Mohammed Kouadir

1.8k total citations · 1 hit paper
24 papers, 1.4k citations indexed

About

Mohammed Kouadir is a scholar working on Molecular Biology, Neurology and Immunology. According to data from OpenAlex, Mohammed Kouadir has authored 24 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 10 papers in Neurology and 5 papers in Immunology. Recurrent topics in Mohammed Kouadir's work include Prion Diseases and Protein Misfolding (14 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Inflammasome and immune disorders (5 papers). Mohammed Kouadir is often cited by papers focused on Prion Diseases and Protein Misfolding (14 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Inflammasome and immune disorders (5 papers). Mohammed Kouadir collaborates with scholars based in China and United States. Mohammed Kouadir's co-authors include Fushan Shi, Yang Yang, Houhui Song, Huanan Wang, Deming Zhao, Xiangmei Zhou, Xiaomin Yin, Lifeng Yang, Jihong Wang and Yongyao Fu and has published in prestigious journals such as PLoS ONE, Journal of Neurochemistry and The Journal of Infectious Diseases.

In The Last Decade

Mohammed Kouadir

24 papers receiving 1.4k citations

Hit Papers

Recent advances in the mechanisms of NLRP3 inflammasome a... 2019 2026 2021 2023 2019 250 500 750

Peers

Mohammed Kouadir
Eric I. Elliott United States
Vanessa Byles United States
Ellen A. Bernstein United States
Eric I. Elliott United States
Mohammed Kouadir
Citations per year, relative to Mohammed Kouadir Mohammed Kouadir (= 1×) peers Eric I. Elliott

Countries citing papers authored by Mohammed Kouadir

Since Specialization
Citations

This map shows the geographic impact of Mohammed Kouadir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mohammed Kouadir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mohammed Kouadir more than expected).

Fields of papers citing papers by Mohammed Kouadir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mohammed Kouadir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mohammed Kouadir. The network helps show where Mohammed Kouadir may publish in the future.

