Monina S. Cabrera

607 total citations
9 papers, 428 citations indexed

About

Monina S. Cabrera is a scholar working on Genetics, Molecular Biology and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Monina S. Cabrera has authored 9 papers receiving a total of 428 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Genetics, 4 papers in Molecular Biology and 3 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Monina S. Cabrera's work include Diabetes and associated disorders (5 papers), Sexual Differentiation and Disorders (3 papers) and T-cell and B-cell Immunology (2 papers). Monina S. Cabrera is often cited by papers focused on Diabetes and associated disorders (5 papers), Sexual Differentiation and Disorders (3 papers) and T-cell and B-cell Immunology (2 papers). Monina S. Cabrera collaborates with scholars based in United States and United Kingdom. Monina S. Cabrera's co-authors include Maria I. New, Maria G. Vogiatzi, Andrea S. Putnam, Ann D. Carlson, Ji‐Qing Wei, Jihad S. Obeid, Karen Lin‐Su, I. Howard Marshall, Robert C. Wilson and Nora Sarvetnick and has published in prestigious journals such as PLoS ONE, The Journal of Clinical Endocrinology & Metabolism and Frontiers in Immunology.

In The Last Decade

Monina S. Cabrera

9 papers receiving 409 citations

Peers

Monina S. Cabrera

MB

EDM

GENET

CB

UROLO

Hílton Kuperman Brazil

G. F. Rondanini Italy

Rieko Tadokoro‐Cuccaro United Kingdom

Bert Elvers Netherlands

Sarar Mohamed Saudi Arabia

C. Lukas-Croisier France

María Caimari Spain

Donna O’Neil United Kingdom

Ivana Joksić Serbia

Noritoshi Enatsu Japan

Hílton Kuperman Brazil

Monina S. Cabrera

150 ×0.5

MB

259 ×1.2

EDM

158 ×1.3

GENET

30 ×0.3

CB

20 ×0.3

UROLO

Citations per year, relative to Monina S. Cabrera Monina S. Cabrera (= 1×) peers Hílton Kuperman

Countries citing papers authored by Monina S. Cabrera

Since Specialization

Citations

This map shows the geographic impact of Monina S. Cabrera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Monina S. Cabrera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Monina S. Cabrera more than expected).

Fields of papers citing papers by Monina S. Cabrera

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Monina S. Cabrera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Monina S. Cabrera. The network helps show where Monina S. Cabrera may publish in the future.

Co-authorship network of co-authors of Monina S. Cabrera

This figure shows the co-authorship network connecting the top 25 collaborators of Monina S. Cabrera. A scholar is included among the top collaborators of Monina S. Cabrera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Monina S. Cabrera. Monina S. Cabrera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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9 of 9 papers shown

1.

Harms, Robert Z., et al.. (2021). Frequencies of CD8 and DN MAIT Cells Among Children Diagnosed With Type 1 Diabetes Are Similar to Age-Matched Controls. Frontiers in Immunology. 12. 604157–604157. 2 indexed citations

2.

Harms, Robert Z., et al.. (2020). Confirmation and Identification of Biomarkers Implicating Environmental Triggers in the Pathogenesis of Type 1 Diabetes. Frontiers in Immunology. 11. 1922–1922. 11 indexed citations

3.

Harms, Robert Z., et al.. (2015). Altered CD161bright CD8+ Mucosal Associated Invariant T (MAIT)-Like Cell Dynamics and Increased Differentiation States among Juvenile Type 1 Diabetics. PLoS ONE. 10(1). e0117335–e0117335. 30 indexed citations

4.

Harms, Robert Z., et al.. (2015). Increased expression of IL-18 in the serum and islets of type 1 diabetics. Molecular Immunology. 64(2). 306–312. 44 indexed citations

5.

Yarde, Danielle N., et al.. (2014). CD28− CD8+ T cells are significantly reduced and correlate with disease duration in juveniles with type 1 diabetes. Human Immunology. 75(10). 1069–1074. 10 indexed citations

6.

New, Maria I., Ann D. Carlson, Jihad S. Obeid, et al.. (2003). Update: Prenatal Diagnosis for Congenital Adrenal Hyperplasia in 595 Pregnancies. The Endocrinologist. 13(3). 233–239. 18 indexed citations

7.

Cabrera, Monina S., Maria G. Vogiatzi, & Maria I. New. (2001). Long Term Outcome in Adult Males with Classic Congenital Adrenal Hyperplasia¹. The Journal of Clinical Endocrinology & Metabolism. 86(7). 3070–3078. 165 indexed citations

8.

New, Maria I., Ann D. Carlson, Jihad S. Obeid, et al.. (2001). EXTENSIVE PERSONAL EXPERIENCE: Prenatal Diagnosis for Congenital Adrenal Hyperplasia in 532 Pregnancies. The Journal of Clinical Endocrinology & Metabolism. 86(12). 5651–5657. 147 indexed citations

9.

Obeid, Jihad S., Robert C. Wilson, Monina S. Cabrera, et al.. (1999). Profile of the Pediatric Endocrine Clinic at New York–Presbyterian Hospital, New York Weill Cornell Center¹. The Journal of Clinical Endocrinology & Metabolism. 84(12). 4444–4449. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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