N S Paranka

1.3k total citations
10 papers, 1.1k citations indexed

About

N S Paranka is a scholar working on Pharmacology, Genetics and Molecular Biology. According to data from OpenAlex, N S Paranka has authored 10 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Pharmacology, 5 papers in Genetics and 4 papers in Molecular Biology. Recurrent topics in N S Paranka's work include Inflammatory mediators and NSAID effects (7 papers), Estrogen and related hormone effects (5 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). N S Paranka is often cited by papers focused on Inflammatory mediators and NSAID effects (7 papers), Estrogen and related hormone effects (5 papers) and Genomics, phytochemicals, and oxidative stress (3 papers). N S Paranka collaborates with scholars based in United States, United Kingdom and India. N S Paranka's co-authors include Rifat Pamukcu, Gary A. Piazza, Klaus Brendel, Paul H. Gross, David S. Alberts, Randall W. Burt, Gerhard Sperl, Dennis J. Ahnen, Mary F. Krutzsch and Lee J. Hixson and has published in prestigious journals such as Gastroenterology, Journal of Cellular Biochemistry and Journal of Pharmaceutical and Biomedical Analysis.

In The Last Decade

N S Paranka

10 papers receiving 1.1k citations

Peers

N S Paranka

PHARM

GENET

MB

ONCOL

CR

Gerhard Sperl United States

Shigehiko Mukai Japan

Carolyn E. Cole United States

Hsin-Hsiung Tai United States

Galal A. Alshafie United States

Paola Gerletti United States

Russell D. Klein United States

Jennie L. Williams United States

Sharada Katkuri United States

Angela M. Acquaviva Italy

Gerhard Sperl United States

N S Paranka

770 ×1.0

PHARM

432 ×1.0

GENET

567 ×1.7

MB

268 ×1.1

ONCOL

231 ×1.1

CR

Citations per year, relative to N S Paranka N S Paranka (= 1×) peers Gerhard Sperl

Countries citing papers authored by N S Paranka

Since Specialization

Citations

This map shows the geographic impact of N S Paranka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by N S Paranka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites N S Paranka more than expected).

Fields of papers citing papers by N S Paranka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by N S Paranka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by N S Paranka. The network helps show where N S Paranka may publish in the future.

Co-authorship network of co-authors of N S Paranka

This figure shows the co-authorship network connecting the top 25 collaborators of N S Paranka. A scholar is included among the top collaborators of N S Paranka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with N S Paranka. N S Paranka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Piazza, Gary A., David S. Alberts, Lee J. Hixson, et al.. (1997). Sulindac sulfone inhibits azoxymethane-induced colon carcinogenesis in rats without reducing prostaglandin levels.. PubMed. 57(14). 2909–15. 315 indexed citations

2.

Thompson, Henry J., Cheng Jiang, Junxuan Lü, et al.. (1997). Sulfone metabolite of sulindac inhibits mammary carcinogenesis.. PubMed. 57(2). 267–71. 180 indexed citations

3.

Ahnen, Dennis J., et al.. (1995). The sulfone metabolite of sulindac (FGN-1) inhibits rat colonic carcinogenesis but does not inhibit either prostaglandin synthesis or colonic proliferation. Gastroenterology. 108(4). A444–A444. 3 indexed citations

4.

Alberts, David S., Lee J. Hixson, Dennis J. Ahnen, et al.. (1995). Do NSAIDs exert their colon cancer chemoprevention activities through the inhibition of mucosal prostaglandin synthetase?. Journal of Cellular Biochemistry. 59(S22). 18–23. 84 indexed citations

5.

Paranka, N S, et al.. (1995). Determination of FGN-1 (an active metabolite of sulindac) in human plasma, urine, and feces by HPLC. Journal of Pharmaceutical and Biomedical Analysis. 14(1-2). 213–220. 11 indexed citations

6.

Piazza, Gary A., Mary F. Krutzsch, Gerhard Sperl, et al.. (1995). Antineoplastic drugs sulindac sulfide and sulfone inhibit cell growth by inducing apoptosis.. PubMed. 55(14). 3110–6. 378 indexed citations

7.

Thompson, Henry J., Stephanie Briggs, N S Paranka, et al.. (1995). Inhibition of mammary carcinogenesis in rats by sulfone metabolite of sulindac.. PubMed. 87(16). 1259–60. 63 indexed citations

8.

Paranka, N S & R T Dorr. (1995). Effect of doxorubicin on glutathione and glutathione-dependent enzymes in cultured rat heart cells.. PubMed. 14(5A). 2047–52. 18 indexed citations

9.

Ahnen, D.J., et al.. (1995). Sulindac sulfide (SS) and sulfone (FGN-1) both inhibit the growth of colon cancer cell lines by inducing apoptosis. Gastroenterology. 108(4). A443–A443. 2 indexed citations

10.

Hixson, Lee J., David S. Alberts, Mary F. Krutzsch, et al.. (1994). Antiproliferative effect of nonsteroidal antiinflammatory drugs against human colon cancer cells.. PubMed. 3(5). 433–8. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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