Moritz F. Eissmann

2.9k total citations · 1 hit paper
27 papers, 2.2k citations indexed

About

Moritz F. Eissmann is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Moritz F. Eissmann has authored 27 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 10 papers in Oncology and 7 papers in Surgery. Recurrent topics in Moritz F. Eissmann's work include IL-33, ST2, and ILC Pathways (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). Moritz F. Eissmann is often cited by papers focused on IL-33, ST2, and ILC Pathways (5 papers), Cancer-related molecular mechanisms research (5 papers) and RNA Research and Splicing (5 papers). Moritz F. Eissmann collaborates with scholars based in Australia, Germany and United States. Moritz F. Eissmann's co-authors include Martin Zörnig, Tony Gutschner, Sven Diederichs, Matthias Groß, Monika Hämmerle, David L. Spector, Gayatri Arun, Jeff Hsu, Gene Hung and Alexey S. Revenko and has published in prestigious journals such as Nature Communications, Blood and Gastroenterology.

In The Last Decade

Moritz F. Eissmann

24 papers receiving 2.2k citations

Hit Papers

The Noncoding RNA MALAT1 Is a Critical Regulator of the M... 2012 2026 2016 2021 2012 400 800 1.2k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Moritz F. Eissmann Australia 15 1.7k 1.5k 306 246 189 27 2.2k
Chengwu Zeng China 22 1.6k 1.0× 1.2k 0.8× 280 0.9× 323 1.3× 57 0.3× 60 2.1k
Hesham A. El‐Mahdy Egypt 31 1.3k 0.8× 1.3k 0.8× 86 0.3× 268 1.1× 251 1.3× 42 1.9k
Ahmed Ismail Egypt 28 1.2k 0.7× 1.2k 0.8× 77 0.3× 228 0.9× 219 1.2× 38 1.7k
Suhail Ahmed Kabeer Rasheed Singapore 13 1.7k 1.0× 1.4k 0.9× 133 0.4× 243 1.0× 73 0.4× 16 2.1k
Chaoyun Pan China 19 1.0k 0.6× 668 0.4× 242 0.8× 272 1.1× 72 0.4× 29 1.5k
Jöerg H. Leupold Germany 9 1.8k 1.1× 1.6k 1.0× 129 0.4× 279 1.1× 84 0.4× 9 2.2k
Fangting Wu United States 13 2.5k 1.5× 2.0k 1.3× 113 0.4× 276 1.1× 69 0.4× 19 2.9k
Sadegh Babashah Iran 23 1.4k 0.8× 1.1k 0.7× 135 0.4× 208 0.8× 55 0.3× 70 1.8k
Elsayed G.E. Elsakka Egypt 26 901 0.5× 895 0.6× 63 0.2× 182 0.7× 184 1.0× 38 1.3k
Qiong Shao China 17 1.3k 0.8× 1.1k 0.7× 125 0.4× 446 1.8× 110 0.6× 41 1.8k

Countries citing papers authored by Moritz F. Eissmann

Since Specialization
Citations

This map shows the geographic impact of Moritz F. Eissmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Moritz F. Eissmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Moritz F. Eissmann more than expected).

Fields of papers citing papers by Moritz F. Eissmann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Moritz F. Eissmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Moritz F. Eissmann. The network helps show where Moritz F. Eissmann may publish in the future.

