Midori Okamura

423 total citations
13 papers, 336 citations indexed

About

Midori Okamura is a scholar working on Molecular Biology, Pathology and Forensic Medicine and Physiology. According to data from OpenAlex, Midori Okamura has authored 13 papers receiving a total of 336 indexed citations (citations by other indexed papers that have themselves been cited), including 4 papers in Molecular Biology, 3 papers in Pathology and Forensic Medicine and 3 papers in Physiology. Recurrent topics in Midori Okamura's work include Ion channel regulation and function (4 papers), Cardiac Ischemia and Reperfusion (3 papers) and Cardiac electrophysiology and arrhythmias (2 papers). Midori Okamura is often cited by papers focused on Ion channel regulation and function (4 papers), Cardiac Ischemia and Reperfusion (3 papers) and Cardiac electrophysiology and arrhythmias (2 papers). Midori Okamura collaborates with scholars based in Japan and United States. Midori Okamura's co-authors include Hitoshi Hara, Genshi Egusa, Michio Yamakido, Kiminori Yamane, Seijiro Kado, Akinori Noma, Satoru Takahashi, Masami Ojima, Naoki Morito and Lai‐Hua Xie and has published in prestigious journals such as The Journal of Physiology, Journal of the American Society of Nephrology and British Journal of Pharmacology.

In The Last Decade

Midori Okamura

13 papers receiving 328 citations

Peers

Midori Okamura
Ruiyan Ma China
Geng Xu China
Linda T. Archer United States
Edu Suárez Puerto Rico
Midori Okamura
Citations per year, relative to Midori Okamura Midori Okamura (= 1×) peers Jean-Claude Dussaule

Countries citing papers authored by Midori Okamura

Since Specialization
Citations

This map shows the geographic impact of Midori Okamura's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Midori Okamura with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Midori Okamura more than expected).

Fields of papers citing papers by Midori Okamura

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Midori Okamura. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Midori Okamura. The network helps show where Midori Okamura may publish in the future.

Co-authorship network of co-authors of Midori Okamura

This figure shows the co-authorship network connecting the top 25 collaborators of Midori Okamura. A scholar is included among the top collaborators of Midori Okamura based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Midori Okamura. Midori Okamura is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

13 of 13 papers shown
1.
Morito, Naoki, Keigyou Yoh, Masami Ojima, et al.. (2014). Overexpression of Mafb in Podocytes Protects against Diabetic Nephropathy. Journal of the American Society of Nephrology. 25(11). 2546–2557. 33 indexed citations
2.
Okamura, Midori, Keigyou Yoh, Masami Ojima, Naoki Morito, & Satoru Takahashi. (2014). Overexpression of GATA-3 in T Cells Accelerates Dextran Sulfate Sodium-Induced Colitis. EXPERIMENTAL ANIMALS. 63(2). 133–140. 25 indexed citations
3.
Yoh, Keigyou, Naoki Morito, Masami Ojima, et al.. (2012). Overexpression of RORγt under control of the CD2 promoter induces polyclonal plasmacytosis and autoantibody production in transgenic mice. European Journal of Immunology. 42(8). 1999–2009. 20 indexed citations
5.
Oketani, Naoya, Masafumi Kakei, Midori Okamura, et al.. (2002). Regulation of KATPchannels by P2Ypurinoceptors coupled to PIP2metabolism in guinea pig ventricular cells. American Journal of Physiology-Heart and Circulatory Physiology. 282(2). H757–H765. 13 indexed citations
6.
Okamura, Midori, et al.. (2001). State-dependent modification of ATP-sensitive K+channels by phosphatidylinositol 4,5-bisphosphate. American Journal of Physiology-Cell Physiology. 280(2). C303–C308. 19 indexed citations
7.
Kakei, Masafumi, et al.. (2000). On the mechanism of ADP‐induced alteration of sulphonylurea sensitivity in cardiac ATP‐sensitive K+ channels. British Journal of Pharmacology. 130(6). 1411–1417. 3 indexed citations
8.
Xie, Lai‐Hua, et al.. (1999). Wortmannin, an inhibitor of phosphatidylinositol kinases, blocks the MgATP‐dependent recovery of Kir6.2/SUR2A channels. The Journal of Physiology. 514(3). 655–665. 52 indexed citations
9.
Matsushita, Kakushi, Naomichi Arima, Hiroshi Fujiwara, et al.. (1999). Spontaneous regression associated with apoptosis in a patient with acute-type adult T-cell leukemia. American Journal of Hematology. 61(2). 144–148. 10 indexed citations
10.
Shiota, Yutaro, James G. Wilson, Midori Okamura, et al.. (1996). Adult Respiratory Distress Syndrome Induced by a Chinese Medicine, Kamisyoyo-San.. Internal Medicine. 35(6). 494–496. 15 indexed citations
12.
Egusa, Genshi, Fumiyo Murakami, Chikako Ito, et al.. (1993). Westernized food habits and concentrations of serum lipids in the Japanese. Atherosclerosis. 100(2). 249–255. 73 indexed citations
13.
Mori, Hiroshi, Masamichi Okubo, Midori Okamura, et al.. (1992). Abnormalities of Pulmonary Function in Patients with Non-insulin-Dependent Diabetes Mellitus.. Internal Medicine. 31(2). 189–193. 44 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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