Mikio Kuraya

797 total citations
17 papers, 671 citations indexed

About

Mikio Kuraya is a scholar working on Immunology, Molecular Biology and Epidemiology. According to data from OpenAlex, Mikio Kuraya has authored 17 papers receiving a total of 671 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Immunology, 4 papers in Molecular Biology and 3 papers in Epidemiology. Recurrent topics in Mikio Kuraya's work include Complement system in diseases (8 papers), Immune Cell Function and Interaction (5 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). Mikio Kuraya is often cited by papers focused on Complement system in diseases (8 papers), Immune Cell Function and Interaction (5 papers) and Monoclonal and Polyclonal Antibodies Research (3 papers). Mikio Kuraya collaborates with scholars based in Japan, Sweden and United Kingdom. Mikio Kuraya's co-authors include Teizo Fujita, Misao Matsushita, Naotaka Hamasaki, Mitsushi Tsujimura, Hiroshi Shiraki, Wilhelm Schwaeble, Zhenping Ming, Nicholas J. Lynch, Silke Roscher and Thomas Härtung and has published in prestigious journals such as The Journal of Immunology, Annals of the Rheumatic Diseases and European Journal of Immunology.

In The Last Decade

Mikio Kuraya

16 papers receiving 666 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Mikio Kuraya Japan	10	516	126	108	106	105	17	671
Troels R. Kjær Denmark	13	435 0.8×	98 0.8×	60 0.6×	126 1.2×	84 0.8×	17	580
Stacey L. Mueller‐Ortiz United States	14	365 0.7×	148 1.2×	51 0.5×	69 0.7×	95 0.9×	22	665
Michelle Elvington United States	11	364 0.7×	90 0.7×	34 0.3×	49 0.5×	55 0.5×	14	526
D. W. Golde United States	12	539 1.0×	220 1.7×	73 0.7×	244 2.3×	97 0.9×	21	975
Dario Magaletti United States	11	853 1.7×	200 1.6×	29 0.3×	49 0.5×	67 0.6×	14	1.1k
László Beinrohr Hungary	9	303 0.6×	88 0.7×	123 1.1×	185 1.7×	33 0.3×	17	463
G. Schmidt Germany	13	244 0.5×	100 0.8×	72 0.7×	147 1.4×	34 0.3×	33	511
Shun‐ichi Hirose Japan	14	332 0.6×	133 1.1×	36 0.3×	86 0.8×	60 0.6×	61	727
Dean D. Metcalfe United States	9	506 1.0×	136 1.1×	30 0.3×	39 0.4×	80 0.8×	11	707
Hideki Tsujimura Japan	13	1.1k 2.1×	242 1.9×	47 0.4×	117 1.1×	111 1.1×	44	1.4k

Countries citing papers authored by Mikio Kuraya

Since Specialization

Citations

This map shows the geographic impact of Mikio Kuraya's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mikio Kuraya with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mikio Kuraya more than expected).

Fields of papers citing papers by Mikio Kuraya

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mikio Kuraya. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mikio Kuraya. The network helps show where Mikio Kuraya may publish in the future.

Co-authorship network of co-authors of Mikio Kuraya

This figure shows the co-authorship network connecting the top 25 collaborators of Mikio Kuraya. A scholar is included among the top collaborators of Mikio Kuraya based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mikio Kuraya. Mikio Kuraya is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Kuraya, Mikio, et al.. (2005). Specific binding of L-ficolin and H-ficolin to apoptotic cells leads to complement activation. Immunobiology. 209(9). 689–697. 120 indexed citations

Lynch, Nicholas J., Silke Roscher, Thomas Härtung, et al.. (2004). L-Ficolin Specifically Binds to Lipoteichoic Acid, a Cell Wall Constituent of Gram-Positive Bacteria, and Activates the Lectin Pathway of Complement. The Journal of Immunology. 172(2). 1198–1202. 210 indexed citations

