Mitsuhiro Ino

772 total citations
20 papers, 595 citations indexed

About

Mitsuhiro Ino is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Behavioral Neuroscience. According to data from OpenAlex, Mitsuhiro Ino has authored 20 papers receiving a total of 595 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Cellular and Molecular Neuroscience and 6 papers in Behavioral Neuroscience. Recurrent topics in Mitsuhiro Ino's work include Stress Responses and Cortisol (6 papers), Hormonal Regulation and Hypertension (5 papers) and Neuroscience and Neuropharmacology Research (4 papers). Mitsuhiro Ino is often cited by papers focused on Stress Responses and Cortisol (6 papers), Hormonal Regulation and Hypertension (5 papers) and Neuroscience and Neuropharmacology Research (4 papers). Mitsuhiro Ino collaborates with scholars based in Japan, United States and United Kingdom. Mitsuhiro Ino's co-authors include Norimasa Miyamoto, Eiki Takahashi, Takeshi Nagasu, Carsten T. Beuckmann, Masashi Yanagisawa, Christopher M. Sinton, Tohru Oki, Isao Tanaka, Yukio Nishizawa and Keiji Imoto and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Cancer Research.

In The Last Decade

Mitsuhiro Ino

20 papers receiving 581 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mitsuhiro Ino Japan	11	349	267	116	61	54	20	595
Erik T. Dustrude United States	14	332 1.0×	323 1.2×	235 2.0×	61 1.0×	27 0.5×	23	596
Xuelai Fan Canada	6	340 1.0×	128 0.5×	186 1.6×	51 0.8×	27 0.5×	7	726
Miguel Martin‐Caraballo United States	17	369 1.1×	199 0.7×	49 0.4×	24 0.4×	28 0.5×	34	672
Chi S. Ho United States	12	395 1.1×	330 1.2×	60 0.5×	41 0.7×	31 0.6×	16	580
Shigeyuki Nonaka Japan	6	286 0.8×	301 1.1×	72 0.6×	35 0.6×	51 0.9×	6	675
Seema Tevar United States	5	322 0.9×	604 2.3×	106 0.9×	55 0.9×	41 0.8×	7	914
Catrin Wernicke Germany	18	246 0.7×	249 0.9×	51 0.4×	82 1.3×	33 0.6×	27	616
Andrew D. Piekarz United States	10	472 1.4×	371 1.4×	287 2.5×	38 0.6×	14 0.3×	13	711
Pierrick Gandolfo France	16	220 0.6×	316 1.2×	96 0.8×	16 0.3×	15 0.3×	22	634
Enrique Castro Spain	20	372 1.1×	357 1.3×	69 0.6×	16 0.3×	13 0.2×	34	1.0k

Countries citing papers authored by Mitsuhiro Ino

Since Specialization

Citations

This map shows the geographic impact of Mitsuhiro Ino's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mitsuhiro Ino with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mitsuhiro Ino more than expected).

Fields of papers citing papers by Mitsuhiro Ino

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mitsuhiro Ino. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mitsuhiro Ino. The network helps show where Mitsuhiro Ino may publish in the future.

Co-authorship network of co-authors of Mitsuhiro Ino

This figure shows the co-authorship network connecting the top 25 collaborators of Mitsuhiro Ino. A scholar is included among the top collaborators of Mitsuhiro Ino based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mitsuhiro Ino. Mitsuhiro Ino is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Landry, Ishani, Jagadeesh Aluri, Edgar Schuck, et al.. (2022). Phase 1 Single Ascending and Multiple Ascending Dose Studies of Phosphodiesterase-9 Inhibitor E2027. Alzheimer Disease & Associated Disorders. 36(3). 200–207. 6 indexed citations

Landry, Ishani, Robert Lai, Peter D. W. Boyd, et al.. (2018). O1‐12‐06: PHASE 1 MULTIPLE ASCENDING DOSE (MAD) STUDY OF PHOSPHODIESTERASE‐9 INHIBITOR E2027: CONFIRMATION OF TARGET ENGAGEMENT AND SELECTION OF PHASE 2 DOSE IN DEMENTIA WITH LEWY BODIES. Alzheimer s & Dementia. 14(7S_Part_4). 1 indexed citations

Schuck, Edgar, Robert Lai, Jagadeesh Aluri, et al.. (2017). [P1–056]: POPULATION PHARMACOKINETIC‐PHARMACODYNAMIC (PK/PD) MODELING OF E2027, A SELECTIVE PHOSPHODIESTERASE‐9 (PDE9) INHIBITOR, FOLLOWING SINGLE ASCENDING ORAL DOSES IN HEALTHY VOLUNTEERS. Alzheimer s & Dementia. 13(7S_Part_5). 2 indexed citations

Furuya, Yoshiaki, et al.. (2016). Selective corticotropin-releasing factor 1 receptor antagonist E2508 reduces restraint stress-induced defecation and visceral pain in rat models. Psychoneuroendocrinology. 75. 110–115. 15 indexed citations

Furuya, Yoshiaki, et al.. (2016). Selective corticotropin-releasing factor 1 receptor antagonist E2508 has potent antidepressant-like and anxiolytic-like properties in rodent models. Behavioural Brain Research. 312. 138–147. 8 indexed citations

