Norimasa Miyamoto
About
Norimasa Miyamoto is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Cardiology and Cardiovascular Medicine.
According to data from OpenAlex, Norimasa Miyamoto has authored 44 papers receiving a total of 2.7k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Cellular and Molecular Neuroscience, 26 papers in Molecular Biology and 14 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Norimasa Miyamoto's work include Neuroscience and Neural Engineering (15 papers), Cardiac electrophysiology and arrhythmias (14 papers) and Ion channel regulation and function (14 papers). Norimasa Miyamoto is often cited by papers focused on Neuroscience and Neural Engineering (15 papers), Cardiac electrophysiology and arrhythmias (14 papers) and Ion channel regulation and function (14 papers). Norimasa Miyamoto collaborates with scholars based in Japan, United States and Slovenia. Norimasa Miyamoto's co-authors include Shinichi Aizawa, Masashi Yanagisawa, Shigeru Kuratani, Michio Yoshida, Isao Matsuo, Kenji Shibata, Toshiyuki Motoike, Rafal M. Kedzierski, Mark A. Valasek and Yumei Xiong and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.
In The Last Decade
Norimasa Miyamoto
43 papers receiving 2.7k citations
Hit Papers
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late) cites · hero ref
| Name | h | Career | Trend | Papers | Cites | |||||
|---|---|---|---|---|---|---|---|---|---|---|
| Norimasa Miyamoto Japan | 22 | 1.7k | 880 | 448 | 420 | 343 | 44 | 2.7k | ||
| T Furukawa Japan | 30 | 1.2k 0.7× | 821 0.9× | 545 1.2× | 243 0.6× | 268 0.8× | 155 | 3.2k | ||
| P.Y.D. Wong Hong Kong | 31 | 1.4k 0.8× | 432 0.5× | 268 0.6× | 289 0.7× | 200 0.6× | 123 | 3.0k | ||
| Junji Yamauchi Japan | 38 | 2.5k 1.5× | 1.3k 1.5× | 368 0.8× | 159 0.4× | 260 0.8× | 181 | 4.4k | ||
| Ralf Middendorff Germany | 31 | 1.1k 0.6× | 409 0.5× | 354 0.8× | 348 0.8× | 282 0.8× | 102 | 2.9k | ||
| Geert Callewaert Belgium | 39 | 2.5k 1.5× | 1.1k 1.3× | 388 0.9× | 702 1.7× | 153 0.4× | 90 | 4.1k | ||
| Toshifumi Itano Japan | 32 | 1.1k 0.7× | 730 0.8× | 289 0.6× | 133 0.3× | 170 0.5× | 132 | 3.0k | ||
| Atsushi Sanbe Japan | 37 | 2.7k 1.6× | 591 0.7× | 420 0.9× | 1.7k 4.0× | 189 0.6× | 112 | 4.5k | ||
| Alfred Bach Germany | 20 | 2.0k 1.1× | 1.2k 1.4× | 264 0.6× | 205 0.5× | 219 0.6× | 24 | 3.6k | ||
| Paul R. Turner New Zealand | 23 | 1.9k 1.1× | 773 0.9× | 924 2.1× | 202 0.5× | 151 0.4× | 36 | 2.9k | ||
| Pedro de la Villa Spain | 34 | 2.4k 1.4× | 1.1k 1.3× | 351 0.8× | 68 0.2× | 134 0.4× | 133 | 3.9k |
Countries citing papers authored by Norimasa Miyamoto
Since
Specialization
Citations
This map shows the geographic impact of Norimasa Miyamoto's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Norimasa Miyamoto with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Norimasa Miyamoto more than expected).
Fields of papers citing papers by Norimasa Miyamoto
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Norimasa Miyamoto. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Norimasa Miyamoto. The network helps show where Norimasa Miyamoto may publish in the future.
Co-authorship network of co-authors of Norimasa Miyamoto
This figure shows the co-authorship network connecting the top 25 collaborators of Norimasa Miyamoto. A scholar is included among the top collaborators of Norimasa Miyamoto based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Norimasa Miyamoto. Norimasa Miyamoto is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Ishihara, Y., et al.. (2025). A novel selective phosphodiesterase 9 inhibitor, irsenontrine (E2027), enhances GluA1 phosphorylation in neurons and improves learning and memory via cyclic GMP elevation. Neuropharmacology. 273. 110428–110428.
2.
Matsuda, Naoki, et al.. (2025). Advanced cardiotoxicity profiling using field potential imaging with UHD-CMOS-MEA in human iPSC-derived cardiomyocytes. Toxicological Sciences. 208(2). 384–400.
3.
Takano, M., Hiroki Ishihara, Yoshitane Nozaki, et al.. (2024). Comparative sensitivity of laboratory animals used for preclinical convulsion risk assessment to drug-induced convulsion. The Journal of Toxicological Sciences. 49(9). 409–423.
4.
Yamazaki, Daiju, Takashi Kitaguchi, Tomohiko Taniguchi, et al.. (2018). Proarrhythmia risk prediction using human induced pluripotent stem cell-derived cardiomyocytes. Journal of Pharmacological Sciences. 136(4). 249–256.