Co-authorship network of co-authors of Moritz F. Eissmann

This figure shows the co-authorship network connecting the top 25 collaborators of Moritz F. Eissmann. A scholar is included among the top collaborators of Moritz F. Eissmann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Moritz F. Eissmann. Moritz F. Eissmann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
O’Brien, Megan A., Christine D. Dijkstra, Amr H. Allam, et al.. (2025). Protocol on utilizing murine gastric cancer organoids for modeling subcutaneous, orthotopic primary, and liver metastatic disease in mice. STAR Protocols. 6(2). 103784–103784.
2.
Afshar‐Sterle, Shoukat, Janson W.T. Tse, Stefanie Fischer, et al.. (2024). DCLK1 induces a pro-tumorigenic phenotype to drive gastric cancer progression. Science Signaling. 17(854). eabq4888–eabq4888. 3 indexed citations
5.
Dijkstra, Christine D., et al.. (2023). Generation of gene-of-interest knockouts in murine organoids using CRISPR-Cas9. STAR Protocols. 4(1). 102076–102076. 4 indexed citations
6.
Chisanga, David, Wei Shi, Shoukat Afshar‐Sterle, et al.. (2023). A tuft cell - ILC2 signaling circuit provides therapeutic targets to inhibit gastric metaplasia and tumor development. Nature Communications. 14(1). 6872–6872. 15 indexed citations
7.
To, Sarah Q., David Chisanga, Moritz F. Eissmann, et al.. (2023). Mechanisms of cellular crosstalk in the gastric tumor microenvironment are mediated by YAP1 and STAT3. Life Science Alliance. 7(2). e202302411–e202302411. 2 indexed citations
9.
Luk, Ian Y., Laura J. Jenkins, Frank Köentgen, et al.. (2021). EHF is essential for epidermal and colonic epithelial homeostasis, and suppresses Apc -initiated colonic tumorigenesis. Development. 148(12). 11 indexed citations
10.
Huynh, Jennifer, David Chisanga, Wei Shi, et al.. (2021). Host IL11 Signaling Suppresses CD4+ T cell–Mediated Antitumor Responses to Colon Cancer in Mice. Cancer Immunology Research. 9(7). 735–747. 29 indexed citations
11.
Poh, Ashleigh R., Amy R. Dwyer, Moritz F. Eissmann, et al.. (2020). Inhibition of the SRC Kinase HCK Impairs STAT3-Dependent Gastric Tumor Growth in Mice. Cancer Immunology Research. 8(4). 428–435. 24 indexed citations
12.
Eissmann, Moritz F., Michael Büchert, & Matthias Ernst. (2020). IL33 and Mast Cells—The Key Regulators of Immune Responses in Gastrointestinal Cancers?. Frontiers in Immunology. 11. 1389–1389. 25 indexed citations
13.
Eissmann, Moritz F., Christine D. Dijkstra, Andrew Jarnicki, et al.. (2019). IL-33-mediated mast cell activation promotes gastric cancer through macrophage mobilization. Nature Communications. 10(1). 2735–2735. 161 indexed citations
14.
Eissmann, Moritz F., Christine D. Dijkstra, Merridee A. Wouters, et al.. (2018). Interleukin 33 Signaling Restrains Sporadic Colon Cancer in an Interferon-γ–Dependent Manner. Cancer Immunology Research. 6(4). 409–421. 34 indexed citations
15.
Thiem, Stefan, Moritz F. Eissmann, Tracy L. Putoczki, et al.. (2016). Stomach-Specific Activation of Oncogenic KRAS and STAT3-Dependent Inflammation Cooperatively Promote Gastric Tumorigenesis in a Preclinical Model. Cancer Research. 76(8). 2277–2287. 34 indexed citations
16.
Ernst, Matthias, Stefan Thiem, Paul M. Nguyen, Moritz F. Eissmann, & Tracy L. Putoczki. (2014). Epithelial gp130/Stat3 functions: An intestinal signaling node in health and disease. Seminars in Immunology. 26(1). 29–37. 53 indexed citations
17.
Eissmann, Moritz F., Bettina Schwamb, Julia Moser, et al.. (2013). A Functional Yeast Survival Screen of Tumor-Derived cDNA Libraries Designed to Identify Anti-Apoptotic Mammalian Oncogenes. PLoS ONE. 8(5). e64873–e64873. 17 indexed citations
18.
Gutschner, Tony, Monika Hämmerle, Moritz F. Eissmann, et al.. (2012). The Noncoding RNA MALAT1 Is a Critical Regulator of the Metastasis Phenotype of Lung Cancer Cells. Cancer Research. 73(3). 1180–1189. 1307 indexed citations breakdown →
19.
Eissmann, Moritz F., Tony Gutschner, Monika Hämmerle, et al.. (2012). Loss of the abundant nuclear non-coding RNAMALAT1is compatible with life and development. RNA Biology. 9(8). 1076–1087. 329 indexed citations
20.
Eissmann, Moritz F., Geert Michel, Martin Hrabě de Angelis, et al.. (2012). Overexpression of the anti-apoptotic protein AVEN contributes to increased malignancy in hematopoietic neoplasms. Oncogene. 32(20). 2586–2591. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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