Mohammed Qatanani

4.1k total citations · 3 hit papers
22 papers, 3.3k citations indexed

About

Mohammed Qatanani is a scholar working on Epidemiology, Physiology and Molecular Biology. According to data from OpenAlex, Mohammed Qatanani has authored 22 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Epidemiology, 7 papers in Physiology and 6 papers in Molecular Biology. Recurrent topics in Mohammed Qatanani's work include Adipokines, Inflammation, and Metabolic Diseases (8 papers), Adipose Tissue and Metabolism (6 papers) and Pharmacogenetics and Drug Metabolism (5 papers). Mohammed Qatanani is often cited by papers focused on Adipokines, Inflammation, and Metabolic Diseases (8 papers), Adipose Tissue and Metabolism (6 papers) and Pharmacogenetics and Drug Metabolism (5 papers). Mohammed Qatanani collaborates with scholars based in United States, Australia and Lebanon. Mohammed Qatanani's co-authors include Mitchell A. Lazar, David D. Moore, Wendong Huang, Bingning Dong, Joshua C. Curtin, Jun Zhang, Ke Ma, Xiongfei Huang, Jun Liu and Robert A. Reid and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Mohammed Qatanani

22 papers receiving 3.2k citations

Hit Papers

Mechanisms of obesity-associated insulin resistance: many... 2006 2026 2012 2019 2007 2007 2006 200 400 600

Peers

Mohammed Qatanani
Ke Ma United States
Xunshan Ding United States
Jacqueline G. Alvarez United States
Giovanni Solinas Switzerland
John M. Stafford United States
Lone Hansen United States
Aldo Grefhorst Netherlands
Ann Marie Zavacki United States
Ke Ma United States
Mohammed Qatanani
Citations per year, relative to Mohammed Qatanani Mohammed Qatanani (= 1×) peers Ke Ma

Countries citing papers authored by Mohammed Qatanani

Since Specialization
Citations

This map shows the geographic impact of Mohammed Qatanani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mohammed Qatanani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mohammed Qatanani more than expected).

Fields of papers citing papers by Mohammed Qatanani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mohammed Qatanani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mohammed Qatanani. The network helps show where Mohammed Qatanani may publish in the future.