5.
Kimura, Takayuki, Mai Uesugi, Kazuma Takase, Norimasa Miyamoto, & Kohei Sawada. (2017). Hsp90 inhibitor geldanamycin attenuates the cytotoxicity of sunitinib in cardiomyocytes via inhibition of the autophagy pathway. Toxicology and Applied Pharmacology. 329. 282–292.
6.
Kitaguchi, Takashi, Tomohiko Taniguchi, Hiroyuki Ando, et al.. (2017). CSAHi study: Detection of drug-induced ion channel/receptor responses, QT prolongation, and arrhythmia using multi-electrode arrays in combination with human induced pluripotent stem cell-derived cardiomyocytes. Journal of Pharmacological and Toxicological Methods. 85. 73–81.
7.
Ando, Hiroyuki, Takashi Yoshinaga, Wataru Yamamoto, et al.. (2016). A new paradigm for drug-induced torsadogenic risk assessment using human iPS cell-derived cardiomyocytes. Journal of Pharmacological and Toxicological Methods. 84. 111–127.
8.
Fukushima, Kazuyuki, Kazuto Yamazaki, Norimasa Miyamoto, & Kohei Sawada. (2016). Functional Characterization of Acetylcholine Receptors Expressed in Human Neurons Differentiated from Hippocampal Neural Stem/Progenitor Cells. SLAS DISCOVERY. 21(10). 1065–1074.
9.
Asakura, Keiichi, Seiji Hayashi, Atsuko Ojima, et al.. (2015). Improvement of acquisition and analysis methods in multi-electrode array experiments with iPS cell-derived cardiomyocytes. Journal of Pharmacological and Toxicological Methods. 75. 17–26.
10.
Kitaguchi, Takashi, Tomohiko Taniguchi, Atsuko Ojima, et al.. (2015). CSAHi study: Evaluation of multi-electrode array in combination with human iPS cell-derived cardiomyocytes to predict drug-induced QT prolongation and arrhythmia — Effects of 7 reference compounds at 10 facilities. Journal of Pharmacological and Toxicological Methods. 78. 93–102.
11.
Narita, Yusuke, Kiyoshi Okamoto, Kazuma Takase, et al.. (2014). Novel ATP-Competitive MEK Inhibitor E6201 Is Effective against Vemurafenib-Resistant Melanoma Harboring the MEK1-C121S Mutation in a Preclinical Model. Molecular Cancer Therapeutics. 13(4). 823–832.
12.
Uesugi, Mai, Atsuko Ojima, Tomohiko Taniguchi, Norimasa Miyamoto, & Kohei Sawada. (2013). Low-density plating is sufficient to induce cardiac hypertrophy and electrical remodeling in highly purified human iPS cell-derived cardiomyocytes. Journal of Pharmacological and Toxicological Methods. 69(2). 177–188.
13.
Shikata, Kohdoh, Mai Uesugi, Hiroyuki Katayama, et al.. (2013). Prediction of relaxin-3-induced downstream pathway resulting in anxiolytic-like behaviors in rats based on a microarray and peptidome analysis. Journal of Receptors and Signal Transduction. 33(4). 224–233.
14.
Tokuhara, Naoki, Kana Namiki, Mai Uesugi, et al.. (2010). N-type Calcium Channel in the Pathogenesis of Experimental Autoimmune Encephalomyelitis*. Journal of Biological Chemistry. 285(43). 33294–33306.
15.
Sagane, Koji, Kazuhiro Hayakawa, Tomoko Hirohashi, et al.. (2005). Ataxia and peripheral nerve hypomyelination in ADAM22-deficient mice. BMC Neuroscience. 6(1). 33–33.
16.
Takahashi, Eiki, Mitsuhiro Ino, Norimasa Miyamoto, & Takeshi Nagasu. (2004). Increased expression of P/Q-type Ca2+ channel α1A subunit mRNA in cerebellum of N-type Ca2+ channel α1B subunit gene-deficient mice. Molecular Brain Research. 124(1). 79–87.
17.
Takahashi, Eiki, et al.. (2000). Expression analysis of Escherichia coli lacZ reporter gene in transgenic mice. Brain Research Protocols. 5(2). 159–166.
18.
Takahashi, Eiki, Yoshiyuki Murata, Tohru Oki, et al.. (1999). Isolation and Functional Characterization of the 5′-Upstream Region of Mouse P/Q-Type Ca2+ Channel α1A Subunit Gene. Biochemical and Biophysical Research Communications. 260(1). 54–59.
19.
Takahashi, Eiki, Norimasa Miyamoto, Tohru Oki, et al.. (1999). Neuron-specific expression of reporter gene in transgenic mice carrying the 5′-upstream region of mouse P/Q-type Ca2+ channel α1A subunit gene fused to E. coli lacZ reporter gene. Brain Research. 850(1-2). 47–54.
20.
Miyamoto, Norimasa, et al.. (1990). Primate's p53 inhibits SV40 DNA replication in vitro. Biochemical and Biophysical Research Communications. 168(2). 604–608.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
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