 19 papers that have together received 385 indexed citations. Recurring topics across this work include Rheumatoid Arthritis Research and Therapies (12 papers), Autoimmune and Inflammatory Disorders Research (4 papers) and Cytokine Signaling Pathways and Interactions (3 papers). The work is most often cited by research in Rheumatology (116 citations), Immunology (92 citations) and Hematology (33 citations). Nadine A. Defranoux has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Matthias Wabl, Hans‐Dieter Volk, Jacqueline Verdière, Mauricette Gaisne, Piotr P. Słonimski, Cynthia L. Stokes, Bernard Guiard, Wanying Li, Andrea van Elsas and Saroja Ramanujan. Their work appears in journals such as Annual Review of Immunology, Journal of Molecular Biology and Journal of Bone and Mineral Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.