Matsui, Junji, Mitsuhiro Ino, Taro Semba, et al.. (2013). Abstract 1413: Eribulin caused re-modeling of tumor vasculature altering gene expression profiling in angiogenesis and Epithelial Mesenchymal Transition (EMT) signaling pathway of host cells within human breast cancer cell (BCC) xenografts in nude mice.. Cancer Research. 73(8_Supplement). 1413–1413. 2 indexed citations

Ino, Mitsuhiro, et al.. (2012). Design, synthesis and structure–activity relationships of 5-alkylaminolquinolines as a novel series of CRF1 receptor antagonists. Bioorganic & Medicinal Chemistry Letters. 22(14). 4756–4761. 7 indexed citations

Takahashi, Y., Minako Hashizume, Shigeki Hibi, et al.. (2012). Design, Synthesis, and Structure–Activity Relationships of Novel Pyrazolo[5,1-b]thiazole Derivatives as Potent and Orally Active Corticotropin-Releasing Factor 1 Receptor Antagonists. Journal of Medicinal Chemistry. 55(19). 8450–8463. 11 indexed citations

Hashizume, Minako, Kohdoh Shikata, Y. Takahashi, et al.. (2012). Synthesis and structure–activity relationships of 8-substituted-2-aryl-5-alkylaminoquinolines: Potent, orally active corticotropin-releasing factor-1 receptor antagonists. Bioorganic & Medicinal Chemistry. 20(22). 6559–6578. 7 indexed citations

10.

Takahashi, Y., Shigeki Hibi, Yorihisa Hoshino, et al.. (2012). Synthesis and Structure–Activity Relationships of Pyrazolo[1,5-a]pyridine Derivatives: Potent and Orally Active Antagonists of Corticotropin-Releasing Factor 1 Receptor. Journal of Medicinal Chemistry. 55(11). 5255–5269. 32 indexed citations

11.

Hashizume, Minako, et al.. (2012). Design, synthesis, and structure–activity relationships of a series of 2-Ar-8-methyl-5-alkylaminoquinolines as novel CRF1 receptor antagonists. Bioorganic & Medicinal Chemistry Letters. 22(17). 5372–5378. 5 indexed citations

12.

Sagane, Koji, Kazuhiro Hayakawa, Tomoko Hirohashi, et al.. (2005). Ataxia and peripheral nerve hypomyelination in ADAM22-deficient mice. BMC Neuroscience. 6(1). 33–33. 125 indexed citations

13.

Takahashi, Eiki, et al.. (2005). Increased glucose tolerance in N-type Ca2+ channel α1B-subunit gene-deficient mice. International Journal of Molecular Medicine. 15(6). 937–44. 15 indexed citations

14.

Takahashi, Eiki, Mitsuhiro Ino, Norimasa Miyamoto, & Takeshi Nagasu. (2004). Increased expression of P/Q-type Ca2+ channel α1A subunit mRNA in cerebellum of N-type Ca2+ channel α1B subunit gene-deficient mice. Molecular Brain Research. 124(1). 79–87. 17 indexed citations

15.

Takahashi, Eiki, Mitsuhiro Ino, Norimasa Miyamoto, & Takeshi Nagasu. (2004). Expression analysis of P/Q-type Ca2+ channel α1A subunit mRNA in olfactory mitral cell in N-type Ca2+ channel α1B subunit gene-deficient mice. Neuroscience Letters. 359(1-2). 37–40. 13 indexed citations

16.

Takahashi, Eiki, Mitsuhiro Ino, & Takeshi Nagasu. (2004). Effect of genetic background on Cav2 channel alpha1 and beta subunit messenger RNA expression in cerebellum of N-type Ca2+ channel alpha1B subunit-deficient mice.. PubMed. 54(6). 690–4. 2 indexed citations

17.

Beuckmann, Carsten T., Christopher M. Sinton, Norimasa Miyamoto, Mitsuhiro Ino, & Masashi Yanagisawa. (2003). N-type calcium channel alpha1B subunit (Cav2.2) knock-out mice display hyperactivity and vigilance state differences.. PubMed. 23(17). 6793–7. 59 indexed citations

18.

Beuckmann, Carsten T., Christopher M. Sinton, Norimasa Miyamoto, Mitsuhiro Ino, & Masashi Yanagisawa. (2003). N-Type Calcium Channel α_1BSubunit (Ca_V2.2) Knock-Out Mice Display Hyperactivity and Vigilance State Differences. Journal of Neuroscience. 23(17). 6793–6797. 49 indexed citations

19.

Hatakeyama, Shinji, Minoru Wakamori, Mitsuhiro Ino, et al.. (2001). Differential nociceptive responses in mice lacking the α1B subunit of N-type Ca2+ channels. Neuroreport. 12(11). 2423–2427. 111 indexed citations

20.

Ino, Mitsuhiro, Takashi Yoshinaga, Minoru Wakamori, et al.. (2001). Functional disorders of the sympathetic nervous system in mice lacking the α _1B subunit (Ca _v 2.2) of N-type calcium channels. Proceedings of the National Academy of Sciences. 98(9). 5323–5328. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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