Co-authorship network of co-authors of Mohammed Kouadir

This figure shows the co-authorship network connecting the top 25 collaborators of Mohammed Kouadir. A scholar is included among the top collaborators of Mohammed Kouadir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mohammed Kouadir. Mohammed Kouadir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Yang, Yang, Huanan Wang, Mohammed Kouadir, Houhui Song, & Fushan Shi. (2019). Recent advances in the mechanisms of NLRP3 inflammasome activation and its inhibitors. Cell Death and Disease. 10(2). 128–128. 996 indexed citations breakdown →
2.
Shi, Fushan, Yang Yang, Tiancheng Wang, et al.. (2016). Cellular Prion Protein Promotes Neuronal Differentiation of Adipose-Derived Stem Cells by Upregulating miRNA-124. Journal of Molecular Neuroscience. 59(1). 48–55. 21 indexed citations
3.
Shi, Fushan, Yang Yang, Mohammed Kouadir, et al.. (2016). Inflammasome-independent role of NLRP12 in suppressing colonic inflammation regulated by Blimp-1. Oncotarget. 7(21). 30575–30584. 17 indexed citations
4.
Wang, Jihong, Deming Zhao, Bo Pan, et al.. (2014). Toll-Like Receptor 2 Deficiency Shifts PrP106-126-Induced Microglial Activation from a Neurotoxic to a Neuroprotective Phenotype. Journal of Molecular Neuroscience. 55(4). 880–890. 14 indexed citations
5.
Yang, Lifeng, Mohammed Kouadir, Jin Wang, et al.. (2013). Expression and Distribution of Laminin Receptor Precursor/Laminin Receptor in Rabbit Tissues. Journal of Molecular Neuroscience. 51(2). 591–601. 3 indexed citations
6.
Yang, Yang, Xiangmei Zhou, Mohammed Kouadir, et al.. (2013). The AIM2 Inflammasome Is Involved in Macrophage Activation During Infection With Virulent Mycobacterium bovis Strain. The Journal of Infectious Diseases. 208(11). 1849–1858. 49 indexed citations
7.
Zhang, Siming, Lifeng Yang, Mohammed Kouadir, et al.. (2013). PP2 and piceatannol inhibit PrP<sub>106–126</sub>-induced iNOS activation mediated by CD36 in BV2 microglia. Acta Biochimica et Biophysica Sinica. 45(9). 763–772. 6 indexed citations
8.
Tu, Jian Cheng, Lifeng Yang, Xiangmei Zhou, et al.. (2013). PrP106-126 and Aβ1-42 Peptides Induce BV-2 Microglia Chemotaxis and Proliferation. Journal of Molecular Neuroscience. 52(1). 107–116. 5 indexed citations
9.
Shi, Fushan, Lifeng Yang, Jihong Wang, et al.. (2013). Inhibition of phagocytosis reduced the classical activation of BV2 microglia induced by amyloidogenic fragments of beta-amyloid and prion proteins. Acta Biochimica et Biophysica Sinica. 45(11). 973–978. 9 indexed citations
10.
Zhou, Xiangmei, Mohammed Kouadir, Fushan Shi, et al.. (2013). Cellular Prion Protein Participates in the Regulation of Inflammatory Response and Apoptosis in BV2 Microglia During Infection with Mycobacterium bovis. Journal of Molecular Neuroscience. 51(1). 118–126. 23 indexed citations
11.
Shi, Fushan, Yang Yang, Mohammed Kouadir, et al.. (2013). Inhibition of phagocytosis and lysosomal acidification suppresses neurotoxic prion peptide-induced NALP3 inflammasome activation in BV2 microglia. Journal of Neuroimmunology. 260(1-2). 121–125. 33 indexed citations
12.
Yang, Lifeng, Mohammed Kouadir, Siming Zhang, et al.. (2012). Antibody-Mediated Inhibition of Integrin α5β1 Blocks Neurotoxic Prion Peptide PrP106–126-Induced Activation of BV2 Microglia. Journal of Molecular Neuroscience. 48(1). 248–252. 12 indexed citations
13.
Shi, Fushan, Lifeng Yang, Mohammed Kouadir, et al.. (2012). The NALP3 inflammasome is involved in neurotoxic prion peptide-induced microglial activation. Journal of Neuroinflammation. 9(1). 73–73. 94 indexed citations
14.
Kouadir, Mohammed, Lifeng Yang, Fushan Shi, et al.. (2012). CD36 Participates in PrP106–126-Induced Activation of Microglia. PLoS ONE. 7(1). e30756–e30756. 27 indexed citations
15.
Kouadir, Mohammed, Lifeng Yang, Jian Cheng Tu, et al.. (2011). Comparison of mRNA Expression Patterns of Class B Scavenger Receptors in BV2 Microglia upon Exposure to Amyloidogenic Fragments of Beta-Amyloid and Prion Proteins. DNA and Cell Biology. 30(11). 893–897. 12 indexed citations
16.
Lu, Yun, Ailing Liu, Xiangmei Zhou, et al.. (2011). Prion Peptide PrP106-126 Induces Inducible Nitric Oxide Synthase and Proinflammatory Cytokine Gene Expression Through the Activation of NF-κB in Macrophage Cells. DNA and Cell Biology. 31(5). 833–838. 10 indexed citations
17.
Zhang, Jiaoer, Ying‐Che Huang, Mohammed Kouadir, et al.. (2010). IFN-γpromotes THP-1 cell apoptosis during early infection withMycobacterium bovisby activating different apoptotic signaling. FEMS Immunology & Medical Microbiology. 60(3). 191–198. 9 indexed citations
18.
Zhou, Haiyun, Xiangmei Zhou, Mohammed Kouadir, et al.. (2009). Induction of macrophage migration by neurotoxic prion protein fragment. Journal of Neuroscience Methods. 181(1). 1–5. 9 indexed citations
19.
Pang, Wanyong, et al.. (2009). Polymorphisms of the prion protein gene (PRNP) in the Tibetan Mastiff. Animal Genetics. 40(6). 1001–1002. 2 indexed citations
20.
Su, Xiaoou, Yiqin Wang, Jianmin Yang, et al.. (2008). Cloning and characterization of full-length coding sequence (CDS) of the ovine 37/67-kDa laminin receptor (RPSA). Molecular Biology Reports. 36(8). 2131–2137. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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