N. Mihailovich
About
In The Last Decade
N. Mihailovich
32 papers receiving 797 citations
Peers
Comparison fields: 5 of 91
- Molecular Biology 378
- Cancer Research 324
- Oncology 195
- Health, Toxicology and Mutagenesis 118
- Hepatology 114
Countries citing papers authored by N. Mihailovich
This map shows the geographic impact of N. Mihailovich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by N. Mihailovich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites N. Mihailovich more than expected).
Fields of papers citing papers by N. Mihailovich
This network shows the impact of papers produced by N. Mihailovich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by N. Mihailovich. The network helps show where N. Mihailovich may publish in the future.
Co-authorship network of co-authors of N. Mihailovich
This figure shows the co-authorship network connecting the top 25 collaborators of N. Mihailovich. A scholar is included among the top collaborators of N. Mihailovich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with N. Mihailovich. N. Mihailovich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Correspondence re: S. D. Vesselinovitch and N. Mihailovich. Kinetics of Diethylnitrosamine Hepatocarcinogenesis in the Infant Mouse. Cancer Res., 43: 4253–4259, 1983—Reply | 0 |
| 2 | Kinetics of diethylnitrosamine hepatocarcinogenesis in the infant mouse. | 146 |
| 3 | Relevance of basophilic foci to promoting effect of sex hormones on hepatocarcinogenesis. | 13 |
| 4 | Modifying role of partial hepatectomy and gonadectomy in ethylnitrosourea-induced hepatocarcinogenesis. | 68 |
| 5 | Transplacental and lactational carcinogenesis by safrole. | 21 |
| 6 | Neoplastic response of mouse tissues during perinatal age periods and its significance in chemical carcinogenesis. | 70 |
| 7 | Morphology and metastatic nature of induced hepatic nodular lesions in C57BL x C3H F1 mice. | 74 |
| 8 | Prenatal multicarcinogenesis by ethylnitrosourea in mice. | 27 |
| 9 | Factors modulating benzidine carcinogenicity bioassay. | 23 |
| 10 | Role of hormonal environment, partial hepatectomy, and dose of ethylnitrosourea in renal carcinogenesis. | 5 |
| 11 | Aflatoxin B 1 , a hepatocarcinogen in the infant mouse. | 82 |
| 12 | Broad spectrum carcinogenicity of ethyl nitroso urea in the new born and infant mice | 2 |
| 13 | Perinatal carcinogenesis by urethan. | 18 |
| 14 | The induction of malignant melanomas in Syrian white hamster by neonatal exposure to urethan. | 10 |
| 15 | Comparative studies on the kinetics of the neoplastic competence in mice. | 5 |
| 16 | The induction of benign and malignant liver tumors by urethan in newborn rats. | 9 |
| 17 | The development of neurogenic neoplasms, embryonal kidney tumors, harderian gland adenomas, anitschkow cell sarcomas of the heart, and other neoplasms in urethan-treated newborn rats. | 16 |
| 18 | The inhibitory effect of griseofulvin on the "promotion" of skin carcinogenesis. | 6 |
| 19 | The neonatal and infant age periods as biologic factors which modify multicarcinogenesis by urethan. | 22 |
| 20 | The prenatal exposure of mice to urethan and the consequent development of tumors in various tissues. | 16 |
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.