Abe, Sumiko, et al.. (2004). Induction of nerve growth factor mRNA in a rat dorsal root ganglion after application of a tourniquet. Acta Neuropathologica. 108(3). 183–8. 4 indexed citations

Abe, Sumiko, et al.. (2003). Changes in mRNA expression patterns for cytokines in blood leukocytes of a rat tourniquet model. Legal Medicine. 5. S275–S277. 1 indexed citations

Abe, Sumiko, et al.. (2003). Nitric oxide synthase expressions in rat dorsal root ganglion after a hind limb tourniquet. Neuroreport. 14(17). 2267–2270. 11 indexed citations

Matsushita, Misao, Mikio Kuraya, Naotaka Hamasaki, et al.. (2002). Activation of the Lectin Complement Pathway by H-Ficolin (Hakata Antigen). The Journal of Immunology. 168(7). 3502–3506. 168 indexed citations

Kuraya, Mikio. (2002). Expression of H-ficolin/Hakata antigen, mannose-binding lectin-associated serine protease (MASP)-1 and MASP-3 by human glioma cell line T98G. International Immunology. 15(1). 109–117. 47 indexed citations

Endo, Yushiro, Minoru Takahashi, Mikio Kuraya, et al.. (2002). Functional characterization of human mannose-binding lectin-associated serine protease (MASP)-1/3 and MASP-2 promoters, and comparison with the C1s promoter. International Immunology. 14(10). 1193–1201. 16 indexed citations

Abe, Sumiko, et al.. (1999). Semen specific γ-glutamyltransferase carries ABH antigens: a sandwich ELISA for simultaneous semen detection and its ABO blood typing. Clinica Chimica Acta. 283(1-2). 183–194. 1 indexed citations

10.

Kuraya, Mikio. (1998). Signal transduction via a protein associated with a glycosylphosphatidylinositol-anchored protein, decay-accelerating factor (DAF/CD55). International Immunology. 10(4). 473–480. 13 indexed citations

11.

Kuraya, Mikio, T. Sato, & Teizo Fujita. (1995). C3d and Epstein‐Barr Virus (CR2/CD21 Ligands) Stimulate Cells of an HTLV‐I Line, MT‐2. Microbiology and Immunology. 39(2). 145–151. 1 indexed citations

12.

Watanabe, Hiroshi, Mikio Kuraya, Reiji Kasukawa, et al.. (1995). Analysis of C5a receptor by monoclonal antibody. Journal of Immunological Methods. 185(1). 19–29. 15 indexed citations

13.

Kuraya, Mikio, János Minárovits, Hidechika Okada, & Eva Klein. (1993). HRF20/CD59 complement regulatory protein expression is phenotype-dependent and inducible by the hypomethylating agent 5-azacytidine on Burkitt's lymphoma cell lines. Immunology Letters. 37(1). 35–39. 3 indexed citations

14.

Sato, Y., Ryo Sato, Hiroshi Watanabe, et al.. (1993). Complement activating properties of monoreactive and polyreactive IgM rheumatoid factors.. Annals of the Rheumatic Diseases. 52(11). 795–800. 18 indexed citations

15.

Kuraya, Mikio, George Klein, Eva Klein, & Eitan Yefenof. (1992). Expression of the complement regulatory proteins CD21, CD55 and CD59 on Burkitt lymphoma lines: their role in sensitivity to human serum‐mediated lysis. European Journal of Immunology. 22(7). 1871–1876. 33 indexed citations

16.

Kuraya, Mikio, Bo Nilsson, Kristina Nilsson‐Ekdahl, & Eva Klein. (1990). C3d-mediated negative and positive signals on the proliferation of human B cells separated from blood. Immunology Letters. 26(1). 51–58. 9 indexed citations

17.

Inoue, Tadashi, et al.. (1987). Antioxidative activities of a reductant in the ultrafiltrate of human placental homogenate.. PubMed. 8(2 2D Half). 47–52. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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