Co-authorship network of co-authors of Mohammed Qatanani

This figure shows the co-authorship network connecting the top 25 collaborators of Mohammed Qatanani. A scholar is included among the top collaborators of Mohammed Qatanani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mohammed Qatanani. Mohammed Qatanani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Schwartz, Daniel R., Erika R. Briggs, Mohammed Qatanani, et al.. (2013). Human Resistin in Chemotherapy-Induced Heart Failure in Humanized Male Mice and in Women Treated for Breast Cancer. Endocrinology. 154(11). 4206–4214. 10 indexed citations
2.
Qatanani, Mohammed, Yejun Tan, Radu Dobrin, et al.. (2012). Inverse Regulation of Inflammation and Mitochondrial Function in Adipose Tissue Defines Extreme Insulin Sensitivity in Morbidly Obese Patients. Diabetes. 62(3). 855–863. 47 indexed citations
3.
Baker, Joshua F., Mohammed Qatanani, Andrew J. Cucchiara, et al.. (2011). Resistin Levels in Lupus and Associations with Disease-specific Measures, Insulin Resistance, and Coronary Calcification. The Journal of Rheumatology. 38(11). 2369–2375. 45 indexed citations
4.
Park, Hyeong Kyu, Mohammed Qatanani, Erika R. Briggs, Rexford S. Ahima, & Mitchell A. Lazar. (2011). Inflammatory Induction of Human Resistin Causes Insulin Resistance in Endotoxemic Mice. Diabetes. 60(3). 775–783. 42 indexed citations
5.
Lefterova, Martina I., David J. Steger, Mohammed Qatanani, et al.. (2010). Cell-Specific Determinants of Peroxisome Proliferator-Activated Receptor γ Function in Adipocytes and Macrophages. Molecular and Cellular Biology. 30(9). 2078–2089. 174 indexed citations
6.
Dong, Bingning, Mohammed Qatanani, & David D. Moore. (2009). Constitutive androstane receptor mediates the induction of drug metabolism in mouse models of type 1 diabetes†. Hepatology. 50(2). 622–629. 35 indexed citations
7.
Schupp, Michael, Ana G. Cristancho, Martina I. Lefterova, et al.. (2009). Re-expression of GATA2 Cooperates with Peroxisome Proliferator-activated Receptor-γ Depletion to Revert the Adipocyte Phenotype. Journal of Biological Chemistry. 284(14). 9458–9464. 68 indexed citations
8.
Qatanani, Mohammed, et al.. (2009). Macrophage-derived human resistin exacerbates adipose tissue inflammation and insulin resistance in mice. Journal of Clinical Investigation. 119(3). 531–539. 183 indexed citations
9.
Schupp, Michael, Martina I. Lefterova, Jürgen Janke, et al.. (2009). Retinol saturase promotes adipogenesis and is downregulated in obesity. Proceedings of the National Academy of Sciences. 106(4). 1105–1110. 77 indexed citations
10.
Park, Young Joo, Mohammed Qatanani, Steven S. Chua, et al.. (2008). Loss of orphan receptor small heterodimer partner sensitizes mice to liver injury from obstructive cholestasis. Hepatology. 47(5). 1578–1586. 65 indexed citations
11.
Yin, Lei, Nan Wu, Joshua C. Curtin, et al.. (2007). Rev-erbα, a Heme Sensor That Coordinates Metabolic and Circadian Pathways. Science. 318(5857). 1786–1789. 572 indexed citations breakdown →
12.
Qatanani, Mohammed & Mitchell A. Lazar. (2007). Mechanisms of obesity-associated insulin resistance: many choices on the menu. Genes & Development. 21(12). 1443–1455. 602 indexed citations breakdown →
13.
Qatanani, Mohammed & David D. Moore. (2005). CAR, The Continuously Advancing Receptor, in Drug Metabolism and Disease. Current Drug Metabolism. 6(4). 329–339. 129 indexed citations
14.
Qatanani, Mohammed, Jun Zhang, & David D. Moore. (2004). Role of the Constitutive Androstane Receptor in Xenobiotic-Induced Thyroid Hormone Metabolism. Endocrinology. 146(3). 995–1002. 136 indexed citations
15.
Qatanani, Mohammed, Ping Wei, & David D. Moore. (2004). Alterations in the distribution and orexigenic effects of dexamethasone in CAR-null mice. Pharmacology Biochemistry and Behavior. 78(2). 285–291. 6 indexed citations
16.
Zhang, Jun, Wendong Huang, Mohammed Qatanani, Ronald M. Evans, & David D. Moore. (2004). The Constitutive Androstane Receptor and Pregnane X Receptor Function Coordinately to Prevent Bile Acid-induced Hepatotoxicity. Journal of Biological Chemistry. 279(47). 49517–49522. 184 indexed citations
17.
Huang, Wendong, Jun Zhang, Steven S. Chua, et al.. (2003). Induction of bilirubin clearance by the constitutive androstane receptor (CAR). Proceedings of the National Academy of Sciences. 100(7). 4156–4161. 309 indexed citations
18.
Qatanani, Mohammed, Alì Taher, Suzanne Koussa, et al.. (2000). β‐Thalassaemia intermedia in Lebanon. European Journal Of Haematology. 64(4). 237–244. 27 indexed citations
19.
Zahed, L., Mohammed Qatanani, Mona Nabulsi, & Alì Taher. (2000). β-Thalassemia Mutations and Haplotype Analysis in Lebanon. Hemoglobin. 24(4). 269–276. 22 indexed citations
20.
Youssoufian, Hagop, et al.. (1999). Analysis of Epitope-Tagged Forms of the Dyskeratosis Congenita Protein (Dyskerin): Identification of a Nuclear Localization Signal. Blood Cells Molecules and Diseases. 25(5). 305–309. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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